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Neurodegenerative Effects of Recombinant HIV-1 Tat(1-86) are Associated with Inhibition of Microtubule Formation and Oxidative Stress-Related Reductions in Microtubule-Associated Protein-2(a,b)

Neurochemical research, 2011-05, Vol.36 (5), p.819-828

2011

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The human immunodeficiency virus type-1 transcription factor Tat produces elevations in intracellular Ca2+ that require function of an N-methyl-D-aspartate receptor polyamine-sensitive site

Brain research, 2004-01, Vol.995 (1), p.39-45

2004

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Neurotoxic effects of the human immunodeficiency virus type-1 transcription factor Tat require function of a polyamine sensitive-site on the N-methyl- d-aspartate receptor

Brain research, 2002-11, Vol.954 (2), p.300-307

2002

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Potentiation of N -methyl- d -aspartate receptor-mediated neuronal injury during methamphetamine withdrawal in vitro requires co-activation of IP3 receptors

Brain research, 2008-01, Vol.1187, p.67-73

2008

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Corticosterone Increases Damage and Cytosolic Calcium Accumulation Associated With Ethanol Withdrawal in Rat Hippocampal Slice Cultures

Alcoholism, clinical and experimental research, 2005-05, Vol.29 (5), p.871-881

2005

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Ethanol Exposure and Withdrawal Sensitizes the Rat Hippocampal CA1 Pyramidal Cell Region to β-Amyloid (25-35)-Induced Cytotoxicity: NMDA Receptor Involvement

Alcoholism, clinical and experimental research, 2005-11, Vol.29 (11), p.2063-2069

2005

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Methamphetamine exposure antagonizes N -methyl- d -aspartate receptor-mediated neurotoxicity in organotypic hippocampal slice cultures

Brain research, 2007-07, Vol.1157, p.74-80

2007

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Choline exposure reduces potentiation of N-methyl- d-aspartate toxicity by corticosterone in the developing hippocampus

Brain research. Developmental brain research, 2004-11, Vol.153 (2), p.203-211

2004

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Sex Differences in the Neurotoxic Effects of Adenosine A1 Receptor Antagonism During Ethanol Withdrawal: Reversal With an A1 Receptor Agonist or an NMDA Receptor Antagonist

Alcoholism, clinical and experimental research, 2008-07, Vol.32 (7), p.1260-1270

2008

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Cytotoxic Effects of Exposure to the Human Immunodeficiency Virus Type 1 Protein Tat in the Hippocampus Are Enhanced by Prior Ethanol Treatment

Alcoholism, clinical and experimental research, 2004-12, Vol.28 (12), p.1916-1924

2004

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A 24 h corticosterone exposure exacerbates excitotoxic insult in rat hippocampal slice cultures independently of glucocorticoid receptor activation or protein synthesis

Brain research, 2006-04, Vol.1082 (1), p.165-172

2006

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Selective vulnerability of hippocampal cornu ammonis 1 pyramidal cells to excitotoxic insult is associated with the expression of polyamine-sensitive N -methyl- d -asparate-type glutamate receptors

Neuroscience, 2010-01, Vol.165 (2), p.525-534

2010

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Opposing effects of ethanol and nicotine on hippocampal calbindin-D 28k expression

Alcohol (Fayetteville, N.Y.), 2003, Vol.31 (1), p.1-10

2003

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Sex Differences in the Neurotoxic Effects of Adenosine A 1 Receptor Antagonism During Ethanol Withdrawal: Reversal With an A 1 Receptor Agonist or an NMDA Receptor Antagonist

Alcoholism, clinical and experimental research, 2008-07, Vol.32 (7), p.1260-1270

2008

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Intra-cornu ammonis 1 administration of the human immunodeficiency virus-1 transcription factor Tat exacerbates the ethanol withdrawal syndrome in rodents and activates N-methyl-d-aspartate glutamate receptors to produce persisting spatial learning deficits

Neuroscience, 2009-07, Vol.163 (3), p.868-876

2009

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Potentiation of N-methyl- d-aspartate receptor-mediated neuronal injury during methamphetamine withdrawal in vitro requires co-activation of IP 3 receptors

Brain research, 2008, Vol.1187, p.67-73

2008

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Exercise for depression: efficacy, safety and clinical trial implications

Psychopharmacology bulletin, 2008, Vol.41 (4), p.65

2008

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Ethanol Exposure and Withdrawal Sensitizes the Rat Hippocampal CA1 Pyramidal Cell Region to ??-Amyloid (25-35)-Induced Cytotoxicity: NMDA Receptor Involvement

Alcoholism, clinical and experimental research, 2005-11, Vol.29 (11), p.2063-2069

2005

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The human immunodeficiency virus type-1 transcription factor Tat produces elevations in intracellular Ca 2+ that require function of an N-methyl- d-aspartate receptor polyamine-sensitive site

Brain research, 2004, Vol.995 (1), p.39-45

2004

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Sex Differences in the Neurotoxic Effects of Adenosine A1Receptor Antagonism During Ethanol Withdrawal: Reversal With an A1Receptor Agonist or an NMDA Receptor Antagonist

