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Chloride channels in cancer: Focus on chloride intracellular channel 1 and 4 (CLIC1 AND CLIC4) proteins in tumor development and as novel therapeutic targets

Biochimica et biophysica acta, 2015-10, Vol.1848 (10), p.2523-2531

2015

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Functional heterogeneity of the four voltage sensors of a human L-type calcium channel

Proceedings of the National Academy of Sciences - PNAS, 2014-12, Vol.111 (51), p.18381-18386

2014

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Functional Role of CLIC1 Ion Channel in Glioblastoma-Derived Stem/Progenitor Cells

JNCI : Journal of the National Cancer Institute, 2013-11, Vol.105 (21), p.1644-1655

2013

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RCK2 domain of the human BKCa channel is a calcium sensor

Proceedings of the National Academy of Sciences - PNAS, 2008-01, Vol.105 (1), p.376-381

2008

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Myocardial fibrosis and calcification are attenuated by microRNA-129-5p targeting Asporin and Sox9 in cardiac fibroblasts

JCI insight, 2023-05, Vol.8 (9)

2023

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Voltage-Dependent Conformational Changes in Human Ca²⁺-and Voltage-Activated K⁺ Channel, Revealed by Voltage-Clamp Fluorometry

Proceedings of the National Academy of Sciences - PNAS, 2006-08, Vol.103 (33), p.12619-12624

2006

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Point mutations in the transmembrane region of the clic1 ion channel selectively modify its biophysical properties

PloS one, 2013-09, Vol.8 (9), p.e74523

2013

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Operation of the voltage sensor of a human voltage- and Ca²⁺-activated K⁺ channel

Proceedings of the National Academy of Sciences - PNAS, 2010-03, Vol.107 (9), p.4459-4464

2010

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Nifedipine binding to human Cav1.1 allosterically sets 3 out of 4 voltage sensors into their “relaxed state”

Biophysical journal, 2022-02, Vol.121 (3), p.97a-97a

2022

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Voltage Sensor Operation in the Embryonic Splice Variant of Skeletal CaV1.1 Channels

Biophysical journal, 2021-02, Vol.120 (3), p.155a-155a

2021

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Nifedipine Modulates the Activation of CaV1.1 Voltage-Sensing Domains

Biophysical journal, 2021-02, Vol.120 (3), p.154a-155a

2021

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Differential Modulation of L-Type CaV1.1 and CaV1.2 Channels by the α2δ-1 Subunit

Biophysical journal, 2020-02, Vol.118 (3), p.499a-499a

2020

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Allosteric mechanism of CaV3.1 inhibition by Z944, a selective T-type channel antagonist

Biophysical journal, 2024-02, Vol.123 (3), p.111a-111a

2024

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Probing the mechanisms of low-voltage activation in T-type CaV3.1 calcium channels

Biophysical journal, 2024-02, Vol.123 (3), p.112a-112a

2024

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The voltage dependent structural rearrangements of the four voltage sensing domains of the human T-type Cav3.1 channel

Biophysical journal, 2022-02, Vol.121 (3), p.99a-99a

2022

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The voltage-dependent structural transitions governing the opening of the human t-type Cav3.1 channel: Insights from voltage-clamp fluorometry

Biophysical journal, 2023-02, Vol.122 (3), p.107a-107a

2023

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Skeletal γ1 Subunit Modulation of Human CaV1.1 and CaV1.2 Channels

Biophysical journal, 2019-02, Vol.116 (3), p.112a-113a

2019

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The distinct role of the four voltage sensors of the skeletal CaV1.1 channel in voltage-dependent activation

The Journal of general physiology, 2021-11, Vol.153 (11), p.1

2021

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The α2δ-1 subunit remodels CaV1.2 voltage sensors and allows Ca2+ influx at physiological membrane potentials

The Journal of general physiology, 2016-08, Vol.148 (2), p.147-159

2016

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The Contribution of RCK Domains to Human BK Channel Allosteric Activation

