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Over-expression of HSP70 attenuates caspase-dependent and caspase-independent pathways and inhibits neuronal apoptosis

Journal of neurochemistry, 2012-11, Vol.123 (4), p.542-554

2012

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Late exercise reduces neuroinflammation and cognitive dysfunction after traumatic brain injury

Neurobiology of disease, 2013-06, Vol.54, p.252-263

2013

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MicroRNA-711-Induced Downregulation of Angiopoietin-1 Mediates Neuronal Cell Death

Journal of neurotrauma, 2018-10, Vol.35 (20), p.2462-2481

2018

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Down-Regulation of miR-23a-3p Mediates Irradiation-Induced Neuronal Apoptosis

International journal of molecular sciences, 2020-05, Vol.21 (10), p.3695

2020

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Irradiation-Induced Upregulation of miR-711 Inhibits DNA Repair and Promotes Neurodegeneration Pathways

International journal of molecular sciences, 2020-07, Vol.21 (15), p.5239

2020

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Ablation of the transcription factors E2F1-2 limits neuroinflammation and associated neurological deficits after contusive spinal cord injury

Cell cycle (Georgetown, Tex.), 2015-12, Vol.14 (23), p.3698-3712

2015

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Mithramycin selectively attenuates DNA-damage-induced neuronal cell death

Cell death & disease, 2020-07, Vol.11 (7), p.587, Article 587

2020

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Inhibition of E2F1/CDK1 pathway attenuates neuronal apoptosis in vitro and confers neuroprotection after spinal cord injury in vivo

PloS one, 2012-07, Vol.7 (7), p.e42129

2012

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Cleavage of proBDNF to BDNF by a Tolloid-Like Metalloproteinase Is Required for Acquisition of In Vitro Eyeblink Classical Conditioning

The Journal of neuroscience, 2009-11, Vol.29 (47), p.14956-14964

2009

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Impaired autophagy flux is associated with neuronal cell death after traumatic brain injury

Autophagy, 2014-12, Vol.10 (12), p.2208-2222

2014

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Spinal cord injury causes brain inflammation associated with cognitive and affective changes: role of cell cycle pathways

The Journal of neuroscience, 2014-08, Vol.34 (33), p.10989-11006

2014

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Downregulation of miR-23a and miR-27a following experimental traumatic brain injury induces neuronal cell death through activation of proapoptotic Bcl-2 proteins

The Journal of neuroscience, 2014-07, Vol.34 (30), p.10055-10071

2014

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Activation of mammalian Tolloid-like 1 expression by hypoxia in human neuroblastoma SH-SY5Y cells

Biochemical and biophysical research communications, 2009-11, Vol.389 (2), p.338-342

2009

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The voltage-gated proton channel Hv1 plays a detrimental role in contusion spinal cord injury via extracellular acidosis-mediated neuroinflammation

Brain, behavior, and immunity, 2021-01, Vol.91, p.267-283

2021

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Oligomeric Amyloid-β Inhibits the Proteolytic Conversion of Brain-derived Neurotrophic Factor (BDNF), AMPA Receptor Trafficking, and Classical Conditioning

The Journal of biological chemistry, 2010-11, Vol.285 (45), p.34708-34717

2010

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Traumatic brain injury in aged animals increases lesion size and chronically alters microglial/macrophage classical and alternative activation states

Neurobiology of aging, 2013-05, Vol.34 (5), p.1397-1411

2013

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Characterization of a novel reptilian tolloid-like gene in the pond turtle, Pseudemys scripta elegans

Brain research, 2007-06, Vol.1154, p.22-30

2007

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Inhibition of NOX2 signaling limits pain-related behavior and improves motor function in male mice after spinal cord injury: Participation of IL-10/miR-155 pathways

Brain, behavior, and immunity, 2019-08, Vol.80, p.73-87

2019

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Delayed microglial depletion after spinal cord injury reduces chronic inflammation and neurodegeneration in the brain and improves neurological recovery in male mice

Theranostics, 2020-01, Vol.10 (25), p.11376-11403

2020

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Proton extrusion during oxidative burst in microglia exacerbates pathological acidosis following traumatic brain injury

