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The Multiple Actions of GLP-1 on the Process of Glucose-Stimulated Insulin Secretion

Diabetes (New York, N.Y.), 2002-12, Vol.51 (suppl 3), p.S434-S442

2002

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Generation of a Phenotypic Array of Hypothalamic Neuronal Cell Models to Study Complex Neuroendocrine Disorders

Endocrinology (Philadelphia), 2004-01, Vol.145 (1), p.393-400

2004

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Exogenous Nitric Oxide and Endogenous Glucose-Stimulated β-Cell Nitric Oxide Augment Insulin Release

Diabetes (New York, N.Y.), 2002-12, Vol.51 (12), p.3450-3460

2002

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Pollinex® Quattro: a novel and well-tolerated, ultra short-course allergy vaccine

Expert review of vaccines, 2006-10, Vol.5 (5), p.617-629

2006

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Increased uncoupling protein-2 levels in β-cells are associated with impaired glucose-stimulated insulin secretion: Mechanism of action

Diabetes (New York, N.Y.), 2001-06, Vol.50 (6), p.1302-1310

2001

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The 25-kDa Synaptosome-associated Protein (SNAP-25) Binds and Inhibits Delayed Rectifier Potassium Channels in Secretory Cells

The Journal of biological chemistry, 2002-06, Vol.277 (23), p.20195-20204

2002

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SNAP-25, a SNARE protein, inhibits two types of K+ channels in esophageal smooth muscle

Gastroenterology (New York, N.Y. 1943), 2002-04, Vol.122 (4), p.994-1006

2002

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Ion channel diversity in the feline smooth muscle esophagus

American journal of physiology: Gastrointestinal and liver physiology, 2002-02, Vol.282 (2), p.G288-G299

2002

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Glucagon-like peptide-1 receptor activation antagonizes voltage-dependent repolarizing K(+) currents in beta-cells: a possible glucose-dependent insulinotropic mechanism

Diabetes (New York, N.Y.), 2002-12, Vol.51 Suppl 3, p.S443-S447

2002

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Non-clinical pharmacology, pharmacokinetics, and safety findings for the antihistamine bepotastine besilate

Current medical research and opinion, 2010-10, Vol.26 (10), p.2329-2338

2010

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Synaptosome-Associated Protein of 25 Kilodaltons Modulates Kv2.1 Voltage-Dependent K+ Channels in Neuroendocrine Islet β-Cells through an Interaction with the Channel N Terminus

Molecular endocrinology (Baltimore, Md.), 2002-11, Vol.16 (11), p.2452-2461

2002

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Temperature and redox state dependence of native Kv2.1 currents in rat pancreatic β‐cells

The Journal of physiology, 2003-02, Vol.546 (3), p.647-653

2003

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Mutations to the Third Cytoplasmic Domain of the Glucagon-Like Peptide 1 (GLP-1) Receptor Can Functionally Uncouple GLP-1-Stimulated Insulin Secretion in HIT-T15 Cells

Molecular endocrinology (Baltimore, Md.), 1999-08, Vol.13 (8), p.1305-1317

1999

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Potassium channel diversity within the muscular components of the feline lower esophageal sphincter

Canadian journal of physiology and pharmacology, 2004-11, Vol.82 (11), p.1006-1017

2004

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Glucagon-Like Peptide-1 Receptor Activation Antagonizes Voltage-Dependent Repolarizing K+ Currents in β-Cells

Diabetes (New York, N.Y.), 2002-12, Vol.51 (suppl 3), p.S443-S447

2002

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Myogenic NOS in canine lower esophageal sphincter: enzyme activation, substrate recycling, and product actions

American Journal of Physiology: Cell Physiology, 1998-04, Vol.274 (4), p.C1145-C1157

1998

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Calcium source diversity in canine lower esophageal sphincter muscle

The Journal of pharmacology and experimental therapeutics, 1998-10, Vol.287 (1), p.98

1998

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Myogenic nitric oxide synthase activity in canine lower oesophageal sphincter: morphological and functional evidence

British journal of pharmacology, 1998-03, Vol.123 (6), p.1055-1064

1998

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Inhibition of Kv2.1 Voltage-dependent K+Channels in Pancreatic β-Cells Enhances Glucose-dependent Insulin Secretion

