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Targeting proteins for degradation

Nature Chemical Biology, 2009-11, Vol.5 (11), p.815-822

2009

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A Three-part Signal Governs Differential Processing of Gli1 and Gli3 Proteins by the Proteasome

The Journal of biological chemistry, 2011-11, Vol.286 (45), p.39051-39058

2011

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Sequence- and Species-Dependence of Proteasomal Processivity

ACS chemical biology, 2012-08, Vol.7 (8), p.1444-1453

2012

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Erratum: Targeting proteins for degradation

Nature chemical biology, 2009-12, Vol.5 (12), p.954-954

2009

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Making It Easier to Regulate Protein Stability

Chemistry & biology, 2010-09, Vol.17 (9), p.917-918

2010

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APE2 Promotes AID-Dependent Somatic Hypermutation in Primary B Cell Cultures That Is Suppressed by APE1

The Journal of immunology (1950), 2023-06, Vol.210 (11), p.1804-1814

2023

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Nbs1 ChIP-Seq Identifies Off-Target DNA Double-Strand Breaks Induced by AID in Activated Splenic B Cells

PLoS genetics, 2015-08, Vol.11 (8), p.e1005438-e1005438

2015

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Differential expression of APE1 and APE2 in germinal centers promotes error-prone repair and A:T mutations during somatic hypermutation

Proceedings of the National Academy of Sciences - PNAS, 2014-06, Vol.111 (25), p.9217-9222

2014

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Proposed Key Characteristics of Male Reproductive Toxicants as an Approach for Organizing and Evaluating Mechanistic Evidence in Human Health Hazard Assessments

Environmental health perspectives, 2019-06, Vol.127 (6), p.65001

2019

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Individual substitution mutations in the AID C terminus that ablate IgH class switch recombination

PloS one, 2015-08, Vol.10 (8), p.e0134397

2015

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APE1- and APE2-dependent DNA breaks in immunoglobulin class switch recombination

The Journal of experimental medicine, 2007-11, Vol.204 (12), p.3017-3026

2007

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Activation-Induced Cytidine Deaminase-Dependent DNA Breaks in Class Switch Recombination Occur during G1 Phase of the Cell Cycle and Depend upon Mismatch Repair

The Journal of immunology (1950), 2007-11, Vol.179 (9), p.6064-6071

2007

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Apurinic/apyrimidinic endonuclease 2 is necessary for normal B cell development and recovery of lymphoid progenitors after chemotherapeutic challenge

The Journal of immunology (1950), 2011-02, Vol.186 (4), p.1943-1950

2011

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AID Binds Cooperatively with UNG and Msh2-Msh6 to Ig Switch Regions Dependent upon the AID C Terminus

The Journal of immunology (1950), 2011-09, Vol.187 (5), p.2464-2475

2011

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Inducible DNA breaks in Ig S regions are dependent on AID and UNG

The Journal of experimental medicine, 2005-08, Vol.202 (4), p.561-568

2005

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Mismatch repair proteins and AID activity are required for the dominant negative function of C-terminally deleted AID in class switching

The Journal of immunology (1950), 2014-08, Vol.193 (3), p.1440-1450

2014

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The DNA glycosylases Ogg1 and Nth1 do not contribute to Ig class switching in activated mouse splenic B cells

PloS one, 2012-04, Vol.7 (4), p.e36061-e36061

2012

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Apurinic/apyrimidinic endonuclease 2 regulates the expansion of germinal centers by protecting against activation-induced cytidine deaminase-independent DNA damage in B cells

The Journal of immunology (1950), 2014-07, Vol.193 (2), p.931-939

2014

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Pan-cancer analysis of advanced patient tumors reveals interactions between therapy and genomic landscapes

Nature cancer, 2020-04, Vol.1 (4), p.452-468

2020

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ATM increases activation-induced cytidine deaminase activity at downstream S regions during class-switch recombination

The Journal of immunology (1950), 2014-05, Vol.192 (10), p.4887-4896

2014

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DNA polymerases β and λ do not directly affect Ig variable region somatic hypermutation although their absence reduces the frequency of mutations

DNA repair, 2013-12, Vol.12 (12), p.1087-1093

2013

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Nbs1 ChIP-Seq identifies off-target DNA double-strand breaks induced by aid in activated saplenic B cells

PLoS genetics, 2015-08

2015

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Nbs1 ChIP-Seq Identifies Off-Target DNA Double-Strand Breaks Induced by AID in Activated Splenic B Cells: e1005438

PLoS genetics, 2015-08, Vol.11 (8)

2015

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p53 represses class switch recombination to IgG2a through its antioxidant function

The Journal of immunology (1950), 2010-06, Vol.184 (11), p.6177-6187

2010

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Cells, Cultured
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Cytidine Deaminase - Genetics
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Immunoglobulin Class Switching - Genetics
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B-Lymphocytes - Metabolism
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Dna Breaks, Double-Stranded
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Targeting proteins for degradation

A Three-part Signal Governs Differential Processing of Gli1 and Gli3 Proteins by the Proteasome

Sequence- and Species-Dependence of Proteasomal Processivity

Erratum: Targeting proteins for degradation

Making It Easier to Regulate Protein Stability

APE2 Promotes AID-Dependent Somatic Hypermutation in Primary B Cell Cultures That Is Suppressed by APE1

Nbs1 ChIP-Seq Identifies Off-Target DNA Double-Strand Breaks Induced by AID in Activated Splenic B Cells

Differential expression of APE1 and APE2 in germinal centers promotes error-prone repair and A:T mutations during somatic hypermutation

Proposed Key Characteristics of Male Reproductive Toxicants as an Approach for Organizing and Evaluating Mechanistic Evidence in Human Health Hazard Assessments

Individual substitution mutations in the AID C terminus that ablate IgH class switch recombination

APE1- and APE2-dependent DNA breaks in immunoglobulin class switch recombination

Activation-Induced Cytidine Deaminase-Dependent DNA Breaks in Class Switch Recombination Occur during G1 Phase of the Cell Cycle and Depend upon Mismatch Repair

Apurinic/apyrimidinic endonuclease 2 is necessary for normal B cell development and recovery of lymphoid progenitors after chemotherapeutic challenge

AID Binds Cooperatively with UNG and Msh2-Msh6 to Ig Switch Regions Dependent upon the AID C Terminus

Inducible DNA breaks in Ig S regions are dependent on AID and UNG

Mismatch repair proteins and AID activity are required for the dominant negative function of C-terminally deleted AID in class switching

The DNA glycosylases Ogg1 and Nth1 do not contribute to Ig class switching in activated mouse splenic B cells

Apurinic/apyrimidinic endonuclease 2 regulates the expansion of germinal centers by protecting against activation-induced cytidine deaminase-independent DNA damage in B cells

Pan-cancer analysis of advanced patient tumors reveals interactions between therapy and genomic landscapes

ATM increases activation-induced cytidine deaminase activity at downstream S regions during class-switch recombination

DNA polymerases β and λ do not directly affect Ig variable region somatic hypermutation although their absence reduces the frequency of mutations

Long-read sequencing of an advanced cancer cohort resolves rearrangements, unravels haplotypes, and reveals methylation landscapes

Nbs1 ChIP-Seq identifies off-target DNA double-strand breaks induced by aid in activated saplenic B cells

Nbs1 ChIP-Seq Identifies Off-Target DNA Double-Strand Breaks Induced by AID in Activated Splenic B Cells: e1005438

p53 represses class switch recombination to IgG2a through its antioxidant function

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