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The molecular mechanics of mixed lineage leukemia

Oncogene, 2016-10, Vol.35 (40), p.5215-5223

Slany, R K

2016

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Alterations of the CxxC domain preclude oncogenic activation of mixed-lineage leukemia 2

Oncogene, 2009-02, Vol.28 (6), p.815-823

2009

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Insulin-like growth factor 1 is a direct HOXA9 target important for hematopoietic transformation

Leukemia, 2015-04, Vol.29 (4), p.901-908

2015

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Efficacy of cyclin-dependent-kinase 9 inhibitors in a murine model of mixed-lineage leukemia

Leukemia, 2014-07, Vol.28 (7), p.1427-1435

2014

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From initiation to eradication: the lifespan of an MLL-rearranged therapy-related paediatric AML

Bone marrow transplantation (Basingstoke), 2015-10, Vol.50 (10), p.1382-1384

2015

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The molecular biology of mixed lineage leukemia

Haematologica (Roma), 2009-07, Vol.94 (7), p.984-993

Slany, Robert K

2009

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Hoxa9 and Meis1 Are Key Targets for MLL-ENL-Mediated Cellular Immortalization

Molecular and Cellular Biology, 2004-01, Vol.24 (2), p.617-628

2004

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ALK fusion genes in children with atypical myeloproliferative leukemia

Leukemia, 2010-06, Vol.24 (6), p.1197-1200

2010

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HOX genes regulate Rac1 activity in hematopoietic cells through control of Vav2 expression

Leukemia, 2013-01, Vol.27 (1), p.236-238

2013

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The Oncoprotein MLL-ENL disturbs hematopoietic lineage determination and transforms a biphenotypic lymphoid myeloid cell

Oncogene, 2003-03, Vol.22 (11), p.1629-1637

2003

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The MT domain of the proto-oncoprotein MLL binds to CpG-containing DNA and discriminates against methylation

Nucleic acids research, 2002-02, Vol.30 (4), p.958-965

2002

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The eleven-nineteen-leukemia protein ENL connects nuclear MLL fusion partners with chromatin

Oncogene, 2005-08, Vol.24 (35), p.5525-5532

2005

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The ENL moiety of the childhood leukemia-associated MLL-ENL oncoprotein recruits human Polycomb 3

Oncogene, 2001-01, Vol.20 (4), p.411-419

2001

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Distinct regulation of c-myb gene expression by HoxA9, Meis1 and Pbx proteins in normal hematopoietic progenitors and transformed myeloid cells

Blood cancer journal (New York), 2012-06, Vol.2 (6), p.e76-e76

2012

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Transcriptional activation is a key function encoded by MLL fusion partners

Leukemia, 2003-02, Vol.17 (2), p.359-365

2003

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Expression of cell-cell interacting genes distinguishes HLXB9 TEL from MLL-positive childhood acute myeloid leukemia

Leukemia, 2010-09, Vol.24 (9), p.1657-1660

2010

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The Oncogenic Capacity of HRX-ENL Requires the Transcriptional Transactivation Activity of ENL and the DNA Binding Motifs of HRX

Molecular and Cellular Biology, 1998-01, Vol.18 (1), p.122-129

1998

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Pbx3 and Meis1 cooperate through multiple mechanisms to support Hox-induced murine leukemia

Haematologica (Roma), 2015-07, Vol.100 (7), p.905-913

2015

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The formin-binding protein 17, FBP17, binds via a TNKS binding motif to tankyrase, a protein involved in telomere maintenance

FEBS letters, 2003-11, Vol.554 (1-2), p.10-16

2003

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The leukemogenic fusion of MLL with ENL creates a novel transcriptional transactivator

Leukemia, 1999-10, Vol.13 (10), p.1525-1533

1999

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ENL, the MLL fusion partner in t(11;19), binds to the c-Abl interactor protein 1 (ABI1) that is fused to MLL in t(10;11)

Oncogene, 2000-03, Vol.19 (14), p.1744-1751

2000

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ArgBP2, encoding a negative regulator of ABL, is fused to MLL in a case of infant M5 acute myeloid leukemia involving 4q35 and 11q23

Leukemia, 2006-07, Vol.20 (7), p.1310-1313

2006

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MLL-ENL causes a reversible and myc-dependent block of myelomonocytic cell differentiation

Cancer research (Chicago, Ill.), 2001-09, Vol.61 (17), p.6480-6486

2001

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Research recommendations toward a better understanding of the causes of childhood leukemia

Blood cancer journal (New York), 2011-01, Vol.1 (1), p.e1-e1

2011

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Dimerization of MLL fusion proteins immortalizes hematopoietic cells

Cancer cell, 2003-09, Vol.4 (3), p.197-207

2003

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The molecular mechanics of mixed lineage leukemia

Alterations of the CxxC domain preclude oncogenic activation of mixed-lineage leukemia 2

Insulin-like growth factor 1 is a direct HOXA9 target important for hematopoietic transformation

Efficacy of cyclin-dependent-kinase 9 inhibitors in a murine model of mixed-lineage leukemia

From initiation to eradication: the lifespan of an MLL-rearranged therapy-related paediatric AML

The molecular biology of mixed lineage leukemia

Hoxa9 and Meis1 Are Key Targets for MLL-ENL-Mediated Cellular Immortalization

ALK fusion genes in children with atypical myeloproliferative leukemia

HOX genes regulate Rac1 activity in hematopoietic cells through control of Vav2 expression

The Oncoprotein MLL-ENL disturbs hematopoietic lineage determination and transforms a biphenotypic lymphoid myeloid cell

The MT domain of the proto-oncoprotein MLL binds to CpG-containing DNA and discriminates against methylation

The eleven-nineteen-leukemia protein ENL connects nuclear MLL fusion partners with chromatin

The ENL moiety of the childhood leukemia-associated MLL-ENL oncoprotein recruits human Polycomb 3

Distinct regulation of c-myb gene expression by HoxA9, Meis1 and Pbx proteins in normal hematopoietic progenitors and transformed myeloid cells

Transcriptional activation is a key function encoded by MLL fusion partners

Expression of cell-cell interacting genes distinguishes HLXB9 TEL from MLL-positive childhood acute myeloid leukemia

The Oncogenic Capacity of HRX-ENL Requires the Transcriptional Transactivation Activity of ENL and the DNA Binding Motifs of HRX

Pbx3 and Meis1 cooperate through multiple mechanisms to support Hox-induced murine leukemia

The formin-binding protein 17, FBP17, binds via a TNKS binding motif to tankyrase, a protein involved in telomere maintenance

The leukemogenic fusion of MLL with ENL creates a novel transcriptional transactivator

ENL, the MLL fusion partner in t(11;19), binds to the c-Abl interactor protein 1 (ABI1) that is fused to MLL in t(10;11)

ArgBP2, encoding a negative regulator of ABL, is fused to MLL in a case of infant M5 acute myeloid leukemia involving 4q35 and 11q23

MLL-ENL causes a reversible and myc-dependent block of myelomonocytic cell differentiation

Research recommendations toward a better understanding of the causes of childhood leukemia

Dimerization of MLL fusion proteins immortalizes hematopoietic cells

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