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Reducing the lipase LIPE in mutant α-synuclein mice improves Parkinson-like deficits and reveals sex differences in fatty acid metabolism

Neurobiology of disease, 2024-09, Vol.199, p.106593, Article 106593

2024

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The APP family of proteins: similarities and differences

Biochemical Society transactions, 2007-04, Vol.35 (Pt 2), p.416

2007

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Folding proteins in fatal ways

Nature (London), 2003-12, Vol.426 (6968), p.900-904

Selkoe, Dennis J

2003

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Sequence of Deposition of Heterogeneous Amyloid β-Peptides and APO E in Down Syndrome: Implications for Initial Events in Amyloid Plaque Formation

Neurobiology of disease, 1996-02, Vol.3 (1), p.16-32

1996

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A Is for Amyloid

The journal of prevention of Alzheimer's disease, 2020, Vol.7 (3), p.140-141

Selkoe, D. J.

2020

Volltextzugriff (PDF)

Mass spectrometry of purified amyloid beta protein in Alzheimer's disease

The Journal of biological chemistry, 1992-08, Vol.267 (24), p.17082-17086

1992

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Activation of protein kinase C inhibits cellular production of the amyloid beta-protein

The Journal of biological chemistry, 1993-11, Vol.268 (31), p.22959-22962

1993

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Naturally occurring free D-aspartate is a nuclear component of cells in the mammalian hypothalamo-neurohypophyseal system

Neuroscience, 2002-01, Vol.109 (1), p.1-4

2002

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Developing Preventive Therapies for Chronic Diseases: Lessons Learned from Alzheimer's Disease

Nutrition reviews, 2007-12, Vol.65 (12), p.S239-S243

Selkoe, D.J

2007

Volltextzugriff (PDF)

Embryonic mosaic deletion of APP results in displaced Reelin-expressing cells in the cerebral cortex

Developmental biology, 2017-04, Vol.424 (2), p.138-146

2017

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P32 Phase III studies of crenezumab in early (prodromal-to-mild) Alzheimers disease (CREAD/CREAD2): Biomarker results

Clinical neurophysiology, 2020-04, Vol.131 (4), p.e194-e195

2020

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The Future of Anti-Amyloid Trials

The journal of prevention of Alzheimer's disease, 2020, Vol.7 (3), p.146-151

2020

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beta-Amyloid peptide and a 3-kDa fragment are derived by distinct cellular mechanisms

The Journal of biological chemistry, 1993-02, Vol.268 (5), p.3021-3024

1993

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Mutations associated with a locus for familial Alzheimer's disease result in alternative processing of amyloid beta-protein precursor

The Journal of biological chemistry, 1994-07, Vol.269 (26), p.17741-17748

1994

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[P2.49]: Secreted amyloid beta and impaired neurotrophin transport: Potential mediators of abnormal cns development and neurodegeneration in down syndrome

International journal of developmental neuroscience, 2010-12, Vol.28 (8), p.703-704

2010

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Microtubule-associated protein tau is a major antigenic component of paired helical filaments in Alzheimer disease

Proceedings of the National Academy of Sciences - PNAS, 1986-06, Vol.83 (11), p.4044-4048

1986

Volltextzugriff (PDF)

Selective ectodomain phosphorylation and regulated cleavage of beta‐amyloid precursor protein

The EMBO journal, 1994-02, Vol.13 (3), p.534-542

1994

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β -amyloid Precursor Protein of Alzheimer Disease Occurs as 110- to 135-Kilodalton Membrane-Associated Proteins in Neural and Nonneural Tissues

Proceedings of the National Academy of Sciences - PNAS, 1988-10, Vol.85 (19), p.7341-7345

1988

Volltextzugriff (PDF)

Chapter 1 - The Complex Pathways to Mechanism-Based Therapeutics in Alzheimer’s Disease

Developing Therapeutics for Alzheimer’s Disease, 2016, p.1-22

Selkoe, D.J.

