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Cytokine induction of tumor necrosis factor receptor 2 is mediated by STAT3 in colon cancer cells

Molecular cancer research, 2011-12, Vol.9 (12), p.1718-1731

2011

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Insulin Resistance, Apoptosis, and Colorectal Adenoma Risk

Cancer epidemiology, biomarkers & prevention, 2005-09, Vol.14 (9), p.2076-2081

2005

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miR-30 Family Controls Proliferation and Differentiation of Intestinal Epithelial Cell Models by Directing a Broad Gene Expression Program That Includes SOX9 and the Ubiquitin Ligase Pathway

The Journal of biological chemistry, 2016-07, Vol.291 (31), p.15975-15984

2016

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Insulin receptor isoform switching in intestinal stem cells, progenitors, differentiated lineages and tumors: evidence that IR-B limits proliferation

Journal of cell science, 2013-12, Vol.126 (Pt 24), p.5645-5656

2013

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Tumor Necrosis Factor (TNF) α Increases Collagen Accumulation and Proliferation in Intestinal Myofibroblasts via TNF Receptor 2

The Journal of biological chemistry, 2005-10, Vol.280 (43), p.36099-36109

2005

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Local IGFBP-3 mRNA expression, apoptosis and risk of colorectal adenomas

BMC cancer, 2008-05, Vol.8 (1), p.143-143, Article 143

2008

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Haplotype Insufficiency for Suppressor of Cytokine Signaling-2 Enhances Intestinal Growth and Promotes Polyp Formation in Growth Hormone-Transgenic Mice

Endocrinology (Philadelphia), 2006-04, Vol.147 (4), p.1632-1641

2006

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Insulin-like growth factor-I is expressed by avian flexor tendon cells

Journal of orthopaedic research, 2000-07, Vol.18 (4), p.546-556

2000

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Insulin Receptor Substrate-1 Deficiency Promotes Apoptosis in the Putative Intestinal Crypt Stem Cell Region, Limits Apcmin/+ Tumors, and Regulates Sox9

Endocrinology (Philadelphia), 2008-01, Vol.149 (1), p.261-267

2008

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IGF-I and TGF-β1 have distinct effects on phenotype and proliferation of intestinal fibroblasts

American journal of physiology: Gastrointestinal and liver physiology, 2002-09, Vol.283 (3), p.G809-G818

2002

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Suppressor of cytokine signaling-2 gene disruption promotes Apc(Min/+) tumorigenesis and activator protein-1 activation

The American journal of pathology, 2010-05, Vol.176 (5), p.2320

2010

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Growth Hormone Reduces the Severity of Fibrosis Associated With Chronic Intestinal Inflammation

Gastroenterology (New York, N.Y. 1943), 2005-07, Vol.129 (1), p.204-219

2005

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Suppressor of cytokine signaling-2: a growth hormone-inducible inhibitor of intestinal epithelial cell proliferation

Gastroenterology (New York, N.Y. 1943), 2004-08, Vol.127 (2), p.570-581

2004

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Suppressor of Cytokine Signaling-2 Gene Disruption Promotes ApcMin/+ Tumorigenesis and Activator Protein-1 Activation

The American journal of pathology, 2010-05, Vol.176 (5), p.2320-2332

2010

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Suppressor of cytokine signaling-2 modulates the fibrogenic actions of GH and IGF-I in intestinal mesenchymal cells

American journal of physiology: Gastrointestinal and liver physiology, 2005-08, Vol.289 (2), p.G342-G350

2005

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Suppressor of cytokine signaling-2 limits intestinal growth and enterotrophic actions of IGF-I in vivo

American journal of physiology: Gastrointestinal and liver physiology, 2006-09, Vol.291 (3), p.G472-G481

2006

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Autocrine and paracrine actions of intestinal fibroblast-derived insulin-like growth factors

American journal of physiology: Gastrointestinal and liver physiology, 1999-04, Vol.276 (4), p.G817-G827

1999

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miR-30 Family Controls Proliferation and Differentiation of Intestinal Epithelial Cell Models by Directing a Broad Gene Expression Program That Includes SOX9 and the Ubiquitin Ligase Pathway<inline-graphic xlink:href="sbox.jpg"/>

The Journal of biological chemistry, 2016-06, Vol.291 (31), p.15975-15984

2016

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IGF‐I and EGF promote synergistic increases in proliferation, enhances IGF‐IR activation and increases nuclear β‐catenin in intestinal epithelial cells

The FASEB journal, 2009-04, Vol.23 (S1)

