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Development and characterization of gemcitabine-resistant pancreatic tumor cells

Annals of surgical oncology, 2007-12, Vol.14 (12), p.3629-3637

2007

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Treatment for advanced tumors: SRC reclaims center stage

Clinical cancer research, 2006-03, Vol.12 (5), p.1398-1401

2006

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Inhibition of Src Expression and Activity Inhibits Tumor Progression and Metastasis of Human Pancreatic Adenocarcinoma Cells in an Orthotopic Nude Mouse Model

The American journal of pathology, 2006-03, Vol.168 (3), p.962-972

2006

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Src activation regulates anoikis in human colon tumor cell lines

Oncogene, 2002-11, Vol.21 (51), p.7797-7807

2002

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Src family kinases in tumor progression and metastasis

Cancer and metastasis reviews, 2003-12, Vol.22 (4), p.337-358

2003

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Expression and activity of src regulate interleukin-8 expression in pancreatic adenocarcinoma cells : Implications for angiogenesis

Cancer research (Chicago, Ill.), 2005-08, Vol.65 (16), p.7214-7222

2005

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AFAP-110 is overexpressed in prostate cancer and contributes to tumorigenic growth by regulating focal contacts

The Journal of clinical investigation, 2007-10, Vol.117 (10), p.2962-2973

2007

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AP23846, a novel and highly potent Src family kinase inhibitor, reduces vascular endothelial growth factor and interleukin-8 expression in human solid tumor cell lines and abrogates downstream angiogenic processes

Molecular cancer therapeutics, 2005-12, Vol.4 (12), p.1900-1911

2005

Volltextzugriff (PDF)

c-Src regulates constitutive and EGF-mediated VEGF expression in pancreatic tumor cells through activation of phosphatidyl inositol-3 kinase and p38 MAPK

Pancreas, 2005-10, Vol.31 (3), p.263-274

2005

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Loss of tyrosine phosphatase-dependent inhibition promotes activation of tyrosine kinase c-Src in detached pancreatic cells

Molecular carcinogenesis, 2010-12, Vol.49 (12), p.1007-1021

2010

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Targeting the yin and the yang: combined inhibition of the tyrosine kinase c-Src and the tyrosine phosphatase SHP-2 disrupts pancreatic cancer signaling and biology in vitro and tumor formation in vivo

Pancreas, 2013-07, Vol.42 (5), p.795-806

2013

Volltextzugriff (PDF)

SRC inhibitors as potential therapeutic agents for human cancers

Mini reviews in medicinal chemistry, 2006-06, Vol.6 (6), p.681

2006

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Src activation of Stat3 is an independent requirement from NF-κB activation for constitutive IL-8 expression in human pancreatic adenocarcinoma cells

Angiogenesis (London), 2006-06, Vol.9 (2), p.101-110

2006

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PKC Phosphorylation Increases the Ability of AFAP-110 to Cross-link Actin Filaments

Molecular biology of the cell, 2002-07, Vol.13 (7), p.2311-2322

2002

Volltextzugriff (PDF)

c-SrcはPI3Kとp38 MAPKのリン酸化を介して膵癌細胞における構造とEGFに誘導されるVEGFの発現を調節する

膵臓, 2006, Vol.21(4), pp.381-383

2006

Volltextzugriff (PDF)

Inhibition of PDGFR phosphorylation and Src and Akt activity by GN963 leads to therapy of human pancreatic cancer growing orthotopically in nude mice

International journal of oncology, 2006-07, Vol.29 (1), p.125-138

2006

Volltextzugriff (PDF)

The SH4-Unique-SH3-SH2 domains dictate specificity in signaling that differentiate c-Yes from c-Src

Journal of cell science, 2003-06, Vol.116 (Pt 12), p.2585-2598

2003

Volltextzugriff (PDF)

PC phosphorylation increases the ability of AFAP-110 to cross-link actin filaments

Molecular biology of the cell, 2002-07, Vol.13 (7), p.2311-2322

2002

Volltextzugriff (PDF)

Specificity in signaling by c-Yes

Frontiers in bioscience, 2003-01, Vol.8, p.s185

2003

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The SH3 and SH2 domains are capable of directing specificity in protein interactions between the non-receptor tyrosine kinases cSrc and cYes

