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Microglia Responses to Pro-inflammatory Stimuli (LPS, IFNγ+TNFα) and Reprogramming by Resolving Cytokines (IL-4, IL-10)

Frontiers in cellular neuroscience, 2018-07, Vol.12, p.215-215

2018

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The microglial activation state regulates migration and roles of matrix-dissolving enzymes for invasion

Journal of neuroinflammation, 2013-06, Vol.10 (1), p.75-75, Article 843

2013

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Neutrophil Depletion Reduces Blood-Brain Barrier Breakdown, Axon Injury, and Inflammation After Intracerebral Hemorrhage

Journal of neuropathology and experimental neurology, 2011-03, Vol.70 (3), p.218-235

2011

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Mechanisms of Microglia-Mediated Neurotoxicity in a New Model of the Stroke Penumbra

The Journal of neuroscience, 2008-02, Vol.28 (9), p.2221-2230

2008

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Selective Activation of KCa3.1 and CRAC Channels by P2Y2 Receptors Promotes Ca2+ Signaling, Store Refilling and Migration of Rat Microglial Cells

PloS one, 2013-04, Vol.8 (4), p.e62345

2013

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Responses of rat and mouse primary microglia to pro- and anti-inflammatory stimuli: molecular profiles, K + channels and migration

Journal of neuroinflammation, 2017-08, Vol.14 (1), p.166-166, Article 166

2017

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PKA reduces the rat and human KCa3.1 current, CaM binding, and Ca2+ signaling, which requires Ser332/334 in the CaM-binding C terminus

The Journal of neuroscience, 2014-10, Vol.34 (40), p.13371-13383

2014

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Expression and contributions of TRPM7 and KCa2.3/SK3 channels to the increased migration and invasion of microglia in anti-inflammatory activation states

PloS one, 2014-08, Vol.9 (8), p.e106087

2014

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Selective activation of KCa3.1 and CRAC channels by P2Y2 receptors promotes Ca(2+) signaling, store refilling and migration of rat microglial cells

PloS one, 2013, Vol.8 (4), p.e62345

2013

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Molecular and Cellular Responses to Interleukin-4 Treatment in a Rat Model of Transient Ischemia

Journal of neuropathology and experimental neurology, 2016-11, Vol.75 (11), p.1058-1071

2016

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Regulation of podosome formation, microglial migration and invasion by Ca2+-signaling molecules expressed in podosomes

Journal of neuroinflammation, 2012-11, Vol.9 (1), p.250-250, Article 250

2012

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Complex molecular and functional outcomes of single versus sequential cytokine stimulation of rat microglia

Journal of neuroinflammation, 2016-03, Vol.13 (1), p.66-66, Article 66

2016

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Minocycline protects the blood–brain barrier and reduces edema following intracerebral hemorrhage in the rat

Experimental neurology, 2007-10, Vol.207 (2), p.227-237

2007

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Podosomes in migrating microglia: components and matrix degradation

Journal of neuroinflammation, 2012-08, Vol.9 (1), p.190-190, Article 190

2012

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IL-4 type 1 receptor signaling up-regulates KCNN4 expression, and increases the KCa3.1 current and its contribution to migration of alternative-activated microglia

Frontiers in cellular neuroscience, 2014-07, Vol.8, p.183

2014

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Age-Related Comparisons of Evolution of the Inflammatory Response After Intracerebral Hemorrhage in Rats

Translational stroke research, 2012-07, Vol.3 (Suppl 1), p.132-146

2012

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Comparing Effects of Transforming Growth Factor β1 on Microglia From Rat and Mouse: Transcriptional Profiles and Potassium Channels

Frontiers in cellular neuroscience, 2018-05, Vol.12, p.115-115

2018

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Inhibition of the Ca²⁺-dependent K⁺ channel, KCNN4/KCa3.1, improves tissue protection and locomotor recovery after spinal cord injury

The Journal of neuroscience, 2011-11, Vol.31 (45), p.16298-16308

2011

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Glial responses, neuron death and lesion resolution after intracerebral hemorrhage in young vs. aged rats

The European journal of neuroscience, 2008-10, Vol.28 (7), p.1316-1328

2008

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Evolution of the inflammatory response in the brain following intracerebral hemorrhage and effects of delayed minocycline treatment

Brain research, 2007-11, Vol.1180, p.140-154

2007

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Sex- and Development-Dependent Responses of Rat Microglia to Pro- and Anti-inflammatory Stimulation

Frontiers in cellular neuroscience, 2018-11, Vol.12, p.433-433

2018

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KCa3.1/IK1 Channel Regulation by cGMP-Dependent Protein Kinase (PKG) via Reactive Oxygen Species and CaMKII in Microglia: An Immune Modulating Feedback System?

