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Tumor Cell Infiltration into the Brain in Glioblastoma: From Mechanisms to Clinical Perspectives

Cancers, 2022-01, Vol.14 (2), p.443

2022

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Identification of SERPINE1 as a Regulator of Glioblastoma Cell Dispersal with Transcriptome Profiling

Cancers, 2019-10, Vol.11 (11), p.1651

2019

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Integrating leiomyoma genetics, epigenomics, and single-cell transcriptomics reveals causal genetic variants, genes, and cell types

Nature communications, 2024-02, Vol.15 (1), p.1169-1169, Article 1169

2024

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The pro-apoptotic Bcl-2 family member Harakiri (HRK) induces cell death in glioblastoma multiforme

Cell death discovery, 2019-02, Vol.5 (1), p.64-64, Article 64

2019

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Engineered MED12 mutations drive leiomyoma-like transcriptional and metabolic programs by altering the 3D genome compartmentalization

Nature communications, 2023-07, Vol.14 (1), p.4057-4057, Article 4057

2023

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Depletion of lamins B1 and B2 promotes chromatin mobility and induces differential gene expression by a mesoscale-motion-dependent mechanism

Genome Biology, 2024-03, Vol.25 (1), p.77-42, Article 77

2024

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Drug Repositioning Screen on a New Primary Cell Line Identifies Potent Therapeutics for Glioblastoma

Frontiers in neuroscience, 2020-12, Vol.14, p.578316-578316

2020

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Parameters Influencing Gene Delivery Efficiency of PEGylated Chitosan Nanoparticles: Experimental and Modeling Approach

Advanced NanoBiomed Research (Online), 2022-01, Vol.2 (1), p.n/a

2022

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Epigenetic-focused CRISPR/Cas9 screen identifies (absent, small, or homeotic)2-like protein (ASH2L) as a regulator of glioblastoma cell survival

Cell communication and signaling, 2023-11, Vol.21 (1), p.1-328, Article 328

2023

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Gelatin Methacryloyl Hydrogels in the Absence of a Crosslinker as 3D Glioblastoma Multiforme (GBM)‐Mimetic Microenvironment

Macromolecular bioscience, 2018-03, Vol.18 (3), p.n/a

2018

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BMET-29ADDRESSING THE MOLECULAR MECHANISMS OF GBM CELL DISPERSAL

Neuro-oncology (Charlottesville, Va.), 2015-11, Vol.17 (suppl 5), p.v51.3-v51

2015

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The fungal metabolite chaetocin is a sensitizer for pro-apoptotic therapies in glioblastoma

Cell death & disease, 2019-11, Vol.10 (12), p.894-20, Article 894

2019

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EXTH-10. COMBINATION OF EPIGENETIC ENZYME INHIBITORS, GSK-J4 AND BELINOSTAT, REVEALS HIGH EFFICACY IN IDH1 MUTANT GLIOMAS

Neuro-oncology (Charlottesville, Va.), 2020-11, Vol.22 (Supplement_2), p.ii88-ii89

2020

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ANGI-08. IDENTIFICATION OF NOVEL MOLECULAR PLAYERS OF GBM CELL DISPERSAL THROUGH AN IN VITRO PROFILING APPROACH

Neuro-oncology (Charlottesville, Va.), 2016-11, Vol.18 (suppl_6), p.vi16-vi16

2016

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Editorial Expression of Concern: Generation of TRAIL-resistant cell line models reveals distinct adaptive mechanisms for acquired resistance and re-sensitization

Oncogene, 2024-08, Vol.43 (34), p.2612-2612

2024

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DDRE-03. IDH1-MUTANT GBM CELLS ARE HIGHLY SENSITIVE TO COMBINATION OF KDM6A/B AND HDAC INHIBITORS

Neuro-oncology advances, 2021-03, Vol.3 (Supplement_1), p.i6-i7

2021

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Generation of TRAIL-resistant cell line models reveals distinct adaptive mechanisms for acquired resistance and re-sensitization

Oncogene, 2021-05, Vol.40 (18), p.3201-3216

2021

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TRAIL-conjugated silver nanoparticles sensitize glioblastoma cells to TRAIL by regulating CHK1 in the DNA repair pathway

Neurological research (New York), 2020-12, Vol.42 (12), p.1061-1069

2020

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VP16.16: Case report of Chiari network with prenatal diagnosis: can Chiari Network be the cause of hypoplastic right heart syndrome?

