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Biomarkers of persistent renal vulnerability after acute kidney injury recovery

Scientific reports, 2021-10, Vol.11 (1), p.21183-21183, Article 21183

2021

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The Urinary Level of Injury Biomarkers Is Not Univocally Reflective of the Extent of Toxic Renal Tubular Injury in Rats

International journal of molecular sciences, 2022-03, Vol.23 (7), p.3494

2022

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Sub-nephrotoxic doses of gentamicin predispose animals to developing acute kidney injury and to excrete ganglioside M2 activator protein

Kidney international, 2010-11, Vol.78 (10), p.1006-1015

2010

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Urinary levels of regenerating islet-derived protein III β and gelsolin differentiate gentamicin from cisplatin-induced acute kidney injury in rats

Kidney international, 2011-03, Vol.79 (5), p.518-528

2011

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Impaired Tubular Reabsorption Is the Main Mechanism Explaining Increases in Urinary NGAL Excretion Following Acute Kidney Injury in Rats

Toxicological sciences, 2020-05, Vol.175 (1), p.75-86

2020

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Mechanisms of triple whammy acute kidney injury

Pharmacology & therapeutics (Oxford), 2016-11, Vol.167, p.132-145

2016

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N-acetylcysteine transforms necrosis into apoptosis and affords tailored protection from cisplatin cytotoxicity

Toxicology and applied pharmacology, 2018-06, Vol.349, p.83-93

2018

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Urinary Plasminogen Activator Inhibitor-1: A Biomarker of Acute Tubular Injury

American journal of nephrology, 2021-11, Vol.52 (9), p.714-724

2021

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Urinary proteomics in renal pathophysiology: Impact of proteinuria

Proteomics. Clinical applications, 2015-06, Vol.9 (5-6), p.636-640

2015

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Necrotic Concentrations of Cisplatin Activate the Apoptotic Machinery but Inhibit Effector Caspases and Interfere with the Execution of Apoptosis

Toxicological sciences, 2011-07, Vol.122 (1), p.73-85

2011

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Identification of Pre-Renal and Intrinsic Acute Kidney Injury by Anamnestic and Biochemical Criteria: Distinct Association with Urinary Injury Biomarkers

International journal of molecular sciences, 2023-01, Vol.24 (3), p.1826

2023

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Combined use of GM2AP and TCP1-eta urinary levels predicts recovery from intrinsic acute kidney injury

Scientific reports, 2020-07, Vol.10 (1), p.11599-11599, Article 11599

2020

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Diagnosis of Cardiac Surgery-Associated Acute Kidney Injury: State of the Art and Perspectives

Journal of clinical medicine, 2022-08, Vol.11 (15), p.4576

2022

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Pathophysiological role of different tubular epithelial cell death modes in acute kidney injury

Clinical kidney journal, 2015-10, Vol.8 (5), p.548-559

2015

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Urinary KIM-1 Correlates with the Subclinical Sequelae of Tubular Damage Persisting after the Apparent Functional Recovery from Intrinsic Acute Kidney Injury

Biomedicines, 2022-05, Vol.10 (5), p.1106

2022

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Increased Klk9 Urinary Excretion Is Associated to Hypertension-Induced Cardiovascular Damage and Renal Alterations

Medicine (Baltimore), 2015-10, Vol.94 (41), p.e1617-e1617

2015

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Pathophysiology of Acute Kidney Frailty

Physiology (Bethesda, Md.), 2023-11, Vol.38 (6), p.275-284

2023

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Urinary GM2AP coincides with renal cortical damage and grades cisplatin nephrotoxicity severity in rats

Toxicology (Amsterdam), 2024-11, Vol.508, p.153919, Article 153919

2024

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Subcellular targets of cisplatin cytotoxicity: an integrated view

Pharmacology & therapeutics (Oxford), 2012-10, Vol.136 (1), p.35-55

2012

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Urinary TCP1-eta: A Cortical Damage Marker for the Pathophysiological Diagnosis and Prognosis of Acute Kidney Injury