2009

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The tumor suppressor SOCS3 limits STAT3 binding to the TNFR2 promoter and promotes proteasomal degradation of TNFR2

The FASEB journal, 2009-04, Vol.23 (S1)

2009

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A new animal model of post-surgical bowel inflammation and fibrosis: the effect of commensal microflora

Gut, 2009-04, Vol.58 (8), p.1104-1112

2009

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Matrix and Mesenchymal Remodeling

Inflammatory bowel diseases, 2006-04, Vol.12, p.S7-S8

2006

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Suppressor of cytokine signaling-2 limits intestinal growth and enterotrophic actions of IGF-I in vivo

American journal of physiology: Gastrointestinal and liver physiology, 2006-09, Vol.291 (3), p.29

2006

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Insulin Receptor Substrate-1 Deficiency Promotes Apoptosis in the Putative Intestinal Crypt Stem Cell Region, Limits Apc super(min/+) Tumors, and Regulates Sox9

Endocrinology (Philadelphia), 2008-01, Vol.149 (1), p.261-267

2008

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Tumor Necrosis Factor (TNF) Î± Increases Collagen Accumulation and Proliferation in Intestinal Myofibroblasts via TNF Receptor 2

The Journal of biological chemistry, 2005-10, Vol.280 (43), p.36099

2005

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Cytokine induction of tumor necrosis factor receptor 2 is mediated by STAT3 in colon cancer cells

Insulin Resistance, Apoptosis, and Colorectal Adenoma Risk

miR-30 Family Controls Proliferation and Differentiation of Intestinal Epithelial Cell Models by Directing a Broad Gene Expression Program That Includes SOX9 and the Ubiquitin Ligase Pathway

Insulin receptor isoform switching in intestinal stem cells, progenitors, differentiated lineages and tumors: evidence that IR-B limits proliferation

Tumor Necrosis Factor (TNF) α Increases Collagen Accumulation and Proliferation in Intestinal Myofibroblasts via TNF Receptor 2

Local IGFBP-3 mRNA expression, apoptosis and risk of colorectal adenomas

Haplotype Insufficiency for Suppressor of Cytokine Signaling-2 Enhances Intestinal Growth and Promotes Polyp Formation in Growth Hormone-Transgenic Mice

Insulin-like growth factor-I is expressed by avian flexor tendon cells

Insulin Receptor Substrate-1 Deficiency Promotes Apoptosis in the Putative Intestinal Crypt Stem Cell Region, Limits Apcmin/+ Tumors, and Regulates Sox9

IGF-I and TGF-β1 have distinct effects on phenotype and proliferation of intestinal fibroblasts

Suppressor of cytokine signaling-2 gene disruption promotes Apc(Min/+) tumorigenesis and activator protein-1 activation

Growth Hormone Reduces the Severity of Fibrosis Associated With Chronic Intestinal Inflammation

Suppressor of cytokine signaling-2: a growth hormone-inducible inhibitor of intestinal epithelial cell proliferation

Suppressor of Cytokine Signaling-2 Gene Disruption Promotes ApcMin/+ Tumorigenesis and Activator Protein-1 Activation

Suppressor of cytokine signaling-2 modulates the fibrogenic actions of GH and IGF-I in intestinal mesenchymal cells

Suppressor of cytokine signaling-2 limits intestinal growth and enterotrophic actions of IGF-I in vivo

Autocrine and paracrine actions of intestinal fibroblast-derived insulin-like growth factors

miR-30 Family Controls Proliferation and Differentiation of Intestinal Epithelial Cell Models by Directing a Broad Gene Expression Program That Includes SOX9 and the Ubiquitin Ligase Pathway<inline-graphic xlink:href="sbox.jpg"/>

IGF‐I and EGF promote synergistic increases in proliferation, enhances IGF‐IR activation and increases nuclear β‐catenin in intestinal epithelial cells

The tumor suppressor SOCS3 limits STAT3 binding to the TNFR2 promoter and promotes proteasomal degradation of TNFR2

A new animal model of post-surgical bowel inflammation and fibrosis: the effect of commensal microflora

Matrix and Mesenchymal Remodeling

Suppressor of cytokine signaling-2 limits intestinal growth and enterotrophic actions of IGF-I in vivo

Insulin Receptor Substrate-1 Deficiency Promotes Apoptosis in the Putative Intestinal Crypt Stem Cell Region, Limits Apc super(min/+) Tumors, and Regulates Sox9

Tumor Necrosis Factor (TNF) Î± Increases Collagen Accumulation and Proliferation in Intestinal Myofibroblasts via TNF Receptor 2

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