2016

Volltextzugriff (PDF)

Inhibition of Amyloid β-Protein Production in Neural Cells by the Serine Protease Inhibitor AEBSF

Neuron (Cambridge, Mass.), 1996-07, Vol.17 (1), p.171-179

1996

Volltextzugriff (PDF)

X-Ray Diffraction from Intraneuronal Paired Helical Filaments and Extraneuronal Amyloid Fibers in Alzheimer Disease Indicates Cross-β Conformation

Proceedings of the National Academy of Sciences - PNAS, 1986-01, Vol.83 (2), p.503-507

1986

Volltextzugriff (PDF)

Enhancer function and novel DNA binding protein activity in the near upstream βAPP gene promoter

Gene, 1999-05, Vol.232 (1), p.125-141

1999

Volltextzugriff (PDF)

The Complex Pathways to Mechanism-Based Therapeutics in Alzheimer's Disease

Developing Therapeutics for Alzheimer's Disease, 2016, p.1-22

2016

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697 Degradation of amyloid β-protein by a secreted metallo-protease in neural and non-neural cells

Neurobiology of aging, 1996, Vol.17 (4), p.S173-S173

1996

Volltextzugriff (PDF)

762 Aβ is transported on lipoproteins and albumin in human biological fluids

Neurobiology of aging, 1996, Vol.17 (4), p.S189-S189

1996

Volltextzugriff (PDF)

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Reducing the lipase LIPE in mutant α-synuclein mice improves Parkinson-like deficits and reveals sex differences in fatty acid metabolism

The APP family of proteins: similarities and differences

Folding proteins in fatal ways

Sequence of Deposition of Heterogeneous Amyloid β-Peptides and APO E in Down Syndrome: Implications for Initial Events in Amyloid Plaque Formation

A Is for Amyloid

Mass spectrometry of purified amyloid beta protein in Alzheimer's disease

Activation of protein kinase C inhibits cellular production of the amyloid beta-protein

Naturally occurring free D-aspartate is a nuclear component of cells in the mammalian hypothalamo-neurohypophyseal system

Developing Preventive Therapies for Chronic Diseases: Lessons Learned from Alzheimer's Disease

Embryonic mosaic deletion of APP results in displaced Reelin-expressing cells in the cerebral cortex

P32 Phase III studies of crenezumab in early (prodromal-to-mild) Alzheimers disease (CREAD/CREAD2): Biomarker results

The Future of Anti-Amyloid Trials

beta-Amyloid peptide and a 3-kDa fragment are derived by distinct cellular mechanisms

Mutations associated with a locus for familial Alzheimer's disease result in alternative processing of amyloid beta-protein precursor

[P2.49]: Secreted amyloid beta and impaired neurotrophin transport: Potential mediators of abnormal cns development and neurodegeneration in down syndrome

Microtubule-associated protein tau is a major antigenic component of paired helical filaments in Alzheimer disease

Selective ectodomain phosphorylation and regulated cleavage of beta‐amyloid precursor protein

β -amyloid Precursor Protein of Alzheimer Disease Occurs as 110- to 135-Kilodalton Membrane-Associated Proteins in Neural and Nonneural Tissues

Chapter 1 - The Complex Pathways to Mechanism-Based Therapeutics in Alzheimer’s Disease

Inhibition of Amyloid β-Protein Production in Neural Cells by the Serine Protease Inhibitor AEBSF

X-Ray Diffraction from Intraneuronal Paired Helical Filaments and Extraneuronal Amyloid Fibers in Alzheimer Disease Indicates Cross-β Conformation

Enhancer function and novel DNA binding protein activity in the near upstream βAPP gene promoter

The Complex Pathways to Mechanism-Based Therapeutics in Alzheimer's Disease

697 Degradation of amyloid β-protein by a secreted metallo-protease in neural and non-neural cells

762 Aβ is transported on lipoproteins and albumin in human biological fluids

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