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Genetic heterogeneity of diffuse large B-cell lymphoma

Proceedings of the National Academy of Sciences - PNAS, 2013-01, Vol.110 (4), p.1398-1403

2013

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Enteropathy-associated T cell lymphoma subtypes are characterized by loss of function of SETD2

The Journal of experimental medicine, 2017-05, Vol.214 (5), p.1371-1386

2017

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IL-10–Producing Regulatory B10 Cells Ameliorate Collagen-Induced Arthritis via Suppressing Th17 Cell Generation

The American journal of pathology, 2012-06, Vol.180 (6), p.2375-2385

2012

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Comprehensive Profiling of EBV Gene Expression and Promoter Methylation Reveals Latency II Viral Infection and Sporadic Abortive Lytic Activation in Peripheral T-Cell Lymphomas

Viruses, 2023-02, Vol.15 (2), p.423

2023

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PRDM1 is a tumor suppressor gene in natural killer cell malignancies

Proceedings of the National Academy of Sciences - PNAS, 2011-12, Vol.108 (50), p.20119-20124

2011

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The Stress-Responsive Gene GADD45G Is a Functional Tumor Suppressor, with Its Response to Environmental Stresses Frequently Disrupted Epigenetically in Multiple Tumors

Clinical cancer research, 2005-09, Vol.11 (18), p.6442-6449

2005

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The epigenetic modifier PRDM5 functions as a tumor suppressor through modulating WNT/β-catenin signaling and is frequently silenced in multiple tumors

PloS one, 2011-11, Vol.6 (11), p.e27346-e27346

2011

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Epigenetic silencing of the 3p22 tumor suppressor DLEC1 by promoter CpG methylation in non-Hodgkin and Hodgkin lymphomas

Journal of translational medicine, 2012-10, Vol.10 (1), p.209-209, Article 209

2012

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Tumor suppressor dual-specificity phosphatase 6 (DUSP6) impairs cell invasion and epithelial-mesenchymal transition (EMT)-associated phenotype

International journal of cancer, 2012-01, Vol.130 (1), p.83-95

2012

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The candidate tumor suppressor gene BLU, located at the commonly deleted region 3p21.3, is an E2F-regulated, stress-responsive gene and inactivated by both epigenetic and genetic mechanisms in nasopharyngeal carcinoma

Oncogene, 2004-06, Vol.23 (27), p.4793-4806

2004

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WNT5A antagonizes WNT/β-catenin signaling and is frequently silenced by promoter CpG methylation in esophageal squamous cell carcinoma

Cancer biology & therapy, 2010-09, Vol.10 (6), p.617-624

2010

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DJ-1 Could Predict Worse Prognosis in Esophageal Squamous Cell Carcinoma

Cancer epidemiology, biomarkers & prevention, 2008-12, Vol.17 (12), p.3593-3602

2008

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Epigenetic silencing of a Ca²⁺-regulated Ras GTPase-activating protein RASAL defines a new mechanism of Ras activation in human cancers

Proceedings of the National Academy of Sciences - PNAS, 2007-07, Vol.104 (30), p.12353-12358

2007

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Characterization of a Novel Tumor-Suppressor Gene PLCδ1 at 3p22 in Esophageal Squamous Cell Carcinoma

Cancer research (Chicago, Ill.), 2007-11, Vol.67 (22), p.10720-10726

2007

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OPCML is a broad tumor suppressor for multiple carcinomas and lymphomas with frequently epigenetic inactivation

PloS one, 2008-08, Vol.3 (8), p.e2990-e2990

2008

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Combinatorial use of bone morphogenetic protein 6, noggin and SOST significantly predicts cancer progression

Cancer science, 2012-06, Vol.103 (6), p.1145-1154

2012

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Identification of an invasion and tumor‐suppressing gene, Endoglin (ENG), silenced by both epigenetic inactivation and allelic loss in esophageal squamous cell carcinoma

International journal of cancer, 2008-12, Vol.123 (12), p.2816-2823

2008

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Impact of oncogenic driver mutations on feedback between the PI3K and MEK pathways in cancer cells

Bioscience reports, 2012-08, Vol.32 (4), p.413-422

2012

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Genetic alterations in a telomerase-immortalized human esophageal epithelial cell line: Implications for carcinogenesis

