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MicroRNAs in epilepsy: pathophysiology and clinical utility

Lancet neurology, 2016-12, Vol.15 (13), p.1368-1376

2016

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dCas9-Based Scn1a Gene Activation Restores Inhibitory Interneuron Excitability and Attenuates Seizures in Dravet Syndrome Mice

Molecular therapy, 2020-01, Vol.28 (1), p.235-253

2020

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Chemical-genetic attenuation of focal neocortical seizures

Nature communications, 2014-05, Vol.5 (1), p.3847-3847, Article 3847

2014

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Optogenetic and potassium channel gene therapy in a rodent model of focal neocortical epilepsy

Science translational medicine, 2012-11, Vol.4 (161), p.161ra152

2012

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Gene Therapy for Neurological Disease: State of the Art and Opportunities for Next-generation Approaches

Neuroscience, 2022-05, Vol.490, p.309-314

2022

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Human ataxias: a genetic dissection of inositol triphosphate receptor ( ITPR1 )-dependent signaling

Trends in neurosciences (Regular ed.), 2010-05, Vol.33 (5), p.211-219

2010

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Altered Hippocampal-Prefrontal Neural Dynamics in Mouse Models of Down Syndrome

Cell reports (Cambridge), 2020-01, Vol.30 (4), p.1152-1163.e4

2020

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Channelopathies go above and beyond the channels

Neuropharmacology, 2018-04, Vol.132, p.1-2

Schorge, Stephanie

2018

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Maximum likelihood fitting of single channel NMDA activity with a mechanism composed of independent dimers of subunits

The Journal of physiology, 2005-12, Vol.569 (2), p.395-418

2005

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Alternative Splicing Modulates Inactivation of Type 1 Voltage-gated Sodium Channels by Toggling an Amino Acid in the First S3-S4 Linker

The Journal of biological chemistry, 2011-10, Vol.286 (42), p.36700-36708

2011

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Genetic Predictors of the Maximum Doses Patients Receive during Clinical Use of the Anti-Epileptic Drugs Carbamazepine and Phenytoin

Proceedings of the National Academy of Sciences - PNAS, 2005-04, Vol.102 (15), p.5507-5512

2005

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Tackling obstacles for gene therapy targeting neurons: disrupting perineural nets with hyaluronidase improves transduction

PloS one, 2013-01, Vol.8 (1), p.e53269-e53269

2013

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BICS01 Mediates Reversible Anti-seizure Effects in Brain Slice Models of Epilepsy

Frontiers in neurology, 2022-01, Vol.12, p.791608-791608

2022

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Semiology, clustering, periodicity and natural history of seizures in an experimental occipital cortical epilepsy model

Disease models & mechanisms, 2018-12, Vol.11 (12)

2018

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Central Nervous System Gene Therapy: Present Developments and Emerging Trends Accelerating Industry-Academia Pathways

Human gene therapy, 2022-09, Vol.33 (17-18), p.913-922

2022

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Episodic Ataxia Type 1: A Neuronal Potassium Channelopathy

Neurotherapeutics, 2007-04, Vol.4 (2), p.258-266

2007

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Changing channels in pain and epilepsy: Exploiting ion channel gene therapy for disorders of neuronal hyperexcitability

FEBS letters, 2015-06, Vol.589 (14), p.1620-1634

2015

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New immunohistochemical method for improved myotonia and chloride channel mutation diagnostics

Neurology, 2012-11, Vol.79 (22), p.2194-2200

2012

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Splice variants of Na(V)1.7 sodium channels have distinct β subunit-dependent biophysical properties

PloS one, 2012, Vol.7 (7), p.e41750-e41750

2012

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Recent advances in gene therapy for neurodevelopmental disorders with epilepsy

Journal of neurochemistry, 2021-04, Vol.157 (2), p.229-262

2021

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Erratum to “Changing channels in pain and epilepsy: Exploiting ion channel gene therapy for disorders of neuronal hyperexcitability” [FEBS Lett. 589 (2015) 1620–1634]

FEBS letters, 2015-08, Vol.589 (18), p.2429-2429

2015

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Episodic ataxia type 1 mutations differentially affect neuronal excitability and transmitter release

Disease models & mechanisms, 2009-11, Vol.2 (11-12), p.612-619

2009

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In vivo CRISPRa decreases seizures and rescues cognitive deficits in a rodent model of epilepsy

Brain (London, England : 1878), 2020-03, Vol.143 (3), p.891-905

2020

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Gene variant effects across sodium channelopathies predict function and guide precision therapy

Brain (London, England : 1878), 2022-12, Vol.145 (12), p.4275-4286

2022

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Epilepsy Gene Therapy Using an Engineered Potassium Channel

The Journal of neuroscience, 2019-04, Vol.39 (16), p.3159-3169

2019

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MicroRNAs in epilepsy: pathophysiology and clinical utility

dCas9-Based Scn1a Gene Activation Restores Inhibitory Interneuron Excitability and Attenuates Seizures in Dravet Syndrome Mice

Chemical-genetic attenuation of focal neocortical seizures

Optogenetic and potassium channel gene therapy in a rodent model of focal neocortical epilepsy

Gene Therapy for Neurological Disease: State of the Art and Opportunities for Next-generation Approaches

Human ataxias: a genetic dissection of inositol triphosphate receptor ( ITPR1 )-dependent signaling

Altered Hippocampal-Prefrontal Neural Dynamics in Mouse Models of Down Syndrome

Channelopathies go above and beyond the channels

Maximum likelihood fitting of single channel NMDA activity with a mechanism composed of independent dimers of subunits

Alternative Splicing Modulates Inactivation of Type 1 Voltage-gated Sodium Channels by Toggling an Amino Acid in the First S3-S4 Linker

Genetic Predictors of the Maximum Doses Patients Receive during Clinical Use of the Anti-Epileptic Drugs Carbamazepine and Phenytoin

Tackling obstacles for gene therapy targeting neurons: disrupting perineural nets with hyaluronidase improves transduction

BICS01 Mediates Reversible Anti-seizure Effects in Brain Slice Models of Epilepsy

Semiology, clustering, periodicity and natural history of seizures in an experimental occipital cortical epilepsy model

Central Nervous System Gene Therapy: Present Developments and Emerging Trends Accelerating Industry-Academia Pathways

Episodic Ataxia Type 1: A Neuronal Potassium Channelopathy

Changing channels in pain and epilepsy: Exploiting ion channel gene therapy for disorders of neuronal hyperexcitability

New immunohistochemical method for improved myotonia and chloride channel mutation diagnostics

Splice variants of Na(V)1.7 sodium channels have distinct β subunit-dependent biophysical properties

Recent advances in gene therapy for neurodevelopmental disorders with epilepsy

Erratum to “Changing channels in pain and epilepsy: Exploiting ion channel gene therapy for disorders of neuronal hyperexcitability” [FEBS Lett. 589 (2015) 1620–1634]

Episodic ataxia type 1 mutations differentially affect neuronal excitability and transmitter release

In vivo CRISPRa decreases seizures and rescues cognitive deficits in a rodent model of epilepsy

Gene variant effects across sodium channelopathies predict function and guide precision therapy

Epilepsy Gene Therapy Using an Engineered Potassium Channel

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