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Antagonists of excitatory opioid receptor functions enhance morphine's analgesic potency and attenuate opioid tolerance/dependence liability

Pain (Amsterdam), 2000-02, Vol.84 (2-3), p.121-131

2000

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Acute thermal hyperalgesia elicited by low-dose morphine in normal mice is blocked by ultra-low-dose naltrexone, unmasking potent opioid analgesia

Brain research, 2001-01, Vol.888 (1), p.75-82

2001

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Complete Genome Sequence of Trueperella pyogenes, Isolated from Infected Farmland Goats

Genome announcements (Washington, DC), 2016-12, Vol.4 (6)

2016

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Rapid detection of sacbrood virus (SBV) by one-step reverse transcription loop-mediated isothermal amplification assay

Virology journal, 2012-02, Vol.9 (1), p.47-47, Article 47

2012

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Neuraminidase inhibitor, oseltamivir blocks GM1 ganglioside-regulated excitatory opioid receptor-mediated hyperalgesia, enhances opioid analgesia and attenuates tolerance in mice

Brain research, 2004-01, Vol.995 (2), p.260-266

2004

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Cholera toxin-B subunit blocks excitatory opioid receptor-mediated hyperalgesic effects in mice, thereby unmasking potent opioid analgesia and attenuating opioid tolerance/dependence

Brain research, 2001-11, Vol.919 (1), p.20-30

2001

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Enhanced analgesic potency and reduced tolerance of morphine in 129/SvEv mice: evidence for a deficiency in GM1 ganglioside-regulated excitatory opioid receptor functions

Brain research, 2000-02, Vol.856 (1), p.227-235

2000

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Opioids can evoke direct receptor-mediated excitatory effects on sensory neurons

Trends in pharmacological sciences (Regular ed.), 1990-02, Vol.11 (2), p.77-81

1990

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Low doses of cyclic AMP-phosphodiesterase inhibitors rapidly evoke opioid receptor-mediated thermal hyperalgesia in naïve mice which is converted to prominent analgesia by cotreatment with ultra-low-dose naltrexone

Brain research, 2008-09, Vol.1231, p.16-24

2008

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Naloxone rapidly evokes endogenous kappa opioid receptor-mediated hyperalgesia in naïve mice pretreated briefly with GM1 ganglioside or in chronic morphine-dependent mice

Brain research, 2007-09, Vol.1167, p.31-41

2007

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Antagonists at excitatory opioid receptors on sensory neurons in culture increase potency and specificity of opiate analgesics and attenuate development of tolerance/dependence

Brain research, 1994-02, Vol.636 (2), p.286-297

1994

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Rapid Detection of Sacbrood Virus (SBV) by One-Step RT-PCR Assay

Journal of animal and veterinary advances : JAVA, 2012-01, Vol.11 (7), p.931-932

2012

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Ultra-Low Concentrations of Naloxone Selectively Antagonize Excitatory Effects of Morphine on Sensory Neurons, Thereby Increasing its Antinociceptive Potency and Attenuating Tolerance/Dependence During Chronic Cotreatment

Proceedings of the National Academy of Sciences - PNAS, 1995-11, Vol.92 (23), p.10540-10544

1995

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Ultra-low doses of naltrexone or etorphine increase morphine's antinociceptive potency and attenuate tolerance/dependence in mice

Brain research, 1997-05, Vol.757 (2), p.176-190

1997

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Cloning and Phylogenetic Analysis of NS1 Gene of H3N2 Subtype Swine Influenza Virus from Different Isolates in Chongqing China 2010-2011

Journal of animal and veterinary advances : JAVA, 2012-01, Vol.11 (12), p.2126-2128

2012

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Diagnosis of Etiological Agent on Caprine Mycoplasma pneumonia in Chongqing China

Journal of animal and veterinary advances : JAVA, 2012-01, Vol.11 (13), p.2247-2249

2012

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GM1 Ganglioside-induced Modulation of Opioid Receptor-mediated Functions

Annals of the New York Academy of Sciences, 1998-06, Vol.845 (1), p.106-125

1998

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Effects of temperature alterations on population and cellular activities in hippocampal slices from mature and immature rabbit

Brain research, 1988-12, Vol.475 (2), p.305-316

1988

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Chronic selective activation of excitatory opioid receptor functions in sensory neurons results in opioid ‘dependence’ without tolerance

Brain research, 1992-11, Vol.597 (1), p.74-83

1992

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Biphalin, an enkephalin analog with unexpectedly high antinociceptive potency and low dependence liability in vivo, selectively antagonizes excitatory opioid receptor functions of sensory neurons in culture

Brain research, 1995-12, Vol.701 (1), p.158-166

1995

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Modulatory effects of Gs-coupled excitatory opioid receptor functions on opioid analgesia, tolerance, and dependence