Cancer letters, 2010-07, Vol.293 (1), p.41-51

2010

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High BCL6 expression predicts better prognosis, independent of BCL6 translocation status, translocation partner, or BCL6-deregulating mutations, in gastric lymphoma

Blood, 2006-10, Vol.108 (7), p.2373-2383

2006

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The Role of Pea3 Group Transcription Factors in Esophageal Squamous Cell Carcinoma

The American journal of pathology, 2011-08, Vol.179 (2), p.992-1003

2011

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Proteasome inhibitor bortezomib-induced apoptosis in natural killer (NK)–cell leukemia and lymphoma: an in vitro and in vivo preclinical evaluation

Blood, 2007-07, Vol.110 (1), p.469-470

2007

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Frequent epigenetic silencing of protocadherin 10 by methylation in multiple haematologic malignancies

British journal of haematology, 2007-03, Vol.136 (6), p.829-832

2007

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Cell death by bortezomib-induced mitotic catastrophe in natural killer lymphoma cells

Molecular cancer therapeutics, 2008-12, Vol.7 (12), p.3807-3815

2008

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Expression and function of TNF family member B cell-activating factor in the development of autoimmune arthritis

International immunology, 2005-08, Vol.17 (8), p.1081-1092

2005

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Genetic heterogeneity of diffuse large B-cell lymphoma

Enteropathy-associated T cell lymphoma subtypes are characterized by loss of function of SETD2

IL-10–Producing Regulatory B10 Cells Ameliorate Collagen-Induced Arthritis via Suppressing Th17 Cell Generation

Comprehensive Profiling of EBV Gene Expression and Promoter Methylation Reveals Latency II Viral Infection and Sporadic Abortive Lytic Activation in Peripheral T-Cell Lymphomas

PRDM1 is a tumor suppressor gene in natural killer cell malignancies

The Stress-Responsive Gene GADD45G Is a Functional Tumor Suppressor, with Its Response to Environmental Stresses Frequently Disrupted Epigenetically in Multiple Tumors

The epigenetic modifier PRDM5 functions as a tumor suppressor through modulating WNT/β-catenin signaling and is frequently silenced in multiple tumors

Epigenetic silencing of the 3p22 tumor suppressor DLEC1 by promoter CpG methylation in non-Hodgkin and Hodgkin lymphomas

Tumor suppressor dual-specificity phosphatase 6 (DUSP6) impairs cell invasion and epithelial-mesenchymal transition (EMT)-associated phenotype

The candidate tumor suppressor gene BLU, located at the commonly deleted region 3p21.3, is an E2F-regulated, stress-responsive gene and inactivated by both epigenetic and genetic mechanisms in nasopharyngeal carcinoma

WNT5A antagonizes WNT/β-catenin signaling and is frequently silenced by promoter CpG methylation in esophageal squamous cell carcinoma

DJ-1 Could Predict Worse Prognosis in Esophageal Squamous Cell Carcinoma

Epigenetic silencing of a Ca²⁺-regulated Ras GTPase-activating protein RASAL defines a new mechanism of Ras activation in human cancers

Characterization of a Novel Tumor-Suppressor Gene PLCδ1 at 3p22 in Esophageal Squamous Cell Carcinoma

OPCML is a broad tumor suppressor for multiple carcinomas and lymphomas with frequently epigenetic inactivation

Combinatorial use of bone morphogenetic protein 6, noggin and SOST significantly predicts cancer progression

Identification of an invasion and tumor‐suppressing gene, Endoglin (ENG), silenced by both epigenetic inactivation and allelic loss in esophageal squamous cell carcinoma

Impact of oncogenic driver mutations on feedback between the PI3K and MEK pathways in cancer cells

Genetic alterations in a telomerase-immortalized human esophageal epithelial cell line: Implications for carcinogenesis

High BCL6 expression predicts better prognosis, independent of BCL6 translocation status, translocation partner, or BCL6-deregulating mutations, in gastric lymphoma

The Role of Pea3 Group Transcription Factors in Esophageal Squamous Cell Carcinoma

Proteasome inhibitor bortezomib-induced apoptosis in natural killer (NK)–cell leukemia and lymphoma: an in vitro and in vivo preclinical evaluation

Frequent epigenetic silencing of protocadherin 10 by methylation in multiple haematologic malignancies

Cell death by bortezomib-induced mitotic catastrophe in natural killer lymphoma cells

Expression and function of TNF family member B cell-activating factor in the development of autoimmune arthritis

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