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Obesity Induces Hypothalamic Endoplasmic Reticulum Stress and Impairs Proopiomelanocortin (POMC) Post-translational Processing

The Journal of biological chemistry, 2013-06, Vol.288 (24), p.17675-17688

2013

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Maintenance of the thyroid axis during diet-induced obesity in rodents is controlled at the central level

American journal of physiology: endocrinology and metabolism, 2010-12, Vol.299 (6), p.E976-E989

2010

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Role of the Central Melanocortin Circuitry in Adaptive Thermogenesis of Brown Adipose Tissue

Endocrinology (Philadelphia), 2007-04, Vol.148 (4), p.1550-1560

2007

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Temporal changes in nutritional state affect hypothalamic POMC peptide levels independently of leptin in adult male mice

American journal of physiology: endocrinology and metabolism, 2014-04, Vol.306 (8), p.E904-E915

2014

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Differential effects of fasting and leptin on proopiomelanocortin peptides in the arcuate nucleus and in the nucleus of the solitary tract

American journal of physiology: endocrinology and metabolism, 2007-05, Vol.292 (5), p.E1348-E1357

2007

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The Role of Intracerebroventricular Administration of Leptin in the Stimulation of Prothyrotropin Releasing Hormone Neurons in the Hypothalamic Paraventricular Nucleus

Endocrinology (Philadelphia), 2006-07, Vol.147 (7), p.3296-3306

2006

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Thyroid Hormones Selectively Regulate the Posttranslational Processing of Prothyrotropin-Releasing Hormone in the Paraventricular Nucleus of the Hypothalamus

Endocrinology (Philadelphia), 2006-06, Vol.147 (6), p.2705-2716

2006

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SIRT1 Deacetylase in POMC Neurons Is Required for Homeostatic Defenses against Diet-Induced Obesity

Cell metabolism, 2010-07, Vol.12 (1), p.78-87

2010

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Regulation of hypothalamic prohormone convertases 1 and 2 and effects on processing of prothyrotropin-releasing hormone

The Journal of clinical investigation, 2004-08, Vol.114 (3), p.357-369

2004

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Family members CREB and CREM control thyrotropin-releasing hormone (TRH) expression in the hypothalamus

Molecular and cellular endocrinology, 2013-01, Vol.365 (1), p.84-94

2013

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N-Acetylation of Hypothalamic α-Melanocyte-Stimulating Hormone and Regulation by Leptin

Proceedings of the National Academy of Sciences - PNAS, 2004-08, Vol.101 (32), p.11797-11802

2004

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Role of a Pro-sequence in the Secretory Pathway of Prothyrotropin-releasing Hormone

The Journal of biological chemistry, 2008-11, Vol.283 (46), p.31438-31448

2008

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Cold Exposure Increases the Biosynthesis and Proteolytic Processing of Prothyrotropin-Releasing Hormone in the Hypothalamic Paraventricular Nucleus via β-Adrenoreceptors

Endocrinology (Philadelphia), 2007-10, Vol.148 (10), p.4952-4964

2007

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Biosynthesis of proTRH-derived peptides in prohormone convertase 1 and 2 knockout mice

Peptides (New York, N.Y. : 1980), 2012-05, Vol.35 (1), p.42-48

2012

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Morphine treatment selectively regulates expression of rat pituitary POMC and the prohormone convertases PC1/3 and PC2

Peptides (New York, N.Y. : 1980), 2013-09, Vol.47, p.99-109

2013

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Deletion of the Nhlh2 Transcription Factor Decreases the Levels of the Anorexigenic Peptides α Melanocyte-Stimulating Hormone and Thyrotropin-Releasing Hormone and Implicates Prohormone Convertases I and II in Obesity

Endocrinology (Philadelphia), 2004-04, Vol.145 (4), p.1503-1513

2004

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Regulation of hypothalamic prohormone convertases 1 and 2 and effects on processing of prothyrotropin-releasing hormone

The Journal of clinical investigation, 2004-08, Vol.114 (3), p.357-369

2004

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Maintenance of the thyroid axis during diet-induced obesity in rodents is controlled at the central level

American journal of physiology: endocrinology and metabolism, 2010, Vol.62 (6)

2010

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Differential effects of fasting and leptin on proopiomelanocortin peptides in the arcuate nucleus and in the nucleus of the solitary tract

American journal of physiology: endocrinology and metabolism, 2007, Vol.55 (5), p.1348-1357

2007

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Cold Exposure Increases the Biosynthesis and Proteolytic Processing of Prothyrotropin-Releasing Hormone in the Hypothalamic Paraventricular Nucleus via {szligbeta}-Adrenoreceptors