Toxicological sciences, 2020-03, Vol.174 (1), p.3-15

2020

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Acute tubular necrosis: An old term in search for a new meaning within the evolving concept of acute kidney injury

European journal of molecular and clinical medicine, 2015-05, Vol.2 (4-5), p.110-117

2015

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Activation of the ALK-5 Pathway is not per se Sufficient for the Antiproliferative Effect of TGF-β1 on Renal Tubule Epithelial Cells

Cellular physiology and biochemistry, 2015, Vol.37 (4), p.1231-1239

2015

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Pathophysiological mechanisms underlying a rat model of triple whammy acute kidney injury

Laboratory investigation, 2020-11, Vol.100 (11), p.1455-1464

2020

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3937 URINARY EXCRETION OF LIVER-TYPE FATTY ACID-BINDING PROTEIN DETECTS KIDNEY HISTOLOGICAL ALTERATIONS DURING PROGRESSIVE RENAL FIBROSIS

Nephrology, dialysis, transplantation, 2023-06, Vol.38 (Supplement_1)

2023

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1973 Factors conditioning cisplatin nephrotoxicity

Nephrology, dialysis, transplantation, 2024-05, Vol.39 (Supplement_1)

2024

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Biomarkers of persistent renal vulnerability after acute kidney injury recovery

The Urinary Level of Injury Biomarkers Is Not Univocally Reflective of the Extent of Toxic Renal Tubular Injury in Rats

Sub-nephrotoxic doses of gentamicin predispose animals to developing acute kidney injury and to excrete ganglioside M2 activator protein

Urinary levels of regenerating islet-derived protein III β and gelsolin differentiate gentamicin from cisplatin-induced acute kidney injury in rats

Impaired Tubular Reabsorption Is the Main Mechanism Explaining Increases in Urinary NGAL Excretion Following Acute Kidney Injury in Rats

Mechanisms of triple whammy acute kidney injury

N-acetylcysteine transforms necrosis into apoptosis and affords tailored protection from cisplatin cytotoxicity

Urinary Plasminogen Activator Inhibitor-1: A Biomarker of Acute Tubular Injury

Urinary proteomics in renal pathophysiology: Impact of proteinuria

Necrotic Concentrations of Cisplatin Activate the Apoptotic Machinery but Inhibit Effector Caspases and Interfere with the Execution of Apoptosis

Identification of Pre-Renal and Intrinsic Acute Kidney Injury by Anamnestic and Biochemical Criteria: Distinct Association with Urinary Injury Biomarkers

Combined use of GM2AP and TCP1-eta urinary levels predicts recovery from intrinsic acute kidney injury

Diagnosis of Cardiac Surgery-Associated Acute Kidney Injury: State of the Art and Perspectives

Pathophysiological role of different tubular epithelial cell death modes in acute kidney injury

Urinary KIM-1 Correlates with the Subclinical Sequelae of Tubular Damage Persisting after the Apparent Functional Recovery from Intrinsic Acute Kidney Injury

Increased Klk9 Urinary Excretion Is Associated to Hypertension-Induced Cardiovascular Damage and Renal Alterations

Pathophysiology of Acute Kidney Frailty

Urinary GM2AP coincides with renal cortical damage and grades cisplatin nephrotoxicity severity in rats

Subcellular targets of cisplatin cytotoxicity: an integrated view

Urinary TCP1-eta: A Cortical Damage Marker for the Pathophysiological Diagnosis and Prognosis of Acute Kidney Injury

Acute tubular necrosis: An old term in search for a new meaning within the evolving concept of acute kidney injury

Activation of the ALK-5 Pathway is not per se Sufficient for the Antiproliferative Effect of TGF-β1 on Renal Tubule Epithelial Cells

Pathophysiological mechanisms underlying a rat model of triple whammy acute kidney injury

3937 URINARY EXCRETION OF LIVER-TYPE FATTY ACID-BINDING PROTEIN DETECTS KIDNEY HISTOLOGICAL ALTERATIONS DURING PROGRESSIVE RENAL FIBROSIS

1973 Factors conditioning cisplatin nephrotoxicity

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