Oncogene, 2000-01, Vol.19 (1), p.155-160

2000

Volltextzugriff (PDF)

Abstract 2859: Dual targeting of protein tyrosine kinase c-Src and protein tyrosine phosphatase SHP-2 is a novel therapeutic strategy that induces potent inhibition of pancreatic cancer cell viability in vitro and tumor progression in vivo

Cancer research (Chicago, Ill.), 2012-04, Vol.72 (8_Supplement), p.2859-2859

2012

Volltextzugriff (PDF)

Abstract 1237: Role of Src in resistance to anoikis in detachedpancreatic cancer cells

Cancer research (Chicago, Ill.), 2011-04, Vol.71 (8_Supplement), p.1237-1237

2011

Volltextzugriff (PDF)

Abstract 4097: Protein tyrosine phosphatase SHP-2 regulates human pancreatic cancer cell biology on collagen I

Cancer research (Chicago, Ill.), 2010-04, Vol.70 (8_Supplement), p.4097-4097

2010

Volltextzugriff (PDF)

Treatment foradvancedtumors : Src reclaims center stage

Clinical cancer research, 2006, Vol.12 (5), p.1398-1401

2006

Volltextzugriff (PDF)

Src family kinases in tumor progression and metastasis : Protein kinases

Cancer and metastasis reviews, 2003, Vol.22 (4), p.337-358

2003

Volltextzugriff (PDF)

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Development and characterization of gemcitabine-resistant pancreatic tumor cells

Treatment for advanced tumors: SRC reclaims center stage

Inhibition of Src Expression and Activity Inhibits Tumor Progression and Metastasis of Human Pancreatic Adenocarcinoma Cells in an Orthotopic Nude Mouse Model

Src activation regulates anoikis in human colon tumor cell lines

Src family kinases in tumor progression and metastasis

Expression and activity of src regulate interleukin-8 expression in pancreatic adenocarcinoma cells : Implications for angiogenesis

AFAP-110 is overexpressed in prostate cancer and contributes to tumorigenic growth by regulating focal contacts

AP23846, a novel and highly potent Src family kinase inhibitor, reduces vascular endothelial growth factor and interleukin-8 expression in human solid tumor cell lines and abrogates downstream angiogenic processes

c-Src regulates constitutive and EGF-mediated VEGF expression in pancreatic tumor cells through activation of phosphatidyl inositol-3 kinase and p38 MAPK

Loss of tyrosine phosphatase-dependent inhibition promotes activation of tyrosine kinase c-Src in detached pancreatic cells

Targeting the yin and the yang: combined inhibition of the tyrosine kinase c-Src and the tyrosine phosphatase SHP-2 disrupts pancreatic cancer signaling and biology in vitro and tumor formation in vivo

SRC inhibitors as potential therapeutic agents for human cancers

Src activation of Stat3 is an independent requirement from NF-κB activation for constitutive IL-8 expression in human pancreatic adenocarcinoma cells

PKC Phosphorylation Increases the Ability of AFAP-110 to Cross-link Actin Filaments

c-SrcはPI3Kとp38 MAPKのリン酸化を介して膵癌細胞における構造とEGFに誘導されるVEGFの発現を調節する

Inhibition of PDGFR phosphorylation and Src and Akt activity by GN963 leads to therapy of human pancreatic cancer growing orthotopically in nude mice

The SH4-Unique-SH3-SH2 domains dictate specificity in signaling that differentiate c-Yes from c-Src

PC phosphorylation increases the ability of AFAP-110 to cross-link actin filaments

Specificity in signaling by c-Yes

The SH3 and SH2 domains are capable of directing specificity in protein interactions between the non-receptor tyrosine kinases cSrc and cYes

Abstract 2859: Dual targeting of protein tyrosine kinase c-Src and protein tyrosine phosphatase SHP-2 is a novel therapeutic strategy that induces potent inhibition of pancreatic cancer cell viability in vitro and tumor progression in vivo

Abstract 1237: Role of Src in resistance to anoikis in detachedpancreatic cancer cells

Abstract 4097: Protein tyrosine phosphatase SHP-2 regulates human pancreatic cancer cell biology on collagen I

Treatment foradvancedtumors : Src reclaims center stage

Src family kinases in tumor progression and metastasis : Protein kinases

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