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Neural circuits for pain: Recent advances and current views

Science (American Association for the Advancement of Science), 2016-11, Vol.354 (6312), p.578-584

2016

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Restoration of Hearing in the VGLUT3 Knockout Mouse Using Virally Mediated Gene Therapy

Neuron (Cambridge, Mass.), 2012-07, Vol.75 (2), p.283-293

2012

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Uncovering the Cells and Circuits of Touch in Normal and Pathological Settings

Neuron (Cambridge, Mass.), 2018-10, Vol.100 (2), p.349-360

2018

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Making sense out of spinal cord somatosensory development

Development (Cambridge), 2016-10, Vol.143 (19), p.3434-3448

2016

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Striatal cholinergic interneurons Drive GABA release from dopamine terminals

Neuron (Cambridge, Mass.), 2014-04, Vol.82 (1), p.63-70

2014

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Dorsal Horn Circuits for Persistent Mechanical Pain

Neuron (Cambridge, Mass.), 2015-08, Vol.87 (4), p.797-812

2015

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An Unconventional Glutamatergic Circuit in the Retina Formed by vGluT3 Amacrine Cells

Neuron (Cambridge, Mass.), 2014-11, Vol.84 (4), p.708-715

2014

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A Non-canonical Pathway from Cochlea to Brain Signals Tissue-Damaging Noise

Current biology, 2015-03, Vol.25 (5), p.606-612

2015

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A Functional Topographic Map for Spinal Sensorimotor Reflexes

Neuron (Cambridge, Mass.), 2021-01, Vol.109 (1), p.91-104.e5

2021

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Mechanical Allodynia Circuitry in the Dorsal Horn Is Defined by the Nature of the Injury

Neuron (Cambridge, Mass.), 2021-01, Vol.109 (1), p.73-90.e7

2021

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Illuminating the Gap: Neuronal Cross-Talk within Sensory Ganglia and Persistent Pain

Neuron (Cambridge, Mass.), 2016-09, Vol.91 (5), p.950-951

Seal, Rebecca P.

2016

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A glutamatergic reward input from the dorsal raphe to ventral tegmental area dopamine neurons

Nature communications, 2014-11, Vol.5 (1), p.5390-5390, Article 5390

2014

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Injury-induced mechanical hypersensitivity requires C-low threshold mechanoreceptors

Nature (London), 2009-12, Vol.462 (7273), p.651-655

2009

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Cholinergic interneurons mediate fast VGluT3-dependent glutamatergic transmission in the striatum

PloS one, 2011-04, Vol.6 (4), p.e19155-e19155

2011

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Do the distinct synaptic properties of VGLUTs shape pain?

Neurochemistry international, 2016-09, Vol.98, p.82-88

Seal, Rebecca P.

2016

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Layer- and cell type-selective co-transmission by a basal forebrain cholinergic projection to the olfactory bulb

Nature communications, 2017-09, Vol.8 (1), p.652-9, Article 652

2017

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Outer Hair Cell Glutamate Signaling through Type II Spiral Ganglion Afferents Activates Neurons in the Cochlear Nucleus in Response to Nondamaging Sounds

The Journal of neuroscience, 2021-03, Vol.41 (13), p.2930-2943

2021

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Sensorineural Deafness and Seizures in Mice Lacking Vesicular Glutamate Transporter 3

Neuron (Cambridge, Mass.), 2008-01, Vol.57 (2), p.263-275

2008

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VGLUTs define subsets of excitatory neurons and suggest novel roles for glutamate

Trends in neurosciences (Regular ed.), 2004-02, Vol.27 (2), p.98-103

2004

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A population of glomerular glutamatergic neurons controls sensory information transfer in the mouse olfactory bulb

Nature communications, 2014-05, Vol.5 (1), p.3791-3791, Article 3791

2014

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Loss of VGLUT3 Produces Circadian-Dependent Hyperdopaminergia and Ameliorates Motor Dysfunction and l-Dopa-Mediated Dyskinesias in a Model of Parkinson's Disease

The Journal of neuroscience, 2015-11, Vol.35 (45), p.14983-14999

2015

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Genetic targeting and physiological features of VGLUT3+ amacrine cells

Visual neuroscience, 2011-09, Vol.28 (5), p.381-392

2011

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Synaptic and Extrasynaptic Factors Governing Glutamatergic Retinal Waves

Neuron (Cambridge, Mass.), 2009-04, Vol.62 (2), p.230-241

2009

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Expression of the vesicular glutamate transporters during development indicates the widespread corelease of multiple neurotransmitters

Journal of comparative neurology (1911), 2004-12, Vol.480 (3), p.264-280

2004

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Alleviation of neuropathic pain with neuropeptide Y requires spinal Npy1r interneurons that coexpress Grp

JCI insight, 2023-11, Vol.8 (22)

2023

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Neural circuits for pain: Recent advances and current views

Restoration of Hearing in the VGLUT3 Knockout Mouse Using Virally Mediated Gene Therapy

Uncovering the Cells and Circuits of Touch in Normal and Pathological Settings

Making sense out of spinal cord somatosensory development

Striatal cholinergic interneurons Drive GABA release from dopamine terminals

Dorsal Horn Circuits for Persistent Mechanical Pain

An Unconventional Glutamatergic Circuit in the Retina Formed by vGluT3 Amacrine Cells

A Non-canonical Pathway from Cochlea to Brain Signals Tissue-Damaging Noise

A Functional Topographic Map for Spinal Sensorimotor Reflexes

Mechanical Allodynia Circuitry in the Dorsal Horn Is Defined by the Nature of the Injury

Illuminating the Gap: Neuronal Cross-Talk within Sensory Ganglia and Persistent Pain

A glutamatergic reward input from the dorsal raphe to ventral tegmental area dopamine neurons

Injury-induced mechanical hypersensitivity requires C-low threshold mechanoreceptors

Cholinergic interneurons mediate fast VGluT3-dependent glutamatergic transmission in the striatum

Do the distinct synaptic properties of VGLUTs shape pain?

Layer- and cell type-selective co-transmission by a basal forebrain cholinergic projection to the olfactory bulb

Outer Hair Cell Glutamate Signaling through Type II Spiral Ganglion Afferents Activates Neurons in the Cochlear Nucleus in Response to Nondamaging Sounds

Sensorineural Deafness and Seizures in Mice Lacking Vesicular Glutamate Transporter 3

VGLUTs define subsets of excitatory neurons and suggest novel roles for glutamate

A population of glomerular glutamatergic neurons controls sensory information transfer in the mouse olfactory bulb

Loss of VGLUT3 Produces Circadian-Dependent Hyperdopaminergia and Ameliorates Motor Dysfunction and l-Dopa-Mediated Dyskinesias in a Model of Parkinson's Disease

Genetic targeting and physiological features of VGLUT3+ amacrine cells

Synaptic and Extrasynaptic Factors Governing Glutamatergic Retinal Waves

Expression of the vesicular glutamate transporters during development indicates the widespread corelease of multiple neurotransmitters

Alleviation of neuropathic pain with neuropeptide Y requires spinal Npy1r interneurons that coexpress Grp

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