Alcoholism, clinical and experimental research, 2008-07, Vol.32 (7), p.1260-1270

2008

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Opposing effects of ethanol and nicotine on hippocampal calbindin-D28k expression

Alcohol (Fayetteville, N.Y.), 2003-08, Vol.31 (1-2), p.1-10

2003

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Chapter 46 - Oxidative Stress, Micronutrients, and HIV Dementia

Diet and Nutrition in Dementia and Cognitive Decline, 2015, p.501-509

2015

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A Neutralizing Monoclonal Antibody for Hospitalized Patients with Covid-19

The New England journal of medicine, 2021-03, Vol.384 (10), p.905-914

2021

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Direct Regulation of Diurnal Drd3 Expression and Cocaine Reward by NPAS2

Biological psychiatry (1969), 2015-03, Vol.77 (5), p.425-433

2015

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Racial and Ethnic Disaggregation of Tuberculosis Incidence and Risk Factors Among American Indian and Alaska Native Persons-United States, 2001-2020

American journal of public health (1971), 2024-02, Vol.114 (2), p.226-236

2024

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Neurodegenerative Effects of Recombinant HIV-1 Tat(1-86) are Associated with Inhibition of Microtubule Formation and Oxidative Stress-Related Reductions in Microtubule-Associated Protein-2(a,b)

The human immunodeficiency virus type-1 transcription factor Tat produces elevations in intracellular Ca2+ that require function of an N-methyl-D-aspartate receptor polyamine-sensitive site

Neurotoxic effects of the human immunodeficiency virus type-1 transcription factor Tat require function of a polyamine sensitive-site on the N-methyl- d-aspartate receptor

Potentiation of N -methyl- d -aspartate receptor-mediated neuronal injury during methamphetamine withdrawal in vitro requires co-activation of IP3 receptors

Corticosterone Increases Damage and Cytosolic Calcium Accumulation Associated With Ethanol Withdrawal in Rat Hippocampal Slice Cultures

Ethanol Exposure and Withdrawal Sensitizes the Rat Hippocampal CA1 Pyramidal Cell Region to β-Amyloid (25-35)-Induced Cytotoxicity: NMDA Receptor Involvement

Methamphetamine exposure antagonizes N -methyl- d -aspartate receptor-mediated neurotoxicity in organotypic hippocampal slice cultures

Choline exposure reduces potentiation of N-methyl- d-aspartate toxicity by corticosterone in the developing hippocampus

Sex Differences in the Neurotoxic Effects of Adenosine A1 Receptor Antagonism During Ethanol Withdrawal: Reversal With an A1 Receptor Agonist or an NMDA Receptor Antagonist

Cytotoxic Effects of Exposure to the Human Immunodeficiency Virus Type 1 Protein Tat in the Hippocampus Are Enhanced by Prior Ethanol Treatment

A 24 h corticosterone exposure exacerbates excitotoxic insult in rat hippocampal slice cultures independently of glucocorticoid receptor activation or protein synthesis

Selective vulnerability of hippocampal cornu ammonis 1 pyramidal cells to excitotoxic insult is associated with the expression of polyamine-sensitive N -methyl- d -asparate-type glutamate receptors

Opposing effects of ethanol and nicotine on hippocampal calbindin-D 28k expression

Sex Differences in the Neurotoxic Effects of Adenosine A 1 Receptor Antagonism During Ethanol Withdrawal: Reversal With an A 1 Receptor Agonist or an NMDA Receptor Antagonist

Intra-cornu ammonis 1 administration of the human immunodeficiency virus-1 transcription factor Tat exacerbates the ethanol withdrawal syndrome in rodents and activates N-methyl-d-aspartate glutamate receptors to produce persisting spatial learning deficits

Potentiation of N-methyl- d-aspartate receptor-mediated neuronal injury during methamphetamine withdrawal in vitro requires co-activation of IP 3 receptors

Exercise for depression: efficacy, safety and clinical trial implications

Ethanol Exposure and Withdrawal Sensitizes the Rat Hippocampal CA1 Pyramidal Cell Region to ??-Amyloid (25-35)-Induced Cytotoxicity: NMDA Receptor Involvement

The human immunodeficiency virus type-1 transcription factor Tat produces elevations in intracellular Ca 2+ that require function of an N-methyl- d-aspartate receptor polyamine-sensitive site

Sex Differences in the Neurotoxic Effects of Adenosine A1Receptor Antagonism During Ethanol Withdrawal: Reversal With an A1Receptor Agonist or an NMDA Receptor Antagonist

Opposing effects of ethanol and nicotine on hippocampal calbindin-D28k expression

Chapter 46 - Oxidative Stress, Micronutrients, and HIV Dementia

A Neutralizing Monoclonal Antibody for Hospitalized Patients with Covid-19

Direct Regulation of Diurnal Drd3 Expression and Cocaine Reward by NPAS2

Racial and Ethnic Disaggregation of Tuberculosis Incidence and Risk Factors Among American Indian and Alaska Native Persons-United States, 2001-2020

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