Endocrinology (Philadelphia), 2007-10, Vol.148 (10), p.4952-4964

2007

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DIFFERENTIAL REGULATION OF PC1/3, PC2 AND PHOSPHORYLATED CREB BY SHORT-TERM AND LONG-TERM MORPHINE TREATMENT: IMPLICATIONS FOR UNDERSTANDING THE “SWITCH” TO OPIATE ADDICTION

Neuroscience, 2008-08, Vol.156 (3), p.788-799

2008

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N-acetylation of hypothalamic -melanocyte-stimulating hormone and regulation by leptin

Proceedings of the National Academy of Sciences - PNAS, 2004-08, Vol.101 (32), p.11797

2004

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Deletion of the Nhlh2 transcription factor decreases the levels of the anorexigenic peptides a melanocyte-stimulating hormone and thyrotropin-releasing hormone and implicates prohormone convertases I and II in obesity

Endocrinology (Philadelphia), 2004, Vol.145 (4), p.1503-1513

2004

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The enterprise knowledge dictionary

Knowledge management research & practice, 2003-12, Vol.1 (2), p.95-101

2003

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IOC consensus statement: dietary supplements and the high-performance athlete

British journal of sports medicine, 2018-04, Vol.52 (7), p.439-455

2018

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Obesity Induces Hypothalamic Endoplasmic Reticulum Stress and Impairs Proopiomelanocortin (POMC) Post-translational Processing

Maintenance of the thyroid axis during diet-induced obesity in rodents is controlled at the central level

Role of the Central Melanocortin Circuitry in Adaptive Thermogenesis of Brown Adipose Tissue

Temporal changes in nutritional state affect hypothalamic POMC peptide levels independently of leptin in adult male mice

Differential effects of fasting and leptin on proopiomelanocortin peptides in the arcuate nucleus and in the nucleus of the solitary tract

The Role of Intracerebroventricular Administration of Leptin in the Stimulation of Prothyrotropin Releasing Hormone Neurons in the Hypothalamic Paraventricular Nucleus

Thyroid Hormones Selectively Regulate the Posttranslational Processing of Prothyrotropin-Releasing Hormone in the Paraventricular Nucleus of the Hypothalamus

SIRT1 Deacetylase in POMC Neurons Is Required for Homeostatic Defenses against Diet-Induced Obesity

Regulation of hypothalamic prohormone convertases 1 and 2 and effects on processing of prothyrotropin-releasing hormone

Family members CREB and CREM control thyrotropin-releasing hormone (TRH) expression in the hypothalamus

N-Acetylation of Hypothalamic α-Melanocyte-Stimulating Hormone and Regulation by Leptin

Role of a Pro-sequence in the Secretory Pathway of Prothyrotropin-releasing Hormone

Cold Exposure Increases the Biosynthesis and Proteolytic Processing of Prothyrotropin-Releasing Hormone in the Hypothalamic Paraventricular Nucleus via β-Adrenoreceptors

Biosynthesis of proTRH-derived peptides in prohormone convertase 1 and 2 knockout mice

Morphine treatment selectively regulates expression of rat pituitary POMC and the prohormone convertases PC1/3 and PC2

Deletion of the Nhlh2 Transcription Factor Decreases the Levels of the Anorexigenic Peptides α Melanocyte-Stimulating Hormone and Thyrotropin-Releasing Hormone and Implicates Prohormone Convertases I and II in Obesity

Regulation of hypothalamic prohormone convertases 1 and 2 and effects on processing of prothyrotropin-releasing hormone

Maintenance of the thyroid axis during diet-induced obesity in rodents is controlled at the central level

Differential effects of fasting and leptin on proopiomelanocortin peptides in the arcuate nucleus and in the nucleus of the solitary tract

Cold Exposure Increases the Biosynthesis and Proteolytic Processing of Prothyrotropin-Releasing Hormone in the Hypothalamic Paraventricular Nucleus via {szligbeta}-Adrenoreceptors

DIFFERENTIAL REGULATION OF PC1/3, PC2 AND PHOSPHORYLATED CREB BY SHORT-TERM AND LONG-TERM MORPHINE TREATMENT: IMPLICATIONS FOR UNDERSTANDING THE “SWITCH” TO OPIATE ADDICTION

N-acetylation of hypothalamic -melanocyte-stimulating hormone and regulation by leptin

Deletion of the Nhlh2 transcription factor decreases the levels of the anorexigenic peptides a melanocyte-stimulating hormone and thyrotropin-releasing hormone and implicates prohormone convertases I and II in obesity

The enterprise knowledge dictionary

IOC consensus statement: dietary supplements and the high-performance athlete

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