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IgH chain class switch recombination: mechanism and regulation

The Journal of immunology (1950), 2014-12, Vol.193 (11), p.5370-5378

2014

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Mechanism and regulation of class switch recombination

Annual review of immunology, 2008-01, Vol.26 (1), p.261-292

2008

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Differential expression of APE1 and APE2 in germinal centers promotes error-prone repair and A:T mutations during somatic hypermutation

Proceedings of the National Academy of Sciences - PNAS, 2014-06, Vol.111 (25), p.9217-9222

2014

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Nbs1 ChIP-Seq Identifies Off-Target DNA Double-Strand Breaks Induced by AID in Activated Splenic B Cells

PLoS genetics, 2015-08, Vol.11 (8), p.e1005438-e1005438

2015

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APE1- and APE2-dependent DNA breaks in immunoglobulin class switch recombination

The Journal of experimental medicine, 2007-11, Vol.204 (12), p.3017-3026

2007

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Activation-Induced Cytidine Deaminase-Dependent DNA Breaks in Class Switch Recombination Occur during G1 Phase of the Cell Cycle and Depend upon Mismatch Repair

The Journal of immunology (1950), 2007-11, Vol.179 (9), p.6064-6071

2007

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Mismatch repair converts AID-instigated nicks to double-strand breaks for antibody class-switch recombination

Trends in genetics, 2006, Vol.22 (1), p.23-28

2006

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The DNA Damage Response Regulates RAG1/2 Expression in Pre-B Cells through ATM-FOXO1 Signaling

The Journal of immunology (1950), 2016-10, Vol.197 (7), p.2918-2929

2016

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Individual substitution mutations in the AID C terminus that ablate IgH class switch recombination

PloS one, 2015-08, Vol.10 (8), p.e0134397

2015

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The roles of APE1, APE2, DNA polymerase beta and mismatch repair in creating S region DNA breaks during antibody class switch

Philosophical transactions of the Royal Society of London. Series B. Biological sciences, 2009-03, Vol.364 (1517), p.645-652

2009

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Mismatch repair proteins and AID activity are required for the dominant negative function of C-terminally deleted AID in class switching

The Journal of immunology (1950), 2014-08, Vol.193 (3), p.1440-1450

2014

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APE2 Promotes AID-Dependent Somatic Hypermutation in Primary B Cell Cultures That Is Suppressed by APE1

The Journal of immunology (1950), 2023-06, Vol.210 (11), p.1804-1814

2023

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Role for mismatch repair proteins Msh2, Mlh1, and Pms2 in immunoglobulin class switching shown by sequence analysis of recombination junctions

The Journal of experimental medicine, 2002-02, Vol.195 (3), p.367-373

2002

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Apurinic/apyrimidinic endonuclease 2 is necessary for normal B cell development and recovery of lymphoid progenitors after chemotherapeutic challenge

The Journal of immunology (1950), 2011-02, Vol.186 (4), p.1943-1950

2011

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The DNA glycosylases Ogg1 and Nth1 do not contribute to Ig class switching in activated mouse splenic B cells

PloS one, 2012-04, Vol.7 (4), p.e36061-e36061

2012

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The role of Apex2 in class-switch recombination of immunoglobulin genes

International immunology, 2010-03, Vol.22 (3), p.213-213

2010

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Mlh1 can function in antibody class switch recombination independently of Msh2

The Journal of experimental medicine, 2003-05, Vol.197 (10), p.1377-1383

2003

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Response to comment on "IgH chain class switch recombination: mechanism and regulation"

The Journal of immunology (1950), 2015-03, Vol.194 (5), p.2040-2040

2015

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AID Binds Cooperatively with UNG and Msh2-Msh6 to Ig Switch Regions Dependent upon the AID C Terminus

The Journal of immunology (1950), 2011-09, Vol.187 (5), p.2464-2475

2011

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Deletion of the nucleotide excision repair gene Ercc1 reduces immunoglobulin class switching and alters mutations near switch recombination junctions

