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Disruption of the insulin-like growth factor-1 gene in osteocytes impairs developmental bone growth in mice

Bone (New York, N.Y.), 2013-01, Vol.52 (1), p.133-144

2013

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A Novel EphA4 Signaling‐Based Therapeutic Strategy for Osteoarthritis in Mice

Journal of bone and mineral research, 2022-04, Vol.37 (4), p.660-674

2022

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Defective bone repletion in aged Balb/cBy mice was caused by impaired osteoblastic differentiation

Journal of bone and mineral metabolism, 2022-11, Vol.40 (6), p.900-913

2022

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Conditional deletion of IGF-I in osteocytes unexpectedly accelerates bony Union of the Fracture Gap in mice

Bone (New York, N.Y.), 2016-11, Vol.92, p.18-28

2016

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Osteocyte-derived insulin-like growth factor I is essential for determining bone mechanosensitivity

American journal of physiology: endocrinology and metabolism, 2013-07, Vol.305 (2), p.E271-E281

2013

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Osteocyte-derived insulin-like growth factor I is not essential for the bone repletion response in mice

PloS one, 2015-01, Vol.10 (1), p.e0115897-e0115897

2015

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An Osteoclastic Transmembrane Protein-Tyrosine Phosphatase Enhances Osteoclast Activity in Part by Dephosphorylating EphA4 in Osteoclasts

Journal of cellular biochemistry, 2015-08, Vol.116 (8), p.1785-1796

2015

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Unique Regenerative Mechanism to Replace Bone Lost During Dietary Bone Depletion in Weanling Mice

Endocrinology (Philadelphia), 2017-04, Vol.158 (4), p.714-729

2017

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Osteoactivin is a novel osteoclastic protein and plays a key role in osteoclast differentiation and activity

FEBS letters, 2008-04, Vol.582 (10), p.1451-1458

2008

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Targeted overexpression of osteoactivin in cells of osteoclastic lineage promotes osteoclastic resorption and bone loss in mice

PloS one, 2012-04, Vol.7 (4), p.e35280-e35280

2012

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Calcium released by osteoclastic resorption stimulates autocrine/paracrine activities in local osteogenic cells to promote coupled bone formation

American Journal of Physiology: Cell Physiology, 2022-05, Vol.322 (5), p.C977-C990

2022

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A Mouse Noninvasive Intraarticular Tibial Plateau Compression Loading-Induced Injury Model of Posttraumatic Osteoarthritis

Calcified tissue international, 2020-02, Vol.106 (2), p.158-171

2020

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Targeted Transgenic Expression of an Osteoclastic Transmembrane Protein-tyrosine Phosphatase in Cells of Osteoclastic Lineage Increases Bone Resorption and Bone Loss in Male Young Adult Mice

The Journal of biological chemistry, 2009-04, Vol.284 (17), p.11531-11545

2009

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Detection of dinitrosyl iron complexes by ozone-based chemiluminescence

Nitric oxide, 2018-09, Vol.79, p.57-67

2018

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EphA4 Receptor Is a Novel Negative Regulator of Osteoclast Activity

Journal of bone and mineral research, 2014-04, Vol.29 (4), p.804-819

2014

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Bax deficiency in mice increases cartilage production during fracture repair through a mechanism involving increased chondrocyte proliferation without changes in apoptosis

Bone (New York, N.Y.), 2008-11, Vol.43 (5), p.880-888

2008

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Local ex vivo gene therapy with bone marrow stromal cells expressing human BMP4 promotes endosteal bone formation in mice

The journal of gene medicine, 2004-01, Vol.6 (1), p.4-15

2004

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An osteoclastic protein-tyrosine phosphatase is a potential positive regulator of the c-Src protein-tyrosine kinase activity: A mediator of osteoclast activity

Journal of cellular biochemistry, 2006-04, Vol.97 (5), p.940-955

2006

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Genetic variation in femur extrinsic strength in 29 different inbred strains of mice is dependent on variations in femur cross-sectional geometry and bone density

Bone (New York, N.Y.), 2005-01, Vol.36 (1), p.111-122

2005

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A novel miR17/protein tyrosine phosphatase-oc/EphA4 regulatory axis of osteoclast activity

