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Isoflurane-induced inactivation of CREB through histone deacetylase 4 is responsible for cognitive impairment in developing brain

Neurobiology of disease, 2016-12, Vol.96, p.12-21

2016

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Nitrosylation of GAPDH augments pathological tau acetylation upon exposure to amyloid-β

Science signaling, 2018-03, Vol.11 (522)

2018

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Histone Deacetylase 4 Downregulation Elicits Post-Traumatic Psychiatric Disorders through Impairment of Neurogenesis

Journal of neurotrauma, 2019-12, Vol.36 (23), p.3284-3296

2019

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Activation of cyclin D1 affects mitochondrial mass following traumatic brain injury

Neurobiology of disease, 2018-10, Vol.118, p.108-116

2018

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Microglial–oligodendrocyte interactions in myelination and neurological function recovery after traumatic brain injury

Journal of neuroinflammation, 2022-10, Vol.19 (1), p.1-246, Article 246

2022

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OGDHL is a modifier of AKT-dependent signaling and NF-κB function

PloS one, 2012-11, Vol.7 (11), p.e48770-e48770

2012

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Corrigendum to ‘Treatment with an activator of hypoxia-inducible factor 1, DMOG provides neuroprotection after traumatic brain injury’ [Neuropharmacology 107 (2016) 79–88]

Neuropharmacology, 2021-09, Vol.195, p.108536-108536, Article 108536

2021

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Activation of PERK Elicits Memory Impairment through Inactivation of CREB and Downregulation of PSD95 After Traumatic Brain Injury

The Journal of neuroscience, 2017-06, Vol.37 (24), p.5900-5911

2017

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ATM kinase is a master switch for the ΔNp63α phosphorylation/degradation in human head and neck squamous cell carcinoma cells upon DNA damage

Cell cycle (Georgetown, Tex.), 2008-09, Vol.7 (18), p.2846-2855

2008

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Sulfhydration of AKT triggers Tau-phosphorylation by activating glycogen synthase kinase 3β in Alzheimer’s disease

Proceedings of the National Academy of Sciences - PNAS, 2020-02, Vol.117 (8), p.4418-4427

2020

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Yes-associated protein 1 regulates the stability of ΔNp63α

Cell cycle (Georgetown, Tex.), 2010-01, Vol.9 (1), p.162-167

2010

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Cytokine-Induced GAPDH Sulfhydration Affects PSD95 Degradation and Memory

Molecular cell, 2014-12, Vol.56 (6), p.786-795

2014

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VEGF 165b in the developing vasculatures of the fetal human eye

Developmental dynamics, 2012-03, Vol.241 (3), p.595-607

2012

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Treatment with an activator of hypoxia-inducible factor 1, DMOG provides neuroprotection after traumatic brain injury

Neuropharmacology, 2016-08, Vol.107, p.79-88

2016

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Aberrant ER Stress Induced Neuronal-IFNβ Elicits White Matter Injury Due to Microglial Activation and T-Cell Infiltration after TBI

The Journal of neuroscience, 2020-01, Vol.40 (2), p.424-446

2020

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Regulation of ΔNp63α by NFκΒ

Cell cycle (Georgetown, Tex.), 2010-12, Vol.9 (24), p.4841-4847

2010

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An augmentation in histone dimethylation at lysine nine residues elicits vision impairment following traumatic brain injury

Free radical biology & medicine, 2019-04, Vol.134, p.630-643

2019

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Diet-driven microbiota dysbiosis is associated with vagal remodeling and obesity

Physiology & behavior, 2017-05, Vol.173, p.305-317

2017

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Hydrogen Sulfide-Linked Sulfhydration of NF-κB Mediates Its Antiapoptotic Actions

Molecular cell, 2012-01, Vol.45 (1), p.13-24

2012

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Energy-dense diet triggers changes in gut microbiota, reorganization of gut‑brain vagal communication and increases body fat accumulation

Acta neurobiologiae experimentalis, 2017-01, Vol.77 (1), p.18-30

2017

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ΔNp63α Confers Tumor Cell Resistance to Cisplatin through the AKT1 Transcriptional Regulation

Cancer research (Chicago, Ill.), 2011-02, Vol.71 (3), p.1167-1176

2011

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Dopamine Oxidation Products Inhibit Na+, K+-ATPase Activity in Crude Synaptosomal-mitochondrial Fraction from Rat Brain

Free radical research, 2003-06, Vol.37 (6), p.597-601

2003

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Lipid peroxidation associated cardiolipin loss and membrane depolarization in rat brain mitochondria

Neurochemistry international, 2006-07, Vol.49 (1), p.20-27

2006

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Regulation of p53 Family Member Isoform ΔNp63α by the Nuclear Factor-κB Targeting Kinase IκB Kinase β

Cancer research (Chicago, Ill.), 2010-02, Vol.70 (4), p.1419-1429

2010

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Regulation of ΔNp63α by NFκB

Cell cycle (Georgetown, Tex.), 2010-12, Vol.9 (24), p.4841-4847

2010

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Isoflurane-induced inactivation of CREB through histone deacetylase 4 is responsible for cognitive impairment in developing brain

Nitrosylation of GAPDH augments pathological tau acetylation upon exposure to amyloid-β

Histone Deacetylase 4 Downregulation Elicits Post-Traumatic Psychiatric Disorders through Impairment of Neurogenesis

Activation of cyclin D1 affects mitochondrial mass following traumatic brain injury

Microglial–oligodendrocyte interactions in myelination and neurological function recovery after traumatic brain injury

OGDHL is a modifier of AKT-dependent signaling and NF-κB function

Corrigendum to ‘Treatment with an activator of hypoxia-inducible factor 1, DMOG provides neuroprotection after traumatic brain injury’ [Neuropharmacology 107 (2016) 79–88]

Activation of PERK Elicits Memory Impairment through Inactivation of CREB and Downregulation of PSD95 After Traumatic Brain Injury

ATM kinase is a master switch for the ΔNp63α phosphorylation/degradation in human head and neck squamous cell carcinoma cells upon DNA damage

Sulfhydration of AKT triggers Tau-phosphorylation by activating glycogen synthase kinase 3β in Alzheimer’s disease

Yes-associated protein 1 regulates the stability of ΔNp63α

Cytokine-Induced GAPDH Sulfhydration Affects PSD95 Degradation and Memory

VEGF 165b in the developing vasculatures of the fetal human eye

Treatment with an activator of hypoxia-inducible factor 1, DMOG provides neuroprotection after traumatic brain injury

Aberrant ER Stress Induced Neuronal-IFNβ Elicits White Matter Injury Due to Microglial Activation and T-Cell Infiltration after TBI

Regulation of ΔNp63α by NFκΒ

An augmentation in histone dimethylation at lysine nine residues elicits vision impairment following traumatic brain injury

Diet-driven microbiota dysbiosis is associated with vagal remodeling and obesity

Hydrogen Sulfide-Linked Sulfhydration of NF-κB Mediates Its Antiapoptotic Actions

Energy-dense diet triggers changes in gut microbiota, reorganization of gut‑brain vagal communication and increases body fat accumulation

ΔNp63α Confers Tumor Cell Resistance to Cisplatin through the AKT1 Transcriptional Regulation

Dopamine Oxidation Products Inhibit Na+, K+-ATPase Activity in Crude Synaptosomal-mitochondrial Fraction from Rat Brain

Lipid peroxidation associated cardiolipin loss and membrane depolarization in rat brain mitochondria

Regulation of p53 Family Member Isoform ΔNp63α by the Nuclear Factor-κB Targeting Kinase IκB Kinase β

Regulation of ΔNp63α by NFκB

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