The Journal of experimental medicine, 2004-08, Vol.200 (3), p.321-330

2004

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Inducible DNA breaks in Ig S regions are dependent on AID and UNG

The Journal of experimental medicine, 2005-08, Vol.202 (4), p.561-568

2005

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Apurinic/apyrimidinic endonuclease 2 regulates the expansion of germinal centers by protecting against activation-induced cytidine deaminase-independent DNA damage in B cells

The Journal of immunology (1950), 2014-07, Vol.193 (2), p.931-939

2014

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ATM increases activation-induced cytidine deaminase activity at downstream S regions during class-switch recombination

The Journal of immunology (1950), 2014-05, Vol.192 (10), p.4887-4896

2014

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Class switch recombination efficiency and junction microhomology patterns in Msh2-, Mlh1-, and Exo1-deficient mice depend on the presence of mu switch region tandem repeats

The Journal of immunology (1950), 2009-07, Vol.183 (2), p.1222-1228

2009

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DNA polymerases β and λ do not directly affect Ig variable region somatic hypermutation although their absence reduces the frequency of mutations

DNA repair, 2013-12, Vol.12 (12), p.1087-1093

2013

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IgH chain class switch recombination: mechanism and regulation

Mechanism and regulation of class switch recombination

Differential expression of APE1 and APE2 in germinal centers promotes error-prone repair and A:T mutations during somatic hypermutation

Nbs1 ChIP-Seq Identifies Off-Target DNA Double-Strand Breaks Induced by AID in Activated Splenic B Cells

APE1- and APE2-dependent DNA breaks in immunoglobulin class switch recombination

Activation-Induced Cytidine Deaminase-Dependent DNA Breaks in Class Switch Recombination Occur during G1 Phase of the Cell Cycle and Depend upon Mismatch Repair

Mismatch repair converts AID-instigated nicks to double-strand breaks for antibody class-switch recombination

The DNA Damage Response Regulates RAG1/2 Expression in Pre-B Cells through ATM-FOXO1 Signaling

Individual substitution mutations in the AID C terminus that ablate IgH class switch recombination

The roles of APE1, APE2, DNA polymerase beta and mismatch repair in creating S region DNA breaks during antibody class switch

Mismatch repair proteins and AID activity are required for the dominant negative function of C-terminally deleted AID in class switching

APE2 Promotes AID-Dependent Somatic Hypermutation in Primary B Cell Cultures That Is Suppressed by APE1

Role for mismatch repair proteins Msh2, Mlh1, and Pms2 in immunoglobulin class switching shown by sequence analysis of recombination junctions

Apurinic/apyrimidinic endonuclease 2 is necessary for normal B cell development and recovery of lymphoid progenitors after chemotherapeutic challenge

The DNA glycosylases Ogg1 and Nth1 do not contribute to Ig class switching in activated mouse splenic B cells

The role of Apex2 in class-switch recombination of immunoglobulin genes

Mlh1 can function in antibody class switch recombination independently of Msh2

Response to comment on "IgH chain class switch recombination: mechanism and regulation"

AID Binds Cooperatively with UNG and Msh2-Msh6 to Ig Switch Regions Dependent upon the AID C Terminus

Deletion of the nucleotide excision repair gene Ercc1 reduces immunoglobulin class switching and alters mutations near switch recombination junctions

Inducible DNA breaks in Ig S regions are dependent on AID and UNG

Apurinic/apyrimidinic endonuclease 2 regulates the expansion of germinal centers by protecting against activation-induced cytidine deaminase-independent DNA damage in B cells

ATM increases activation-induced cytidine deaminase activity at downstream S regions during class-switch recombination

Class switch recombination efficiency and junction microhomology patterns in Msh2-, Mlh1-, and Exo1-deficient mice depend on the presence of mu switch region tandem repeats

DNA polymerases β and λ do not directly affect Ig variable region somatic hypermutation although their absence reduces the frequency of mutations

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