Archives of biochemistry and biophysics, 2018-07, Vol.650, p.30-38

2018

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Conditional Disruption of miR17~92 in Osteoclasts Led to Activation of Osteoclasts and Loss of Trabecular Bone In Part Through Suppression of the miR17-Mediated Downregulation of Protein-Tyrosine Phosphatase-oc in Mice

JBMR plus, 2017-10, Vol.1 (2), p.73-85

2017

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The EphA4 Signaling is Anti-catabolic in Synoviocytes but Pro-anabolic in Articular Chondrocytes

Calcified tissue international, 2020-12, Vol.107 (6), p.576-592

2020

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Retroviral-based gene therapy with cyclooxygenase-2 promotes the union of bony callus tissues and accelerates fracture healing in the rat

The journal of gene medicine, 2008-03, Vol.10 (3), p.229-241

2008

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In vivo and in vitro evidence that the high osteoblastic activity in C3H/HeJ mice compared to C57BL/6J mice is intrinsic to bone cells

Bone (New York, N.Y.), 2004-09, Vol.35 (3), p.711-719

2004

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In vivo bone formation in fracture repair induced by direct retroviral-based gene therapy with bone morphogenetic protein-4

Bone (New York, N.Y.), 2003-06, Vol.32 (6), p.591-601

2003

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Disruption of the insulin-like growth factor-1 gene in osteocytes impairs developmental bone growth in mice

A Novel EphA4 Signaling‐Based Therapeutic Strategy for Osteoarthritis in Mice

Defective bone repletion in aged Balb/cBy mice was caused by impaired osteoblastic differentiation

Conditional deletion of IGF-I in osteocytes unexpectedly accelerates bony Union of the Fracture Gap in mice

Osteocyte-derived insulin-like growth factor I is essential for determining bone mechanosensitivity

Osteocyte-derived insulin-like growth factor I is not essential for the bone repletion response in mice

An Osteoclastic Transmembrane Protein-Tyrosine Phosphatase Enhances Osteoclast Activity in Part by Dephosphorylating EphA4 in Osteoclasts

Unique Regenerative Mechanism to Replace Bone Lost During Dietary Bone Depletion in Weanling Mice

Osteoactivin is a novel osteoclastic protein and plays a key role in osteoclast differentiation and activity

Targeted overexpression of osteoactivin in cells of osteoclastic lineage promotes osteoclastic resorption and bone loss in mice

Calcium released by osteoclastic resorption stimulates autocrine/paracrine activities in local osteogenic cells to promote coupled bone formation

A Mouse Noninvasive Intraarticular Tibial Plateau Compression Loading-Induced Injury Model of Posttraumatic Osteoarthritis

Targeted Transgenic Expression of an Osteoclastic Transmembrane Protein-tyrosine Phosphatase in Cells of Osteoclastic Lineage Increases Bone Resorption and Bone Loss in Male Young Adult Mice

Detection of dinitrosyl iron complexes by ozone-based chemiluminescence

EphA4 Receptor Is a Novel Negative Regulator of Osteoclast Activity

Bax deficiency in mice increases cartilage production during fracture repair through a mechanism involving increased chondrocyte proliferation without changes in apoptosis

Local ex vivo gene therapy with bone marrow stromal cells expressing human BMP4 promotes endosteal bone formation in mice

An osteoclastic protein-tyrosine phosphatase is a potential positive regulator of the c-Src protein-tyrosine kinase activity: A mediator of osteoclast activity

Genetic variation in femur extrinsic strength in 29 different inbred strains of mice is dependent on variations in femur cross-sectional geometry and bone density

A novel miR17/protein tyrosine phosphatase-oc/EphA4 regulatory axis of osteoclast activity

Conditional Disruption of miR17~92 in Osteoclasts Led to Activation of Osteoclasts and Loss of Trabecular Bone In Part Through Suppression of the miR17-Mediated Downregulation of Protein-Tyrosine Phosphatase-oc in Mice

The EphA4 Signaling is Anti-catabolic in Synoviocytes but Pro-anabolic in Articular Chondrocytes

Retroviral-based gene therapy with cyclooxygenase-2 promotes the union of bony callus tissues and accelerates fracture healing in the rat

In vivo and in vitro evidence that the high osteoblastic activity in C3H/HeJ mice compared to C57BL/6J mice is intrinsic to bone cells

In vivo bone formation in fracture repair induced by direct retroviral-based gene therapy with bone morphogenetic protein-4

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