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The Onset and Extent of Genomic Instability in Sporadic Colorectal Tumor Progression

Proceedings of the National Academy of Sciences - PNAS, 1999-12, Vol.96 (26), p.15121-15126

1999

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p53 Tumor Suppressor Gene Status and the Degree of Genomic Instability in Sporadic Colorectal Cancers

JNCI : Journal of the National Cancer Institute, 1996-11, Vol.88 (22), p.1665-1670

1996

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Genome-wide allelotyping indicates increased loss of heterozygosity on 9p and 14q in early age of onset colorectal cancer

Cytogenetic and genome research, 1999-01, Vol.86 (2), p.142-147

1999

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An anoxia inducible endonuclease and enhanced DNA breakage as contributors to genomic instability in cancer

Cancer research (Chicago, Ill.), 1995-03, Vol.55 (5), p.1122-1128

1995

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Stereotactic core biopsy breast and blood cell by-products: a source of material for molecular genetics research--initial experience

Radiology, 2001-03, Vol.218 (3), p.881-885

2001

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Normal Fibroblasts Responding to Anoxia Exhibit Features of the Malignant Phenotype

The Journal of biological chemistry, 1989-09, Vol.264 (25), p.14885-14892

1989

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Anoxic induction of a sarcoma virus-related VL30 retrotransposon is mediated by a cis-acting element which binds hypoxia-inducible factor 1 and an anoxia-inducible factor

Journal of Virology, 1995-10, Vol.69 (10), p.6335-6341

1995

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Cancer: the evolved consequence of a destabilized genome

BioEssays, 2001-11, Vol.23 (11), p.1037-1046

2001

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Germline and somatic mutation analyses in the DNA mismatch repair gene MLH3: Evidence for somatic mutation in colorectal cancers

Human mutation, 2001-05, Vol.17 (5), p.389-396

2001

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Intrachromosomal genomic instability in human sporadic colorectal cancer measured by genome-wide allelotyping and inter-(simple sequence repeat) PCR

Cancer research (Chicago, Ill.), 2001-11, Vol.61 (22), p.8274-8283

2001

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Anoxia-inducible rat VL30 elements and their relationship to ras-containing sarcoma viruses

Journal of Virology, 1993-11, Vol.67 (11), p.6857-6862

1993

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p53 tumor suppressor gene mutations predict decreased survival of patients with sporadic colorectal carcinoma

Cancer, 2000-04, Vol.88 (8), p.1814-1819

2000

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Anoxia-inducible endonuclease activity as a potential basis of the genomic instability of cancer cells

Cancer research (Chicago, Ill.), 1992-08, Vol.52 (16), p.4372-4378

1992

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De‐regulation of endogenous retrotransposons in mouse mammary carcinomas of diverse etiologies

International journal of cancer, 1993-07, Vol.54 (5), p.813-819

1993

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Mutations involving the endogenous ecotropic murine leukemia virus in primary mammary carcinomas of BALB/c mice

Cancer letters, 1996-01, Vol.99 (1), p.121-127

1996

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The Anoxic Fibroblast Response Is an Early-Stage Wound Healing Program

The Journal of surgical research, 1995-12, Vol.59 (6), p.666-674

1995

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Aberrant Crypt Foci

Anticancer research, 2006-01, Vol.26 (1A), p.107-119

2006

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Harvey sarcoma virus genome contains no extensive sequences unrelated to those of other retroviruses except ras

Journal of Virology, 1988-09, Vol.62 (9), p.3540-3543

1988

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Normal Fibroblasts Induce the C/EBPβ and ATF-4 bZIP Transcription Factors in Response to Anoxia

Experimental cell research, 1995-09, Vol.220 (1), p.47-54

1995

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Genomic instability in sporadic colorectal cancer quantitated by inter-simple sequence repeat PCR analysis

Genes chromosomes & cancer, 1997-01, Vol.18 (1), p.19-29

1997

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Retrotransposon-like VL30 elements are efficiently induced in anoxic rat fibroblasts

Journal of molecular biology, 1989-02, Vol.205 (4), p.765-769

1989

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Colorectal cancer: how does it start? How does it metastasize?

Surgical oncology clinics of North America, 2000-10, Vol.9 (4), p.643

2000

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Transformed cells overexpress a cytosolic nucleic acid binding protein

Journal of cellular physiology, 1989-10, Vol.141 (1), p.111-118

1989

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aCGH local copy number aberrations associated with overall copy number genomic instability in colorectal cancer: Coordinate involvement of the regions including BCR and ABL

Mutation Research-Fundamental and Molecular Mechanisms of Mutagenesis, 2007-02, Vol.615 (1), p.1-11

2007

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Anoxia, wound healing, VL30 elements, and the molecular basis of malignant conversion

BioEssays, 1993-04, Vol.15 (4), p.265-272

1993

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The Onset and Extent of Genomic Instability in Sporadic Colorectal Tumor Progression

p53 Tumor Suppressor Gene Status and the Degree of Genomic Instability in Sporadic Colorectal Cancers

Genome-wide allelotyping indicates increased loss of heterozygosity on 9p and 14q in early age of onset colorectal cancer

An anoxia inducible endonuclease and enhanced DNA breakage as contributors to genomic instability in cancer

Stereotactic core biopsy breast and blood cell by-products: a source of material for molecular genetics research--initial experience

Normal Fibroblasts Responding to Anoxia Exhibit Features of the Malignant Phenotype

Anoxic induction of a sarcoma virus-related VL30 retrotransposon is mediated by a cis-acting element which binds hypoxia-inducible factor 1 and an anoxia-inducible factor

Cancer: the evolved consequence of a destabilized genome

Germline and somatic mutation analyses in the DNA mismatch repair gene MLH3: Evidence for somatic mutation in colorectal cancers

Intrachromosomal genomic instability in human sporadic colorectal cancer measured by genome-wide allelotyping and inter-(simple sequence repeat) PCR

Anoxia-inducible rat VL30 elements and their relationship to ras-containing sarcoma viruses

p53 tumor suppressor gene mutations predict decreased survival of patients with sporadic colorectal carcinoma

Anoxia-inducible endonuclease activity as a potential basis of the genomic instability of cancer cells

De‐regulation of endogenous retrotransposons in mouse mammary carcinomas of diverse etiologies

Mutations involving the endogenous ecotropic murine leukemia virus in primary mammary carcinomas of BALB/c mice

The Anoxic Fibroblast Response Is an Early-Stage Wound Healing Program

Aberrant Crypt Foci

Harvey sarcoma virus genome contains no extensive sequences unrelated to those of other retroviruses except ras

Normal Fibroblasts Induce the C/EBPβ and ATF-4 bZIP Transcription Factors in Response to Anoxia

Genomic instability in sporadic colorectal cancer quantitated by inter-simple sequence repeat PCR analysis

Retrotransposon-like VL30 elements are efficiently induced in anoxic rat fibroblasts

Colorectal cancer: how does it start? How does it metastasize?

Transformed cells overexpress a cytosolic nucleic acid binding protein

aCGH local copy number aberrations associated with overall copy number genomic instability in colorectal cancer: Coordinate involvement of the regions including BCR and ABL

Anoxia, wound healing, VL30 elements, and the molecular basis of malignant conversion

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