Ultrasound in obstetrics & gynecology, 2020-10, Vol.56 (S1), p.123-123

2020

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Determine the impact of hybrid capture-based comprehensive genomic profiling (CGP) on the treatment strategies in patients with solid tumors: A national, multicenter, retrospective study

Journal of clinical oncology, 2020-05, Vol.38 (15_suppl), p.e13646-e13646

2020

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A MULTIDIMENSIONAL EVALUATION OF THE USE OF SOCIAL NETWORKS IN EDUCATIONAL PROCESSES

European Journal of Education Studies, 2022-06, Vol.9 (6)

2022

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TURKISH AS THE HERITAGE LANGUAGE AMONG IMMIGRANT COMMUNITIES IN FRANCE

Uluslararası Halkbilimi Araştırmaları Dergisi, 2023-11, Vol.6 (2), p.284-298

2023

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A Quantisation of Cognitive Learning Process by Computer Graphics-Games: Towards More Efficient Learning Models

OAlib (Online), 2016, Vol.3 (1), p.1-12

2016

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The Impact of Hybrid Capture-Based Comprehensive Genomic Profiling on Treatment Strategies in Patients with Solid Tumors

Journal of oncological science, 2022, Vol.8 (2), p.87-93

2022

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A Research About One City Monopoly Phenomenon in Turkey

Sosyoekonomi, 2022-01, Vol.30 (51), p.53-70

2022

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Tumor Cell Infiltration into the Brain in Glioblastoma: From Mechanisms to Clinical Perspectives

Identification of SERPINE1 as a Regulator of Glioblastoma Cell Dispersal with Transcriptome Profiling

Integrating leiomyoma genetics, epigenomics, and single-cell transcriptomics reveals causal genetic variants, genes, and cell types

The pro-apoptotic Bcl-2 family member Harakiri (HRK) induces cell death in glioblastoma multiforme

Engineered MED12 mutations drive leiomyoma-like transcriptional and metabolic programs by altering the 3D genome compartmentalization

Depletion of lamins B1 and B2 promotes chromatin mobility and induces differential gene expression by a mesoscale-motion-dependent mechanism

Drug Repositioning Screen on a New Primary Cell Line Identifies Potent Therapeutics for Glioblastoma

Parameters Influencing Gene Delivery Efficiency of PEGylated Chitosan Nanoparticles: Experimental and Modeling Approach

Epigenetic-focused CRISPR/Cas9 screen identifies (absent, small, or homeotic)2-like protein (ASH2L) as a regulator of glioblastoma cell survival

Gelatin Methacryloyl Hydrogels in the Absence of a Crosslinker as 3D Glioblastoma Multiforme (GBM)‐Mimetic Microenvironment

BMET-29ADDRESSING THE MOLECULAR MECHANISMS OF GBM CELL DISPERSAL

The fungal metabolite chaetocin is a sensitizer for pro-apoptotic therapies in glioblastoma

EXTH-10. COMBINATION OF EPIGENETIC ENZYME INHIBITORS, GSK-J4 AND BELINOSTAT, REVEALS HIGH EFFICACY IN IDH1 MUTANT GLIOMAS

ANGI-08. IDENTIFICATION OF NOVEL MOLECULAR PLAYERS OF GBM CELL DISPERSAL THROUGH AN IN VITRO PROFILING APPROACH

Editorial Expression of Concern: Generation of TRAIL-resistant cell line models reveals distinct adaptive mechanisms for acquired resistance and re-sensitization

DDRE-03. IDH1-MUTANT GBM CELLS ARE HIGHLY SENSITIVE TO COMBINATION OF KDM6A/B AND HDAC INHIBITORS

Generation of TRAIL-resistant cell line models reveals distinct adaptive mechanisms for acquired resistance and re-sensitization

TRAIL-conjugated silver nanoparticles sensitize glioblastoma cells to TRAIL by regulating CHK1 in the DNA repair pathway

VP16.16: Case report of Chiari network with prenatal diagnosis: can Chiari Network be the cause of hypoplastic right heart syndrome?

Determine the impact of hybrid capture-based comprehensive genomic profiling (CGP) on the treatment strategies in patients with solid tumors: A national, multicenter, retrospective study

A MULTIDIMENSIONAL EVALUATION OF THE USE OF SOCIAL NETWORKS IN EDUCATIONAL PROCESSES

TURKISH AS THE HERITAGE LANGUAGE AMONG IMMIGRANT COMMUNITIES IN FRANCE

A Quantisation of Cognitive Learning Process by Computer Graphics-Games: Towards More Efficient Learning Models

The Impact of Hybrid Capture-Based Comprehensive Genomic Profiling on Treatment Strategies in Patients with Solid Tumors

A Research About One City Monopoly Phenomenon in Turkey

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