The Journal of biological chemistry, 2012-06, Vol.287 (26), p.21741-21750

2012

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Abstract P475: Microrna-129-5p Rescues Myocardial Fibrosis And Calcification By Targeting Asporin And Sox9 In Cardiac Fibroblasts

Circulation research, 2021-09, Vol.129 (Suppl_1)

2021

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Influence of channel subunit composition on L-type Ca2+ current kinetics and cardiac wave stability

American journal of physiology. Heart and circulatory physiology, 2007-09, Vol.293 (3), p.H1805-H1815

2007

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How Does the α2δ-1 Subunit Modulate Skeletal CaV1.1 Channels?

Biophysical journal, 2019-02, Vol.116 (3), p.113a-113a

2019

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A Mutation Linked to Malignant Hyperthermia in the Skeletal CaV1.1 Channel Stabilizes the Resting State of Voltage Sensor I and Impairs Channel Activation

Biophysical journal, 2019-02, Vol.116 (3), p.111a-111a

2019

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The Contribution of the Individual Voltage Sensors to the Activation of Skeletal CaV1.1 Channels

Biophysical journal, 2020-02, Vol.118 (3), p.105a-105a

2020

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Chloride channels in cancer: Focus on chloride intracellular channel 1 and 4 (CLIC1 AND CLIC4) proteins in tumor development and as novel therapeutic targets

Functional heterogeneity of the four voltage sensors of a human L-type calcium channel

Functional Role of CLIC1 Ion Channel in Glioblastoma-Derived Stem/Progenitor Cells

RCK2 domain of the human BKCa channel is a calcium sensor

Myocardial fibrosis and calcification are attenuated by microRNA-129-5p targeting Asporin and Sox9 in cardiac fibroblasts

Voltage-Dependent Conformational Changes in Human Ca²⁺-and Voltage-Activated K⁺ Channel, Revealed by Voltage-Clamp Fluorometry

Point mutations in the transmembrane region of the clic1 ion channel selectively modify its biophysical properties

Operation of the voltage sensor of a human voltage- and Ca²⁺-activated K⁺ channel

Nifedipine binding to human Cav1.1 allosterically sets 3 out of 4 voltage sensors into their “relaxed state”

Voltage Sensor Operation in the Embryonic Splice Variant of Skeletal CaV1.1 Channels

Nifedipine Modulates the Activation of CaV1.1 Voltage-Sensing Domains

Differential Modulation of L-Type CaV1.1 and CaV1.2 Channels by the α2δ-1 Subunit

Allosteric mechanism of CaV3.1 inhibition by Z944, a selective T-type channel antagonist

Probing the mechanisms of low-voltage activation in T-type CaV3.1 calcium channels

The voltage dependent structural rearrangements of the four voltage sensing domains of the human T-type Cav3.1 channel

The voltage-dependent structural transitions governing the opening of the human t-type Cav3.1 channel: Insights from voltage-clamp fluorometry

Skeletal γ1 Subunit Modulation of Human CaV1.1 and CaV1.2 Channels

The distinct role of the four voltage sensors of the skeletal CaV1.1 channel in voltage-dependent activation

The α2δ-1 subunit remodels CaV1.2 voltage sensors and allows Ca2+ influx at physiological membrane potentials

The Contribution of RCK Domains to Human BK Channel Allosteric Activation

Abstract P475: Microrna-129-5p Rescues Myocardial Fibrosis And Calcification By Targeting Asporin And Sox9 In Cardiac Fibroblasts

Influence of channel subunit composition on L-type Ca2+ current kinetics and cardiac wave stability

How Does the α2δ-1 Subunit Modulate Skeletal CaV1.1 Channels?

A Mutation Linked to Malignant Hyperthermia in the Skeletal CaV1.1 Channel Stabilizes the Resting State of Voltage Sensor I and Impairs Channel Activation

The Contribution of the Individual Voltage Sensors to the Activation of Skeletal CaV1.1 Channels

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