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Lipopolysaccharide-induced-neuroinflammation increases intracellular accumulation of amyloid precursor protein and amyloid β peptide in APPswe transgenic mice

Neurobiology of disease, 2003-10, Vol.14 (1), p.133-145

2003

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Neuronal-glial interactions mediated by interleukin-1 enhance neuronal acetylcholinesterase activity and mRNA expression

The Journal of neuroscience, 2000-01, Vol.20 (1), p.149-155

2000

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Interleukin-1 promotion of MAPK-p38 overexpression in experimental animals and in Alzheimer's disease: potential significance for tau protein phosphorylation

Neurochemistry international, 2001-11, Vol.39 (5), p.341-348

2001

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Glial cytokines in Alzheimer's disease: Review and pathogenic implications

Human pathology, 1995-08, Vol.26 (8), p.816-823

1995

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The β-amyloid-related proteins presenilin 1 and BACE1 are axonally transported to nerve terminals in the brain

Experimental neurology, 2003-12, Vol.184 (2), p.1053-1057

2003

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Neuritic plaque evolution in Alzheimer's disease is accompanied by transition of activated microglia from primed to enlarged to phagocytic forms

Acta neuropathologica, 1997-07, Vol.94 (1), p.1-5

1997

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Glial-Neuronal Interactions in Alzheimer Disease: Progressive Association of IL-1α+ Microglia and S100β+ Astrocytes with Neurofibrillary Tangle Stages

Journal of neuropathology and experimental neurology, 1997-03, Vol.56 (3), p.285-290

1997

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Correlation of Astrocytic S100β Expression with Dystrophic Neurites in Amyloid Plaques of Alzheimerʼs Disease

Journal of neuropathology and experimental neurology, 1996-03, Vol.55 (3), p.273-279

1996

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S100β Increases Levels of β‐Amyloid Precursor Protein and Its Encoding mRNA in Rat Neuronal Cultures

Journal of neurochemistry, 1998-10, Vol.71 (4), p.1421-1428

1998

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Progressive Neuronal Injury Associated with Amyloid Plaque Formation in Alzheimer Disease

Journal of neuropathology and experimental neurology, 1998-07, Vol.57 (7), p.714-717

1998

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Progressive Neuronal DNA Damage Associated with Neurofibrillary Tangle Formation in Alzheimer Disease

Journal of neuropathology and experimental neurology, 1998-04, Vol.57 (4), p.323-328

1998

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Alterations in striatal glial fibrillary acidic protein expression in response to 6-hydroxydopamine-induced denervation

Experimental brain research, 1993-08, Vol.95 (3), p.450-456

1993

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Disruption of Corticocortical Connections Ameliorates Amyloid Burden in Terminal Fields in a Transgenic Model of Aβ Amyloidosis

The Journal of neuroscience, 2002-11, Vol.22 (22), p.9794-9799

2002

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Enlarged and phagocytic, but not primed, interleukin-1α-immunoreactive microglia increase with age in normal human brain

Acta neuropathologica, 1998-03, Vol.95 (3), p.229-234

1998

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Increased Neuronal β‐Amyloid Precursor Protein Expression in Human Temporal Lobe Epilepsy: Association with Interleukin‐1α Immunoreactivity

Journal of neurochemistry, 1994-11, Vol.63 (5), p.1872-1879

1994

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Human brain S100β and S100β mRNA expression increases with age: Pathogenic implications for Alzheimer's disease

Neurobiology of aging, 1996-05, Vol.17 (3), p.359-363

1996

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China cardiovascular diseases report 2015： a summary

Journal of geriatric cardiology : JGC, 2017, Vol.14 (1), p.1-10

2017

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Microglial interleukin-1α expression in human head injury : correlations with neuronal and neuritic β-amyloid precursor protein expression

Neuroscience letters, 1994-08, Vol.176 (2), p.133-136

1994

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Microglial interleukin-lα expression in human head injury: Correlations with neuronal and neuritic β-amyloid precursor protein expression

Neuroscience letters, 1994-08, Vol.176 (2), p.133-136

1994

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Disruption of Corticocortical Connections Ameliorates Amyloid Burden in Terminal Fields in a Transgenic Model of Abeta Amyloidosis