Glia, 2021-03, Vol.69 (3), p.746-764

2021

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Transcriptional profiling predicts behavioral performance in experimental mouse model of traumatic brain injury (TBI)

The Journal of immunology (1950), 2020-05, Vol.204 (1_Supplement), p.75-75.17

2020

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Anxiolytic reversal of classically conditioned / chronic stress-induced gene expression and learning in the Stress Alternatives Model

Behavioural brain research, 2023-02, Vol.440, p.114258-114258, Article 114258

2023

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Voluntary Exercise Preconditioning Activates Multiple Antiapoptotic Mechanisms and Improves Neurological Recovery after Experimental Traumatic Brain Injury

Journal of neurotrauma, 2015-09, Vol.32 (17), p.1347-1360

2015

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Activation of Tolloid-like 1 gene expression by the cardiac specific homeobox gene Nkx2-5

Development, growth & differentiation, 2009-05, Vol.51 (4), p.403-410

2009

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Inhibition of miR-155 Limits Neuroinflammation and Improves Functional Recovery After Experimental Traumatic Brain Injury in Mice

Neurotherapeutics, 2019-01, Vol.16 (1), p.216-230

2019

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Over-expression of HSP70 attenuates caspase-dependent and caspase-independent pathways and inhibits neuronal apoptosis

Late exercise reduces neuroinflammation and cognitive dysfunction after traumatic brain injury

MicroRNA-711-Induced Downregulation of Angiopoietin-1 Mediates Neuronal Cell Death

Down-Regulation of miR-23a-3p Mediates Irradiation-Induced Neuronal Apoptosis

Irradiation-Induced Upregulation of miR-711 Inhibits DNA Repair and Promotes Neurodegeneration Pathways

Ablation of the transcription factors E2F1-2 limits neuroinflammation and associated neurological deficits after contusive spinal cord injury

Mithramycin selectively attenuates DNA-damage-induced neuronal cell death

Inhibition of E2F1/CDK1 pathway attenuates neuronal apoptosis in vitro and confers neuroprotection after spinal cord injury in vivo

Cleavage of proBDNF to BDNF by a Tolloid-Like Metalloproteinase Is Required for Acquisition of In Vitro Eyeblink Classical Conditioning

Impaired autophagy flux is associated with neuronal cell death after traumatic brain injury

Spinal cord injury causes brain inflammation associated with cognitive and affective changes: role of cell cycle pathways

Downregulation of miR-23a and miR-27a following experimental traumatic brain injury induces neuronal cell death through activation of proapoptotic Bcl-2 proteins

Activation of mammalian Tolloid-like 1 expression by hypoxia in human neuroblastoma SH-SY5Y cells

The voltage-gated proton channel Hv1 plays a detrimental role in contusion spinal cord injury via extracellular acidosis-mediated neuroinflammation

Oligomeric Amyloid-β Inhibits the Proteolytic Conversion of Brain-derived Neurotrophic Factor (BDNF), AMPA Receptor Trafficking, and Classical Conditioning

Traumatic brain injury in aged animals increases lesion size and chronically alters microglial/macrophage classical and alternative activation states

Characterization of a novel reptilian tolloid-like gene in the pond turtle, Pseudemys scripta elegans

Inhibition of NOX2 signaling limits pain-related behavior and improves motor function in male mice after spinal cord injury: Participation of IL-10/miR-155 pathways

Delayed microglial depletion after spinal cord injury reduces chronic inflammation and neurodegeneration in the brain and improves neurological recovery in male mice

Proton extrusion during oxidative burst in microglia exacerbates pathological acidosis following traumatic brain injury

Transcriptional profiling predicts behavioral performance in experimental mouse model of traumatic brain injury (TBI)

Anxiolytic reversal of classically conditioned / chronic stress-induced gene expression and learning in the Stress Alternatives Model

Voluntary Exercise Preconditioning Activates Multiple Antiapoptotic Mechanisms and Improves Neurological Recovery after Experimental Traumatic Brain Injury

Activation of Tolloid-like 1 gene expression by the cardiac specific homeobox gene Nkx2-5

Inhibition of miR-155 Limits Neuroinflammation and Improves Functional Recovery After Experimental Traumatic Brain Injury in Mice

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