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Mechanical loading enhances the anabolic effects of intermittent parathyroid hormone (1–34) on trabecular and cortical bone in mice

Bone (New York, N.Y.), 2008-08, Vol.43 (2), p.238-248

2008

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The mouse fibula as a suitable bone for the study of functional adaptation to mechanical loading

Bone (New York, N.Y.), 2009-05, Vol.44 (5), p.930-935

2009

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Cellular accommodation and the response of bone to mechanical loading

Journal of biomechanics, 2005-09, Vol.38 (9), p.1838-1845

2005

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Loading-related Regulation of Transcription Factor EGR2/Krox-20 in Bone Cells Is ERK1/2 Protein-mediated and Prostaglandin, Wnt Signaling Pathway-, and Insulin-like Growth Factor-I Axis-dependent

The Journal of biological chemistry, 2012-02, Vol.287 (6), p.3946-3962

2012

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Lrp5 is not required for the proliferative response of osteoblasts to strain but regulates proliferation and apoptosis in a cell autonomous manner

PloS one, 2012-05, Vol.7 (5), p.e35726-e35726

2012

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Wnt/β-Catenin Signaling Is a Component of Osteoblastic Bone Cell Early Responses to Load-bearing and Requires Estrogen Receptor α

The Journal of biological chemistry, 2007-07, Vol.282 (28), p.20715-20727

2007

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Analysis of multiple bone responses to graded strains above functional levels, and to disuse, in mice in vivo show that the human Lrp5 G171V High Bone Mass mutation increases the osteogenic response to loading but that lack of Lrp5 activity reduces it

Bone (New York, N.Y.), 2011-08, Vol.49 (2), p.184-193

2011

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Loading-related regulation of gene expression in bone in the contexts of estrogen deficiency, lack of estrogen receptor α and disuse

Bone (New York, N.Y.), 2010-03, Vol.46 (3), p.628-642

2010

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Low-dose estrogen treatment suppresses periosteal bone formation in response to mechanical loading

Bone (New York, N.Y.), 2006-12, Vol.39 (6), p.1261-1267

2006

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Genetic selection for fast growth generates bone architecture characterised by enhanced periosteal expansion and limited consolidation of the cortices but a diminution in the early responses to mechanical loading

Bone (New York, N.Y.), 2009-08, Vol.45 (2), p.357-366

2009

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Knee ligament mechanical properties are not influenced by estrogen or its receptors

American journal of physiology: endocrinology and metabolism, 2006-05, Vol.290 (5), p.E1034-E1040

2006

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Erratum to “Mechanical loading enhances the anabolic effects of intermittent parathyroid hormone (1–34) on trabecular and cortical bone in mice” [Bone 43 (2008) 238–248]

Bone (New York, N.Y.), 2009-12, Vol.45 (6), p.1192-1195

2009

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Loading-related regulation of gene expression in bone in the contexts of estrogen deficiency, lack of estrogen receptor a and disuse

Bone (New York, N.Y.), 2010-03, Vol.46 (3), p.628-642

2010

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Wnt/Î²-Catenin Signaling Is a Component of Osteoblastic Bone Cell Early Responses to Load-bearing and Requires Estrogen Receptor Î

The Journal of biological chemistry, 2007-07, Vol.282 (28), p.20715

2007

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Regulation of bone cell sensitivity to mechanical stimulation

The FASEB journal, 2006-03, Vol.20 (4), p.A417-A417

2006

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Knee ligament mechanical properties are not influenced by estrogen or its receptors

American journal of physiology: endocrinology and metabolism, 2006-05, Vol.53 (5), p.E1034

2006

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Estrogen receptor‐α is required for the osteogenic response to mechanical loading in a ligand‐independent manner involving its activation function 1 but not 2

Journal of bone and mineral research, 2013-02, Vol.28 (2), p.291-301

2013

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Regional Specificity of Exercise and Calcium During Skeletal Growth in Girls: A Randomized Controlled Trial

Journal of bone and mineral research, 2003-01, Vol.18 (1), p.156-162

2003

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Secondary prevention for patients after a myocardial infarction: summary of updated NICE guidance

BMJ (Online), 2013-11, Vol.347 (nov13 3), p.f6544-f6544

2013

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Estrogen receptor-[alpha] is required for the osteogenic response to mechanical loading in a ligand-independent manner involving its activation function 1 but not 2

Journal of bone and mineral research, 2013-02, Vol.28 (2), p.291

2013

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Mechanical loading enhances the anabolic effects of intermittent parathyroid hormone (1–34) on trabecular and cortical bone in mice

The mouse fibula as a suitable bone for the study of functional adaptation to mechanical loading

Cellular accommodation and the response of bone to mechanical loading

Loading-related Regulation of Transcription Factor EGR2/Krox-20 in Bone Cells Is ERK1/2 Protein-mediated and Prostaglandin, Wnt Signaling Pathway-, and Insulin-like Growth Factor-I Axis-dependent

Lrp5 is not required for the proliferative response of osteoblasts to strain but regulates proliferation and apoptosis in a cell autonomous manner

Wnt/β-Catenin Signaling Is a Component of Osteoblastic Bone Cell Early Responses to Load-bearing and Requires Estrogen Receptor α

Analysis of multiple bone responses to graded strains above functional levels, and to disuse, in mice in vivo show that the human Lrp5 G171V High Bone Mass mutation increases the osteogenic response to loading but that lack of Lrp5 activity reduces it

Loading-related regulation of gene expression in bone in the contexts of estrogen deficiency, lack of estrogen receptor α and disuse

Low-dose estrogen treatment suppresses periosteal bone formation in response to mechanical loading

Genetic selection for fast growth generates bone architecture characterised by enhanced periosteal expansion and limited consolidation of the cortices but a diminution in the early responses to mechanical loading

Knee ligament mechanical properties are not influenced by estrogen or its receptors

Erratum to “Mechanical loading enhances the anabolic effects of intermittent parathyroid hormone (1–34) on trabecular and cortical bone in mice” [Bone 43 (2008) 238–248]

Loading-related regulation of gene expression in bone in the contexts of estrogen deficiency, lack of estrogen receptor a and disuse

Wnt/Î²-Catenin Signaling Is a Component of Osteoblastic Bone Cell Early Responses to Load-bearing and Requires Estrogen Receptor Î

Regulation of bone cell sensitivity to mechanical stimulation

Knee ligament mechanical properties are not influenced by estrogen or its receptors

Estrogen receptor‐α is required for the osteogenic response to mechanical loading in a ligand‐independent manner involving its activation function 1 but not 2

Regional Specificity of Exercise and Calcium During Skeletal Growth in Girls: A Randomized Controlled Trial

Secondary prevention for patients after a myocardial infarction: summary of updated NICE guidance

Estrogen receptor-[alpha] is required for the osteogenic response to mechanical loading in a ligand-independent manner involving its activation function 1 but not 2

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