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Sphingolipids in viral infection

Biological chemistry, 2015-06, Vol.396 (6-7), p.585-595

2015

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Sphingosine 1-phosphate metabolism and insulin signaling

Cellular signalling, 2021-06, Vol.82, p.109959, Article 109959

2021

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DC-SIGN mediated sphingomyelinase-activation and ceramide generation is essential for enhancement of viral uptake in dendritic cells

PLoS pathogens, 2011-02, Vol.7 (2), p.e1001290-e1001290

2011

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Measles virus nucleocapsid transport to the plasma membrane requires stable expression and surface accumulation of the viral matrix protein

Cellular microbiology, 2007-05, Vol.9 (5), p.1203-1214

2007

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Differential activation of acid sphingomyelinase and ceramide release determines invasiveness of Neisseria meningitidis into brain endothelial cells

PLoS pathogens, 2014-06, Vol.10 (6), p.e1004160

2014

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CD9 Clustering and Formation of Microvilli Zippers Between Contacting Cells Regulates Virus-Induced Cell Fusion

Traffic (Copenhagen, Denmark), 2008-06, Vol.9 (6), p.924-935

2008

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Human endogenous retrovirus envelope proteins target dendritic cells to suppress T‐cell activation

European journal of immunology, 2015-06, Vol.45 (6), p.1748-1759

2015

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Membrane dynamics and interactions in measles virus dendritic cell infections

Cellular microbiology, 2013-02, Vol.15 (2), p.161-169

2013

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Hemagglutinin Protein of Wild-Type Measles Virus Activates Toll-Like Receptor 2 Signaling

Journal of Virology, 2002-09, Vol.76 (17), p.8729-8736

2002

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Measles Virus Transmission from Dendritic Cells to T Cells: Formation of Synapse-Like Interfaces Concentrating Viral and Cellular Components

Journal of Virology, 2012-09, Vol.86 (18), p.9773-9781

2012

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Azido-Ceramides, a Tool to Analyse SARS-CoV-2 Replication and Inhibition-SARS-CoV-2 Is Inhibited by Ceramides

International journal of molecular sciences, 2023-04, Vol.24 (8), p.7281

2023

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Neutral sphingomyelinase in physiological and measles virus induced T cell suppression

PLoS pathogens, 2014-12, Vol.10 (12), p.e1004574-e1004574

2014

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Dynamic changes in the proximitome of neutral sphingomyelinase-2 (nSMase2) in TNFα stimulated Jurkat cells

Frontiers in immunology, 2024-07, Vol.15, p.1435701

2024

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The Role of Sphingomyelin Breakdown in Measles Virus Immunmodulation

Cellular physiology and biochemistry, 2014-01, Vol.34 (1), p.20-26

2014

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Editorial: Sphingolipids in Infection Control

Frontiers in cell and developmental biology, 2021-06, Vol.9, p.697290-697290

2021

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The Activity of the Neutral Sphingomyelinase Is Important in T Cell Recruitment and Directional Migration

Frontiers in immunology, 2017-08, Vol.8, p.1007-1007

2017

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Sphingomyelin Breakdown in T Cells: Role of Membrane Compartmentalization in T Cell Signaling and Interference by a Pathogen

Frontiers in cell and developmental biology, 2019-08, Vol.7, p.152-152

2019

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Measles Virus Infection Fosters Dendritic Cell Motility in a 3D Environment to Enhance Transmission to Target Cells in the Respiratory Epithelium

Frontiers in immunology, 2019-06, Vol.10, p.1294-1294

2019

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Neutral sphingomyelinase 2 is a key factor for PorB‐dependent invasion of Neisseria gonorrhoeae

Cellular microbiology, 2015-02, Vol.17 (2), p.241-253

2015

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Sphingolipids: Effectors and Achilles Heals in Viral Infections?

Cells (Basel, Switzerland), 2021-08, Vol.10 (9), p.2175

2021

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The Acid Ceramidase Is a SARS-CoV-2 Host Factor

Cells (Basel, Switzerland), 2022-08, Vol.11 (16), p.2532

2022

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The Neutral Sphingomyelinase 2 Is Required to Polarize and Sustain T Cell Receptor Signaling

Frontiers in immunology, 2018-04, Vol.9, p.815-815

2018

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Measles Virus Targets DC-SIGN To Enhance Dendritic Cell Infection

Journal of Virology, 2006-04, Vol.80 (7), p.3477-3486

2006

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Neutral Sphingomyelinase-2 (NSM 2) Controls T Cell Metabolic Homeostasis and Reprogramming During Activation

Frontiers in molecular biosciences, 2020-09, Vol.7, p.217-217

2020

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CD150 (SLAM) Is a Receptor for Measles Virus but Is Not Involved in Viral Contact-Mediated Proliferation Inhibition

Journal of Virology, 2001-05, Vol.75 (10), p.4499-4505

2001

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Sphingolipids in viral infection

Sphingosine 1-phosphate metabolism and insulin signaling

DC-SIGN mediated sphingomyelinase-activation and ceramide generation is essential for enhancement of viral uptake in dendritic cells

Measles virus nucleocapsid transport to the plasma membrane requires stable expression and surface accumulation of the viral matrix protein

Differential activation of acid sphingomyelinase and ceramide release determines invasiveness of Neisseria meningitidis into brain endothelial cells

CD9 Clustering and Formation of Microvilli Zippers Between Contacting Cells Regulates Virus-Induced Cell Fusion

Human endogenous retrovirus envelope proteins target dendritic cells to suppress T‐cell activation

Membrane dynamics and interactions in measles virus dendritic cell infections

Hemagglutinin Protein of Wild-Type Measles Virus Activates Toll-Like Receptor 2 Signaling

Measles Virus Transmission from Dendritic Cells to T Cells: Formation of Synapse-Like Interfaces Concentrating Viral and Cellular Components

Azido-Ceramides, a Tool to Analyse SARS-CoV-2 Replication and Inhibition-SARS-CoV-2 Is Inhibited by Ceramides

Neutral sphingomyelinase in physiological and measles virus induced T cell suppression

Dynamic changes in the proximitome of neutral sphingomyelinase-2 (nSMase2) in TNFα stimulated Jurkat cells

The Role of Sphingomyelin Breakdown in Measles Virus Immunmodulation

Editorial: Sphingolipids in Infection Control

The Activity of the Neutral Sphingomyelinase Is Important in T Cell Recruitment and Directional Migration

Sphingomyelin Breakdown in T Cells: Role of Membrane Compartmentalization in T Cell Signaling and Interference by a Pathogen

Measles Virus Infection Fosters Dendritic Cell Motility in a 3D Environment to Enhance Transmission to Target Cells in the Respiratory Epithelium

Neutral sphingomyelinase 2 is a key factor for PorB‐dependent invasion of Neisseria gonorrhoeae

Sphingolipids: Effectors and Achilles Heals in Viral Infections?

The Acid Ceramidase Is a SARS-CoV-2 Host Factor

The Neutral Sphingomyelinase 2 Is Required to Polarize and Sustain T Cell Receptor Signaling

Measles Virus Targets DC-SIGN To Enhance Dendritic Cell Infection

Neutral Sphingomyelinase-2 (NSM 2) Controls T Cell Metabolic Homeostasis and Reprogramming During Activation

CD150 (SLAM) Is a Receptor for Measles Virus but Is Not Involved in Viral Contact-Mediated Proliferation Inhibition

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