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Defects in limb, craniofacial, and thymic development in Jagged2 mutant mice

Genes & development, 1998-04, Vol.12 (7), p.1046-1057

1998

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Acceleration of type 1 diabetes by a coxsackievirus infection requires a preexisting critical mass of autoreactive T-cells in pancreatic islets

Diabetes (New York, N.Y.), 2000-05, Vol.49 (5), p.708-711

2000

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Th1 to Th2 Cytokine Shifts in Nonobese Diabetic Mice: Sometimes an Outcome, Rather Than the Cause, of Diabetes Resistance Elicited by Immunostimulation

The Journal of immunology (1950), 2001-01, Vol.166 (2), p.1352-1359

2001

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NOD mice have a generalized defect in their response to transplantation tolerance induction

Diabetes (New York, N.Y.), 1999-05, Vol.48 (5), p.967

1999

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Antigen-specific mediated suppression of beta cell autoimmunity by plasmid DNA vaccination

The Journal of immunology (1950), 2001-02, Vol.166 (3), p.2122-2132

2001

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Interferon-gamma receptor signaling is dispensable in the development of autoimmune type 1 diabetes in NOD mice

Diabetes (New York, N.Y.), 2000-12, Vol.49 (12), p.2007-2011

2000

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Defects in the differentiation and function of antigen presenting cells in NOD/Lt mice

The Journal of immunology (1950), 1993-03, Vol.150 (6), p.2534

1993

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Interleukin 4 reverses T cell proliferative unresponsiveness and prevents the onset of diabetes in nonobese diabetic mice

The Journal of experimental medicine, 1993-07, Vol.178 (1), p.87-99

1993

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A locus on mouse chromosome 13 inversely regulates CD1d expression and the development of invariant natural killer T-cells

Genes and immunity, 2015-04, Vol.16 (3), p.221-230

2015

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Hematopoietic Stem-Cell Defects Underlying Abnormal Macrophage Development and Maturation in NOD/Lt Mice: Defective Regulation of Cytokine Receptors and Protein Kinase C

Proceedings of the National Academy of Sciences - PNAS, 1993-10, Vol.90 (20), p.9625-9629

1993

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Genetic control of murine invariant natural killer T-cell development dynamically differs dependent on the examined tissue type

Genes and immunity, 2012-02, Vol.13 (2), p.164-174

2012

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Induction of Glutamic Acid Decarboxylase 65-Specific Th2 Cells and Suppression of Autoimmune Diabetes at Late Stages of Disease Is Epitope Dependent

The Journal of immunology (1950), 1999-08, Vol.163 (3), p.1178-1187

1999

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B Lymphocytes Are Critical Antigen-Presenting Cells for the Initiation of T Cell-Mediated Autoimmune Diabetes in Nonobese Diabetic Mice

The Journal of immunology (1950), 1998-10, Vol.161 (8), p.3912-3918

1998

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Comparative therapeutic effects of orally administered 1,25-dihydroxyvitamin D₃ and 1alpha-hydroxyvitamin D₃ on type-1 diabetes in non-obese diabetic mice fed a normal-calcaemic diet

Clinical and experimental immunology, 2008-01, Vol.151 (1), p.76-85

2008

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The preferential ability of B lymphocytes to act as diabetogenic APC in NOD mice depends on expression of self‐antigen‐specific immunoglobulin receptors

European journal of immunology, 2002-12, Vol.32 (12), p.3657-3666

2002

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Defective activation of T suppressor cell function in nonobese diabetic mice. Potential relation to cytokine deficiencies

The Journal of immunology (1950), 1988-06, Vol.140 (11), p.3801

1988

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Inhibition of Autoimmune Diabetes in Nonobese Diabetic Mice by Transgenic Restoration of H2-E MHC Class II Expression: Additive, But Unequal, Involvement of Multiple APC Subtypes

The Journal of immunology (1950), 2001-08, Vol.167 (4), p.2404-2410

2001

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Use of recombinant congenic and congenic strains of NOD mice to identify a new insulin-dependent diabetes resistance gene

The Journal of experimental medicine, 1994-10, Vol.180 (4), p.1553-1558

1994

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Conditioning the genome identifies additional diabetes resistance loci in Type I diabetes resistant NOR Lt mice

Genes and immunity, 2005-09, Vol.6 (6), p.528-538

2005

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Genetic and pathogenic basis of autoimmune diabetes in NOD mice

