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Perichondrium phenotype and border function are regulated by Ext1 and heparan sulfate in developing long bones: A mechanism likely deranged in Hereditary Multiple Exostoses

Developmental biology, 2013-05, Vol.377 (1), p.100-112

2013

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Unsuspected osteochondroma-like outgrowths in the cranial base of Hereditary Multiple Exostoses patients and modeling and treatment with a BMP antagonist in mice

PLoS genetics, 2017-04, Vol.13 (4), p.e1006742-e1006742

2017

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Heparan sulfate in skeletal development, growth, and pathology: The case of hereditary multiple exostoses

Developmental dynamics, 2013-09, Vol.242 (9), p.1021-1032

2013

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Epiphyseal abnormalities, trabecular bone loss and articular chondrocyte hypertrophy develop in the long bones of postnatal Ext1 -deficient mice

Bone (New York, N.Y.), 2013-11, Vol.57 (1), p.220-231

2013

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Heparanase stimulates chondrogenesis and is up-regulated in human ectopic cartilage: a mechanism possibly involved in hereditary multiple exostoses

The American journal of pathology, 2015-06, Vol.185 (6), p.1676-1685

2015

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HhAntag, a Hedgehog Signaling Antagonist, Suppresses Chondrogenesis and Modulates Canonical and Non-Canonical BMP Signaling

Journal of cellular physiology, 2016-05, Vol.231 (5), p.1033-1044

2016

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Genotype-phenotype correlation study in 529 patients with multiple hereditary exostoses: identification of "protective" and "risk" factors

Journal of bone and joint surgery. American volume, 2011-12, Vol.93 (24), p.2294-2302

2011

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HhAntag, a Hedgehog Signaling Antagonist, Suppresses Chondrogenesis and Modulates Canonical and Non-Canonical BMP Signaling: HhANTAG SUPPRESSES CHONDROGENESIS

Journal of cellular physiology, 2016-05, Vol.231 (5), p.1033-1044

2016

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Heparanase Stimulates Chondrogenesis and Is Up-Regulated in Human Ectopic Cartilage

The American journal of pathology, 2015-06, Vol.185 (6), p.1676-1685

2015

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Epiphyseal abnormalities, trabecular bone loss and articular chondrocyte hypertrophy develop in the long bones of postnatal Ext1-deficient mice1

Bone (New York, N.Y.), 2013-08, Vol.57 (1), p.220-231

2013

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Loss of β-Catenin Induces Multifocal Periosteal Chondroma-Like Masses in Mice

The American journal of pathology, 2013-03, Vol.182 (3), p.917-927

2013

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The type 2 diabetes associated rs7903146 T allele within TCF7L2 is significantly under-represented in Hereditary Multiple Exostoses: Insights into pathogenesis

Bone (New York, N.Y.), 2015-03, Vol.72, p.123-127

2015

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Metabolic syndrome and adipokine levels in systemic lupus erythematosus and systemic sclerosis

Clinical rheumatology, 2021-10, Vol.40 (10), p.4253-4258

2021

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Disentangling the Association of Hydroxychloroquine Treatment with Mortality in Covid-19 Hospitalized Patients through Hierarchical Clustering

Journal of healthcare engineering, 2021-06, Vol.2021, p.1-10

2021

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Perichondrium phenotype and border function are regulated by Ext1 and heparan sulfate in developing long bones: A mechanism likely deranged in Hereditary Multiple Exostoses

Unsuspected osteochondroma-like outgrowths in the cranial base of Hereditary Multiple Exostoses patients and modeling and treatment with a BMP antagonist in mice

Heparan sulfate in skeletal development, growth, and pathology: The case of hereditary multiple exostoses

Epiphyseal abnormalities, trabecular bone loss and articular chondrocyte hypertrophy develop in the long bones of postnatal Ext1 -deficient mice

Heparanase stimulates chondrogenesis and is up-regulated in human ectopic cartilage: a mechanism possibly involved in hereditary multiple exostoses

HhAntag, a Hedgehog Signaling Antagonist, Suppresses Chondrogenesis and Modulates Canonical and Non-Canonical BMP Signaling

Genotype-phenotype correlation study in 529 patients with multiple hereditary exostoses: identification of "protective" and "risk" factors

HhAntag, a Hedgehog Signaling Antagonist, Suppresses Chondrogenesis and Modulates Canonical and Non-Canonical BMP Signaling: HhANTAG SUPPRESSES CHONDROGENESIS

Heparanase Stimulates Chondrogenesis and Is Up-Regulated in Human Ectopic Cartilage

Epiphyseal abnormalities, trabecular bone loss and articular chondrocyte hypertrophy develop in the long bones of postnatal Ext1-deficient mice1

Loss of β-Catenin Induces Multifocal Periosteal Chondroma-Like Masses in Mice

The type 2 diabetes associated rs7903146 T allele within TCF7L2 is significantly under-represented in Hereditary Multiple Exostoses: Insights into pathogenesis

Metabolic syndrome and adipokine levels in systemic lupus erythematosus and systemic sclerosis

Disentangling the Association of Hydroxychloroquine Treatment with Mortality in Covid-19 Hospitalized Patients through Hierarchical Clustering

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