Neurochemical research, 1996-11, Vol.21 (11), p.1347-1351

1996

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Chronic morphine-treated sensory ganglion neurons remain supersensitive to the excitatory effects of naloxone for months after return to normal culture medium: an in vitro model of ‘protracted opioid dependence’

Brain research, 1995-10, Vol.694 (1), p.103-110

1995

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Cholera toxin-B subunit blocks excitatory effects of opioids on sensory neuron action potentials indicating that GM1 ganglioside may regulate GS-linked opioid receptor functions

Brain research, 1990-10, Vol.531 (1-2), p.1-7

1990

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Specific N- or C-terminus modified dynorphin and β-endorphin peptides can selectively block excitatory opioid receptor functions in sensory neurons and unmask potent inhibitory effects of opioid agonists

Brain research, 1995-02, Vol.673 (1), p.30-38

1995

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Nerve growth factor rapidly prolongs the action potential of mature sensory ganglion neurons in culture, and this effect requires activation of Gs-coupled excitatory kappa-opioid receptors on these cells

The Journal of neuroscience, 1994-09, Vol.14 (9), p.5570-5579

1994

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Antagonists of excitatory opioid receptor functions enhance morphine's analgesic potency and attenuate opioid tolerance/dependence liability

Acute thermal hyperalgesia elicited by low-dose morphine in normal mice is blocked by ultra-low-dose naltrexone, unmasking potent opioid analgesia

Complete Genome Sequence of Trueperella pyogenes, Isolated from Infected Farmland Goats

Rapid detection of sacbrood virus (SBV) by one-step reverse transcription loop-mediated isothermal amplification assay

Neuraminidase inhibitor, oseltamivir blocks GM1 ganglioside-regulated excitatory opioid receptor-mediated hyperalgesia, enhances opioid analgesia and attenuates tolerance in mice

Cholera toxin-B subunit blocks excitatory opioid receptor-mediated hyperalgesic effects in mice, thereby unmasking potent opioid analgesia and attenuating opioid tolerance/dependence

Enhanced analgesic potency and reduced tolerance of morphine in 129/SvEv mice: evidence for a deficiency in GM1 ganglioside-regulated excitatory opioid receptor functions

Opioids can evoke direct receptor-mediated excitatory effects on sensory neurons

Low doses of cyclic AMP-phosphodiesterase inhibitors rapidly evoke opioid receptor-mediated thermal hyperalgesia in naïve mice which is converted to prominent analgesia by cotreatment with ultra-low-dose naltrexone

Naloxone rapidly evokes endogenous kappa opioid receptor-mediated hyperalgesia in naïve mice pretreated briefly with GM1 ganglioside or in chronic morphine-dependent mice

Antagonists at excitatory opioid receptors on sensory neurons in culture increase potency and specificity of opiate analgesics and attenuate development of tolerance/dependence

Rapid Detection of Sacbrood Virus (SBV) by One-Step RT-PCR Assay

Ultra-Low Concentrations of Naloxone Selectively Antagonize Excitatory Effects of Morphine on Sensory Neurons, Thereby Increasing its Antinociceptive Potency and Attenuating Tolerance/Dependence During Chronic Cotreatment

Ultra-low doses of naltrexone or etorphine increase morphine's antinociceptive potency and attenuate tolerance/dependence in mice

Cloning and Phylogenetic Analysis of NS1 Gene of H3N2 Subtype Swine Influenza Virus from Different Isolates in Chongqing China 2010-2011

Diagnosis of Etiological Agent on Caprine Mycoplasma pneumonia in Chongqing China

GM1 Ganglioside-induced Modulation of Opioid Receptor-mediated Functions

Effects of temperature alterations on population and cellular activities in hippocampal slices from mature and immature rabbit

Chronic selective activation of excitatory opioid receptor functions in sensory neurons results in opioid ‘dependence’ without tolerance

Biphalin, an enkephalin analog with unexpectedly high antinociceptive potency and low dependence liability in vivo, selectively antagonizes excitatory opioid receptor functions of sensory neurons in culture

Modulatory effects of Gs-coupled excitatory opioid receptor functions on opioid analgesia, tolerance, and dependence

Chronic morphine-treated sensory ganglion neurons remain supersensitive to the excitatory effects of naloxone for months after return to normal culture medium: an in vitro model of ‘protracted opioid dependence’

Cholera toxin-B subunit blocks excitatory effects of opioids on sensory neuron action potentials indicating that GM1 ganglioside may regulate GS-linked opioid receptor functions

Specific N- or C-terminus modified dynorphin and β-endorphin peptides can selectively block excitatory opioid receptor functions in sensory neurons and unmask potent inhibitory effects of opioid agonists

Nerve growth factor rapidly prolongs the action potential of mature sensory ganglion neurons in culture, and this effect requires activation of Gs-coupled excitatory kappa-opioid receptors on these cells

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