The Journal of biological chemistry, 2002-11, Vol.277 (47), p.44938-44945

2002

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Inhibition of Kv2.1 voltage-dependent K+ channels in pancreatic beta-cells enhances glucose-dependent insulin secretion

The Journal of biological chemistry, 2002-11, Vol.277 (47), p.44938

2002

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Supramaximal cholecystokinin displaces Munc18c from the pancreatic acinar basal surface, redirecting apical exocytosis to the basal membrane

The Journal of clinical investigation, 2001-12, Vol.108 (11), p.1597-1611

2001

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Increased Uncoupling Protein-2 Levels in β-cells Are Associated With Impaired Glucose-Stimulated Insulin Secretion

Diabetes (New York, N.Y.), 2001-06, Vol.50 (6), p.1302-1310

2001

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Supramaximal cholecystokinin displaces Munc18c from the pancreatic acinar basal surface, redirecting apical exocytosis to the basal membrane

The Journal of clinical investigation, 2001-12, Vol.108 (11), p.1597-1611

2001

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Pollinex super( registered ) Quattro: a novel and well-tolerated, ultra short-course allergy vaccine

Expert review of vaccines, 2006-10, Vol.5 (5), p.617-629

2006

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Temperature and redox state dependence of native Kv2.1 currents in rat pancreatic Î²-cells

The Journal of physiology, 2003-02, Vol.546 (3), p.647

2003

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The Multiple Actions of GLP-1 on the Process of Glucose-Stimulated Insulin Secretion

Generation of a Phenotypic Array of Hypothalamic Neuronal Cell Models to Study Complex Neuroendocrine Disorders

Exogenous Nitric Oxide and Endogenous Glucose-Stimulated β-Cell Nitric Oxide Augment Insulin Release

Pollinex® Quattro: a novel and well-tolerated, ultra short-course allergy vaccine

Increased uncoupling protein-2 levels in β-cells are associated with impaired glucose-stimulated insulin secretion: Mechanism of action

The 25-kDa Synaptosome-associated Protein (SNAP-25) Binds and Inhibits Delayed Rectifier Potassium Channels in Secretory Cells

SNAP-25, a SNARE protein, inhibits two types of K+ channels in esophageal smooth muscle

Ion channel diversity in the feline smooth muscle esophagus

Glucagon-like peptide-1 receptor activation antagonizes voltage-dependent repolarizing K(+) currents in beta-cells: a possible glucose-dependent insulinotropic mechanism

Non-clinical pharmacology, pharmacokinetics, and safety findings for the antihistamine bepotastine besilate

Synaptosome-Associated Protein of 25 Kilodaltons Modulates Kv2.1 Voltage-Dependent K+ Channels in Neuroendocrine Islet β-Cells through an Interaction with the Channel N Terminus

Temperature and redox state dependence of native Kv2.1 currents in rat pancreatic β‐cells

Mutations to the Third Cytoplasmic Domain of the Glucagon-Like Peptide 1 (GLP-1) Receptor Can Functionally Uncouple GLP-1-Stimulated Insulin Secretion in HIT-T15 Cells

Potassium channel diversity within the muscular components of the feline lower esophageal sphincter

Glucagon-Like Peptide-1 Receptor Activation Antagonizes Voltage-Dependent Repolarizing K+ Currents in β-Cells

Myogenic NOS in canine lower esophageal sphincter: enzyme activation, substrate recycling, and product actions

Calcium source diversity in canine lower esophageal sphincter muscle

Myogenic nitric oxide synthase activity in canine lower oesophageal sphincter: morphological and functional evidence

Inhibition of Kv2.1 Voltage-dependent K+Channels in Pancreatic β-Cells Enhances Glucose-dependent Insulin Secretion

Inhibition of Kv2.1 voltage-dependent K+ channels in pancreatic beta-cells enhances glucose-dependent insulin secretion

Supramaximal cholecystokinin displaces Munc18c from the pancreatic acinar basal surface, redirecting apical exocytosis to the basal membrane

Increased Uncoupling Protein-2 Levels in β-cells Are Associated With Impaired Glucose-Stimulated Insulin Secretion

Supramaximal cholecystokinin displaces Munc18c from the pancreatic acinar basal surface, redirecting apical exocytosis to the basal membrane

Pollinex super( registered ) Quattro: a novel and well-tolerated, ultra short-course allergy vaccine

Temperature and redox state dependence of native Kv2.1 currents in rat pancreatic Î²-cells

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