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Clinical effects of electromagnetic and electric fields on fracture healing

Clinical orthopaedics and related research, 1998-10, Vol.355S (355), p.205-215

RYABY, J. T

1998

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Three‐Dimensional Morphometry of the L6 Vertebra in the Ovariectomized Rat Model of Osteoporosis: Biomechanical Implications

Journal of bone and mineral research, 2000-10, Vol.15 (10), p.1981-1991

2000

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Computational methods for ultrasonic bone assessment

Ultrasound in medicine & biology, 1999-06, Vol.25 (5), p.823-830

1999

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Thrombin peptide (TP508) treatment of rat growth plate cartilage cells promotes proliferation and retention of the chondrocytic phenotype while blocking terminal endochondral differentiation

Journal of cellular physiology, 2005-02, Vol.202 (2), p.336-343

2005

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Pulsed electromagnetic field stimulation may improve fusion rates in cervical arthrodesis in high-risk populations

Bone & joint research, 2018-02, Vol.7 (2), p.124-130

2018

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Microarray analysis of gene expression during the inflammation and endochondral bone formation stages of rat femur fracture repair

Bone (New York, N.Y.), 2006-04, Vol.38 (4), p.521-529

2006

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Direct modulation of osteoblastic activity with estrogen

Journal of bone and joint surgery. American volume, 1994-05, Vol.76 (5), p.713

1994

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Combined magnetic fields increased net calcium flux in bone cells

Calcified tissue international, 1994-11, Vol.55 (5), p.376-380

1994

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IGF‐II receptor number is increased in TE‐85 osteosarcoma cells by combined magnetic fields

Journal of bone and mineral research, 1995-05, Vol.10 (5), p.812-819

1995

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Bone formation is enhanced by thrombin-related peptide TP508 during distraction osteogenesis

Journal of orthopaedic research, 2005, Vol.23 (1), p.196-202

2005

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Ultrasonic assessment of human and bovine trabecular bone: a comparison study

IEEE transactions on biomedical engineering, 1996-03, Vol.43 (3), p.249-258

1996

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Tissue Engineered Bone Repair of Calvarial Defects Using Cultured Periosteal Cells

Plastic and reconstructive surgery (1963), 1998-03, Vol.101 (3), p.567-574

1998

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Thrombin peptide TP508 prevents nitric oxide mediated apoptosis in chondrocytes in the endochondral developmental pathway

Biochimica et biophysica acta, 2008-01, Vol.1783 (1), p.12-22

2008

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Influence of marrow on ultrasonic velocity and attenuation in bovine trabecular bone

Calcified tissue international, 1996-05, Vol.58 (5), p.362-367

1996

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An orientation distribution function for trabecular bone

Bone (New York, N.Y.), 2005-02, Vol.36 (2), p.193-201

2005

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Repair of rabbit segmental defects with the thrombin peptide, TP508

Journal of orthopaedic research, 2004-09, Vol.22 (5), p.1094-1099

2004

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Resonant markers for noninvasive, three-dimensional dynamic bone histomorphometry with x-ray microtomography

Review of scientific instruments, 2001-03, Vol.72 (3), p.1921-1923

2001

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Fracture healing and micro architecture

Advances in experimental medicine and biology, 2001, Vol.496, p.99

2001

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Evaluation of early osteochondral defect repair in a rabbit model utilizing fourier transform-infrared imaging spectroscopy, magnetic resonance imaging, and quantitative T2 mapping

Tissue engineering. Part C, Methods, 2010-06, Vol.16 (3), p.355

2010

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TP508 accelerates fracture repair by promoting cell growth over cell death

Biochemical and biophysical research communications, 2007-12, Vol.364 (1), p.187-193

2007

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Osteotropic agents induce the differential secretion of granulocyte‐macrophage colony‐stimulating factor by the osteoblast cell line MC3T3‐E1

Journal of bone and mineral research, 1989-12, Vol.4 (6), p.911-921

1989

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Thrombin peptide (TP508) promotes fracture repair by up-regulating inflammatory mediators, early growth factors, and increasing angiogenesis

Journal of orthopaedic research, 2005-05, Vol.23 (3), p.671-679

2005

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Combined magnetic fields accelerate and increase spine fusion: a double-blind, randomized, placebo controlled study

Spine (Philadelphia, Pa. 1976), 2002-07, Vol.27 (13), p.1383-1388

2002

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Early signals for fracture healing

Journal of cellular biochemistry, 2005-05, Vol.95 (1), p.189-205

2005

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The non-proteolytically active thrombin peptide TP508 stimulates angiogenic sprouting

Journal of cellular physiology, 2006-01, Vol.206 (1), p.175-180

2006

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Clinical effects of electromagnetic and electric fields on fracture healing

Three‐Dimensional Morphometry of the L6 Vertebra in the Ovariectomized Rat Model of Osteoporosis: Biomechanical Implications

Computational methods for ultrasonic bone assessment

Thrombin peptide (TP508) treatment of rat growth plate cartilage cells promotes proliferation and retention of the chondrocytic phenotype while blocking terminal endochondral differentiation

Pulsed electromagnetic field stimulation may improve fusion rates in cervical arthrodesis in high-risk populations

Microarray analysis of gene expression during the inflammation and endochondral bone formation stages of rat femur fracture repair

Direct modulation of osteoblastic activity with estrogen

Combined magnetic fields increased net calcium flux in bone cells

IGF‐II receptor number is increased in TE‐85 osteosarcoma cells by combined magnetic fields

Bone formation is enhanced by thrombin-related peptide TP508 during distraction osteogenesis

Ultrasonic assessment of human and bovine trabecular bone: a comparison study

Tissue Engineered Bone Repair of Calvarial Defects Using Cultured Periosteal Cells

Thrombin peptide TP508 prevents nitric oxide mediated apoptosis in chondrocytes in the endochondral developmental pathway

Influence of marrow on ultrasonic velocity and attenuation in bovine trabecular bone

An orientation distribution function for trabecular bone

Repair of rabbit segmental defects with the thrombin peptide, TP508

Resonant markers for noninvasive, three-dimensional dynamic bone histomorphometry with x-ray microtomography

Fracture healing and micro architecture

Evaluation of early osteochondral defect repair in a rabbit model utilizing fourier transform-infrared imaging spectroscopy, magnetic resonance imaging, and quantitative T2 mapping

TP508 accelerates fracture repair by promoting cell growth over cell death

Osteotropic agents induce the differential secretion of granulocyte‐macrophage colony‐stimulating factor by the osteoblast cell line MC3T3‐E1

Thrombin peptide (TP508) promotes fracture repair by up-regulating inflammatory mediators, early growth factors, and increasing angiogenesis

Combined magnetic fields accelerate and increase spine fusion: a double-blind, randomized, placebo controlled study

Early signals for fracture healing

The non-proteolytically active thrombin peptide TP508 stimulates angiogenic sprouting

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