Current opinion in immunology, 1994, Vol.6 (6), p.900-906

1994

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B Cell Selection Defects Underlie the Development of Diabetogenic APCs in Nonobese Diabetic Mice

The Journal of immunology (1950), 2004-04, Vol.172 (8), p.5086-5094

2004

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Functional Evidence for the Mediation of Diabetogenic T Cell Responses by HLA-A2.1 MHC Class I Molecules through Transgenic Expression in NOD Mice

Proceedings of the National Academy of Sciences - PNAS, 2002-10, Vol.99 (21), p.13753-13758

2002

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Immunostimulation circumvents diabetes in NOD/Lt mice

Journal of autoimmunity, 1989-12, Vol.2 (6), p.759-776

1989

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Reevaluation of the major histocompatibility complex genes of the NOD-progenitor CTS/Shi strain

Diabetes (New York, N.Y.), 2000-01, Vol.49 (1), p.131-134

2000

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Human Clonal CD8 Autoreactivity to an IGRP Islet Epitope Shared between Mice and Men

Annals of the New York Academy of Sciences, 2007-04, Vol.1103 (1), p.192-195

2007

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Defects in limb, craniofacial, and thymic development in Jagged2 mutant mice

Acceleration of type 1 diabetes by a coxsackievirus infection requires a preexisting critical mass of autoreactive T-cells in pancreatic islets

Th1 to Th2 Cytokine Shifts in Nonobese Diabetic Mice: Sometimes an Outcome, Rather Than the Cause, of Diabetes Resistance Elicited by Immunostimulation

NOD mice have a generalized defect in their response to transplantation tolerance induction

Antigen-specific mediated suppression of beta cell autoimmunity by plasmid DNA vaccination

Interferon-gamma receptor signaling is dispensable in the development of autoimmune type 1 diabetes in NOD mice

Defects in the differentiation and function of antigen presenting cells in NOD/Lt mice

Interleukin 4 reverses T cell proliferative unresponsiveness and prevents the onset of diabetes in nonobese diabetic mice

A locus on mouse chromosome 13 inversely regulates CD1d expression and the development of invariant natural killer T-cells

Hematopoietic Stem-Cell Defects Underlying Abnormal Macrophage Development and Maturation in NOD/Lt Mice: Defective Regulation of Cytokine Receptors and Protein Kinase C

Genetic control of murine invariant natural killer T-cell development dynamically differs dependent on the examined tissue type

Induction of Glutamic Acid Decarboxylase 65-Specific Th2 Cells and Suppression of Autoimmune Diabetes at Late Stages of Disease Is Epitope Dependent

B Lymphocytes Are Critical Antigen-Presenting Cells for the Initiation of T Cell-Mediated Autoimmune Diabetes in Nonobese Diabetic Mice

Comparative therapeutic effects of orally administered 1,25-dihydroxyvitamin D₃ and 1alpha-hydroxyvitamin D₃ on type-1 diabetes in non-obese diabetic mice fed a normal-calcaemic diet

The preferential ability of B lymphocytes to act as diabetogenic APC in NOD mice depends on expression of self‐antigen‐specific immunoglobulin receptors

Defective activation of T suppressor cell function in nonobese diabetic mice. Potential relation to cytokine deficiencies

Inhibition of Autoimmune Diabetes in Nonobese Diabetic Mice by Transgenic Restoration of H2-E MHC Class II Expression: Additive, But Unequal, Involvement of Multiple APC Subtypes

Use of recombinant congenic and congenic strains of NOD mice to identify a new insulin-dependent diabetes resistance gene

Conditioning the genome identifies additional diabetes resistance loci in Type I diabetes resistant NOR Lt mice

Genetic and pathogenic basis of autoimmune diabetes in NOD mice

B Cell Selection Defects Underlie the Development of Diabetogenic APCs in Nonobese Diabetic Mice

Functional Evidence for the Mediation of Diabetogenic T Cell Responses by HLA-A2.1 MHC Class I Molecules through Transgenic Expression in NOD Mice

Immunostimulation circumvents diabetes in NOD/Lt mice

Reevaluation of the major histocompatibility complex genes of the NOD-progenitor CTS/Shi strain

Human Clonal CD8 Autoreactivity to an IGRP Islet Epitope Shared between Mice and Men

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