Frontiers in immunology, 2015-04, Vol.6, p.153-153

2015

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N-type Ca2+ channels carry the largest current: implications for nanodomains and transmitter release

Nature neuroscience, 2010-11, Vol.13 (11), p.1348-1350

2010

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Neuron death and inflammation in a rat model of intracerebral hemorrhage: Effects of delayed minocycline treatment

Brain research, 2007-03, Vol.1136 (1), p.208-218

2007

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Molecular and Cellular Responses to Interleukin-4 Treatment in a Rat Model of Transient Ischemia

Journal of neuropathology and experimental neurology, 2016-11, Vol.75 (11), p.1058-1071

2016

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Microglia Responses to Pro-inflammatory Stimuli (LPS, IFNγ+TNFα) and Reprogramming by Resolving Cytokines (IL-4, IL-10)

The microglial activation state regulates migration and roles of matrix-dissolving enzymes for invasion

Neutrophil Depletion Reduces Blood-Brain Barrier Breakdown, Axon Injury, and Inflammation After Intracerebral Hemorrhage

Mechanisms of Microglia-Mediated Neurotoxicity in a New Model of the Stroke Penumbra

Selective Activation of KCa3.1 and CRAC Channels by P2Y2 Receptors Promotes Ca2+ Signaling, Store Refilling and Migration of Rat Microglial Cells

Responses of rat and mouse primary microglia to pro- and anti-inflammatory stimuli: molecular profiles, K + channels and migration

PKA reduces the rat and human KCa3.1 current, CaM binding, and Ca2+ signaling, which requires Ser332/334 in the CaM-binding C terminus

Expression and contributions of TRPM7 and KCa2.3/SK3 channels to the increased migration and invasion of microglia in anti-inflammatory activation states

Selective activation of KCa3.1 and CRAC channels by P2Y2 receptors promotes Ca(2+) signaling, store refilling and migration of rat microglial cells

Molecular and Cellular Responses to Interleukin-4 Treatment in a Rat Model of Transient Ischemia

Regulation of podosome formation, microglial migration and invasion by Ca2+-signaling molecules expressed in podosomes

Complex molecular and functional outcomes of single versus sequential cytokine stimulation of rat microglia

Minocycline protects the blood–brain barrier and reduces edema following intracerebral hemorrhage in the rat

Podosomes in migrating microglia: components and matrix degradation

IL-4 type 1 receptor signaling up-regulates KCNN4 expression, and increases the KCa3.1 current and its contribution to migration of alternative-activated microglia

Age-Related Comparisons of Evolution of the Inflammatory Response After Intracerebral Hemorrhage in Rats

Comparing Effects of Transforming Growth Factor β1 on Microglia From Rat and Mouse: Transcriptional Profiles and Potassium Channels

Inhibition of the Ca²⁺-dependent K⁺ channel, KCNN4/KCa3.1, improves tissue protection and locomotor recovery after spinal cord injury

Glial responses, neuron death and lesion resolution after intracerebral hemorrhage in young vs. aged rats

Evolution of the inflammatory response in the brain following intracerebral hemorrhage and effects of delayed minocycline treatment

Sex- and Development-Dependent Responses of Rat Microglia to Pro- and Anti-inflammatory Stimulation

KCa3.1/IK1 Channel Regulation by cGMP-Dependent Protein Kinase (PKG) via Reactive Oxygen Species and CaMKII in Microglia: An Immune Modulating Feedback System?

N-type Ca2+ channels carry the largest current: implications for nanodomains and transmitter release

Neuron death and inflammation in a rat model of intracerebral hemorrhage: Effects of delayed minocycline treatment

Molecular and Cellular Responses to Interleukin-4 Treatment in a Rat Model of Transient Ischemia

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