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Regulation of Insulin-Like Growth Factor (IGF)-I Action by Matrix Metalloproteinase-3 Involves Selective Disruption of IGF-I/IGF-Binding Protein-3 Complexes

Endocrinology (Philadelphia), 2004-02, Vol.145 (2), p.620-626

2004

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Characterization of Glycosaminoglycan-binding Domains Present in Insulin-like Growth Factor-binding Protein-3

The Journal of biological chemistry, 1996-06, Vol.271 (25), p.14676-14679

1996

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Insulin-like Growth Factor (IGF)-binding Protein-3 (IGFBP-3) Functions as an IGF-reversible Inhibitor of IGFBP-4 Proteolysis

The Journal of biological chemistry, 1995-11, Vol.270 (46), p.27481-27488

1995

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Field performance of transgenic tomato with reduced pectin methylesterase activity

Journal of the American Society for Horticultural Science, 1995-09, Vol.120 (5), p.765-770

1995

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Heparin-Binding, Highly Basic Regions within the Thyroglobulin Type-1 Repeat of Insulin-Like Growth Factor (IGF)-Binding Proteins (IGFBPs) -3, -5, and -6 Inhibit IGFBP-4 Degradation

Endocrinology (Philadelphia), 1997-06, Vol.138 (6), p.2280-2285

1997

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Characterization of insulin-like growth factor-binding protein 5-degrading proteases produced throughout murine osteoblast differentiation

Endocrinology (Philadelphia), 1995-08, Vol.136 (8), p.3527-3533

1995

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MMPs Are IGFBP‐Degrading Proteinases: Implications for Cell Proliferation and Tissue Growth

Annals of the New York Academy of Sciences, 1999-06, Vol.878 (1), p.696-699

1999

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Heparin-Binding, Highly Basic Regions within the Thyroglobulin Type-1 Repeat of Insulin-Like Growth Factor (IGF)-Binding Proteins (IGFBPs) -3, -5, and -6 Inhibit IGFBP-4 Degradation1

Endocrinology (Philadelphia), 1997-06, Vol.138 (6), p.2280-2285

1997

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Insulin-like growth factor binding protein (IGFBP) substrate zymography. A new tool to identify and characterize IGFBP-degrading proteinases

Endocrine, 1997-08, Vol.7 (1), p.33-36

1997

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Matrix metalloproteinases as insulin-like growth factor binding protein-degrading proteinases

Progress in growth factor research, 1995, Vol.6 (2), p.255-263

1995

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Depletion of human regulatory T cells specifically enhances antigen-specific immune responses to cancer vaccines

Blood, 2008-08, Vol.112 (3), p.610-618

2008

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Adenovirus Infection Triggers a Rapid, MyD88-Regulated Transcriptome Response Critical to Acute-Phase and Adaptive Immune Responses In Vivo

Journal of Virology, 2007-02, Vol.81 (4), p.1796-1812

2007

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Multiple Innate Inflammatory Responses Induced after Systemic Adenovirus Vector Delivery Depend on a Functional Complement System

Molecular therapy, 2006-10, Vol.14 (4), p.588-598

2006

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Production and Characterization of Improved Adenovirus Vectors with the E1, E2b, and E3 Genes Deleted

Journal of Virology, 1998-02, Vol.72 (2), p.926-933

1998

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Replication-attenuated Human Adenoviral Type 4 vectors elicit capsid dependent enhanced innate immune responses that are partially dependent upon interactions with the complement system

Virology (New York, N.Y.), 2008-05, Vol.374 (2), p.453-467

2008

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Efficacy of Gene Therapy for a Prototypical Lysosomal Storage Disease (GSD-II) Is Critically Dependent on Vector Dose, Transgene Promoter, and the Tissues Targeted for Vector Transduction

Molecular therapy, 2002-04, Vol.5 (4), p.436-446

2002

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Persistence of an [E1-, polymerase-] adenovirus vector despite transduction of a neoantigen into immune-competent mice

Human gene therapy, 1999-02, Vol.10 (3), p.355-364

1999

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Multiply deleted [E1, polymerase-, and pTP-] adenovirus vector persists despite deletion of the preterminal protein

The journal of gene medicine, 2000-07, Vol.2 (4), p.250-259

2000

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1012. Ad Interactions with the Complement Systems of Humans and Mice

Molecular therapy, 2004-05, Vol.9 (S1), p.S387-S388

2004

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Regulation of Insulin-Like Growth Factor (IGF)-I Action by Matrix Metalloproteinase-3 Involves Selective Disruption of IGF-I/IGF-Binding Protein-3 Complexes

Characterization of Glycosaminoglycan-binding Domains Present in Insulin-like Growth Factor-binding Protein-3

Insulin-like Growth Factor (IGF)-binding Protein-3 (IGFBP-3) Functions as an IGF-reversible Inhibitor of IGFBP-4 Proteolysis

Field performance of transgenic tomato with reduced pectin methylesterase activity

Heparin-Binding, Highly Basic Regions within the Thyroglobulin Type-1 Repeat of Insulin-Like Growth Factor (IGF)-Binding Proteins (IGFBPs) -3, -5, and -6 Inhibit IGFBP-4 Degradation

Characterization of insulin-like growth factor-binding protein 5-degrading proteases produced throughout murine osteoblast differentiation

MMPs Are IGFBP‐Degrading Proteinases: Implications for Cell Proliferation and Tissue Growth

Heparin-Binding, Highly Basic Regions within the Thyroglobulin Type-1 Repeat of Insulin-Like Growth Factor (IGF)-Binding Proteins (IGFBPs) -3, -5, and -6 Inhibit IGFBP-4 Degradation1

Insulin-like growth factor binding protein (IGFBP) substrate zymography. A new tool to identify and characterize IGFBP-degrading proteinases

Matrix metalloproteinases as insulin-like growth factor binding protein-degrading proteinases

Depletion of human regulatory T cells specifically enhances antigen-specific immune responses to cancer vaccines

Adenovirus Infection Triggers a Rapid, MyD88-Regulated Transcriptome Response Critical to Acute-Phase and Adaptive Immune Responses In Vivo

Multiple Innate Inflammatory Responses Induced after Systemic Adenovirus Vector Delivery Depend on a Functional Complement System

Production and Characterization of Improved Adenovirus Vectors with the E1, E2b, and E3 Genes Deleted

Replication-attenuated Human Adenoviral Type 4 vectors elicit capsid dependent enhanced innate immune responses that are partially dependent upon interactions with the complement system

Efficacy of Gene Therapy for a Prototypical Lysosomal Storage Disease (GSD-II) Is Critically Dependent on Vector Dose, Transgene Promoter, and the Tissues Targeted for Vector Transduction

Persistence of an [E1-, polymerase-] adenovirus vector despite transduction of a neoantigen into immune-competent mice

Multiply deleted [E1, polymerase-, and pTP-] adenovirus vector persists despite deletion of the preterminal protein

1012. Ad Interactions with the Complement Systems of Humans and Mice

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