The Journal of neuroscience, 2002-11, Vol.22 (22), p.9794-9799

2002

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Thirty years (1984–2014) of groundwater science teaching and research in China: A dissertation-based bibliometric survey

Journal of Groundwater Science and Engineering, 2015-09, Vol.3 (3), p.222-237

2015

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Phylogenetic beta diversity in tropical forests： Implications for the roles of geographical and environmental distance

Journal of systematics and evolution : JSE, 2013, Vol.51 (1), p.71-85

2013

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On Coleman Automorphisms of Finite Nilpotent Groups by Cyclic Groups

Acta mathematica Sinica. English series, 2016-12, Vol.32 (12), p.1459-1464

2016

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PN401 decreases neurodegeneration in models of Alzheimer’s, Huntington’s and Parkinson’s disease, neuroinflammation, excitotoxicity and has antiepileptic activity

Alzheimer's & dementia, 2005-07, Vol.1 (1), p.S68-S68

2005

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Neuronal-Glial Interactions Mediated by Interleukin-1 Enhance Neuronal Acety1cholinesterase Activity and mRNA Expression

The Journal of neuroscience, 2000-01, Vol.20 (1), p.149-155

2000

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Lipopolysaccharide-induced-neuroinflammation increases intracellular accumulation of amyloid precursor protein and amyloid β peptide in APPswe transgenic mice

Neuronal-glial interactions mediated by interleukin-1 enhance neuronal acetylcholinesterase activity and mRNA expression

Interleukin-1 promotion of MAPK-p38 overexpression in experimental animals and in Alzheimer's disease: potential significance for tau protein phosphorylation

Glial cytokines in Alzheimer's disease: Review and pathogenic implications

The β-amyloid-related proteins presenilin 1 and BACE1 are axonally transported to nerve terminals in the brain

Neuritic plaque evolution in Alzheimer's disease is accompanied by transition of activated microglia from primed to enlarged to phagocytic forms

Glial-Neuronal Interactions in Alzheimer Disease: Progressive Association of IL-1α+ Microglia and S100β+ Astrocytes with Neurofibrillary Tangle Stages

Correlation of Astrocytic S100β Expression with Dystrophic Neurites in Amyloid Plaques of Alzheimerʼs Disease

S100β Increases Levels of β‐Amyloid Precursor Protein and Its Encoding mRNA in Rat Neuronal Cultures

Progressive Neuronal Injury Associated with Amyloid Plaque Formation in Alzheimer Disease

Progressive Neuronal DNA Damage Associated with Neurofibrillary Tangle Formation in Alzheimer Disease

Alterations in striatal glial fibrillary acidic protein expression in response to 6-hydroxydopamine-induced denervation

Disruption of Corticocortical Connections Ameliorates Amyloid Burden in Terminal Fields in a Transgenic Model of Aβ Amyloidosis

Enlarged and phagocytic, but not primed, interleukin-1α-immunoreactive microglia increase with age in normal human brain

Increased Neuronal β‐Amyloid Precursor Protein Expression in Human Temporal Lobe Epilepsy: Association with Interleukin‐1α Immunoreactivity

Human brain S100β and S100β mRNA expression increases with age: Pathogenic implications for Alzheimer's disease

China cardiovascular diseases report 2015： a summary

Microglial interleukin-1α expression in human head injury : correlations with neuronal and neuritic β-amyloid precursor protein expression

Microglial interleukin-lα expression in human head injury: Correlations with neuronal and neuritic β-amyloid precursor protein expression

Disruption of Corticocortical Connections Ameliorates Amyloid Burden in Terminal Fields in a Transgenic Model of Abeta Amyloidosis

Thirty years (1984–2014) of groundwater science teaching and research in China: A dissertation-based bibliometric survey

Phylogenetic beta diversity in tropical forests： Implications for the roles of geographical and environmental distance

On Coleman Automorphisms of Finite Nilpotent Groups by Cyclic Groups

PN401 decreases neurodegeneration in models of Alzheimer’s, Huntington’s and Parkinson’s disease, neuroinflammation, excitotoxicity and has antiepileptic activity

Neuronal-Glial Interactions Mediated by Interleukin-1 Enhance Neuronal Acety1cholinesterase Activity and mRNA Expression

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