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Reduced Immune Response to Borrelia burgdorferi in the Absence of γδ T Cells

Infection and Immunity, 2011-10, Vol.79 (10), p.3940-3946

2011

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c-FLIPS reduces activation of caspase and NF-κB pathways and decreases T cell survival

European journal of immunology, 2008, Vol.38 (1), p.54-63

2008

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Proteolytic Regulation of Nuclear Factor of Activated T (NFAT) c2 Cells and NFAT Activity by Caspase-3

The Journal of biological chemistry, 2006-04, Vol.281 (16), p.10682-10690

2006

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The c-FLIPL Cleavage Product p43FLIP Promotes Activation of Extracellular Signal-regulated Kinase (ERK), Nuclear Factor κB (NF-κB), and Caspase-8 and T Cell Survival

The Journal of biological chemistry, 2014-01, Vol.289 (2), p.1183-1191

2014

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Cellular FLIP Long Form-Transgenic Mice Manifest a Th2 Cytokine Bias and Enhanced Allergic Airway Inflammation

The Journal of immunology (1950), 2004-04, Vol.172 (8), p.4724-4732

2004

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Cellular FLIP (long form) regulates CD8+ T cell activation through caspase-8-dependent NF-kappa B activation

The Journal of immunology (1950), 2005-05, Vol.174 (9), p.5270-5278

2005

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Fas Ligand Deficiency Impairs Host Inflammatory Response against Infection with the Spirochete Borrelia burgdorferi

Infection and Immunity, 2006-02, Vol.74 (2), p.1156-1160

2006

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Activation of gamma delta T cells by Borrelia burgdorferi is indirect via a TLR- and caspase-dependent pathway

The Journal of immunology (1950), 2008-08, Vol.181 (4), p.2392-2398

2008

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Cellular FLIP Long Form Augments Caspase Activity and Death of T Cells through Heterodimerization with and Activation of Caspase-8

The Journal of immunology (1950), 2005-07, Vol.175 (1), p.311-318

2005

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Effector CD4+ T Cells Generate Intermediate Caspase Activity and Cleavage of Caspase-8 Substrates

The Journal of immunology (1950), 2005-04, Vol.174 (7), p.3999-4009

2005

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Liver Damage by Infiltrating CD8+ T Cells Is Fas Dependent

The Journal of immunology (1950), 2001-12, Vol.167 (11), p.6654-6662

2001

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Activation of γδ T Cells by Borrelia burgdorferi Is Indirect via a TLR- and Caspase-Dependent Pathway

The Journal of immunology (1950), 2008-08, Vol.181 (4), p.2392-2398

2008

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Caspase-8 and c-FLIPL Associate in Lipid Rafts with NF-κB Adaptors during T Cell Activation

The Journal of biological chemistry, 2007-07, Vol.282 (27), p.19365-19374

2007

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Spatial differences in active caspase-8 defines its role in T-cell activation versus cell death

Cell death and differentiation, 2008-11, Vol.15 (11), p.1701-1711

2008

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Anti-CD40L Accelerates Renal Disease and Adenopathy in MRL-lpr Mice in Parallel with Decreased Thymocyte Apoptosis

The Journal of immunology (1950), 1998-07, Vol.161 (2), p.729-739

1998

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A Model for the Origin of TCR-αβ+ CD4−CD8− B220+ Cells Based on High Affinity TCR Signals

The Journal of immunology (1950), 1999-05, Vol.162 (10), p.5747-5756

1999

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Activation of γδ T Cells by Borrelia burgdorferi is Indirect via a TLR- and Caspase-Dependent Pathway1

The Journal of immunology (1950), 2008-08, Vol.181 (4), p.2392-2398

2008

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c‐FLIP S reduces activation of caspase and NF‐κB pathways and decreases T cell survival

European journal of immunology, 2008-01, Vol.38 (1), p.54-63

2008

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c-FLIP(S) reduces activation of caspase and NF-kappaB pathways and decreases T cell survival

European journal of immunology, 2008-01, Vol.38 (1), p.54-63

2008

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Cellular FLIP (Long Form) Regulates CD8 super(+) T Cell Activation through Caspase-8-Dependent NF- Kappa B Activation

The Journal of immunology (1950), 2005-05, Vol.174 (9), p.5270-5278

2005

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Caspase-8 and c-FLIPL associate in lipid rafts with NF-kappaB adaptors during T cell activation

The Journal of biological chemistry, 2007-07, Vol.282 (27), p.19365-19374

2007

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Caspase-8 and c-FLIP sub(L) Associate in Lipid Rafts with NF- Kappa B Adaptors during T Cell Activation

The Journal of biological chemistry, 2007-07, Vol.282 (27), p.19365-19374

2007

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Caspase-8 and c-FLIPL Associate in Lipid Rafts with NF-ÎºB Adaptors during T Cell Activation

The Journal of biological chemistry, 2007-07, Vol.282 (27), p.19365

2007

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Effector CD4+ T Cells Generate Intermediate Caspase Activity and Cleavage of Caspase-8 Substrates1

The Journal of immunology (1950), 2005-04, Vol.174 (7), p.3999-4009

2005

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Effector CD4 super(+) T Cells Generate Intermediate Caspase Activity and Cleavage of Caspase-8 Substrates

The Journal of immunology (1950), 2005-04, Vol.174 (7), p.3999-4009

2005

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Reduced Immune Response to Borrelia burgdorferi in the Absence of γδ T Cells

c-FLIPS reduces activation of caspase and NF-κB pathways and decreases T cell survival

Proteolytic Regulation of Nuclear Factor of Activated T (NFAT) c2 Cells and NFAT Activity by Caspase-3

The c-FLIPL Cleavage Product p43FLIP Promotes Activation of Extracellular Signal-regulated Kinase (ERK), Nuclear Factor κB (NF-κB), and Caspase-8 and T Cell Survival

Cellular FLIP Long Form-Transgenic Mice Manifest a Th2 Cytokine Bias and Enhanced Allergic Airway Inflammation

Cellular FLIP (long form) regulates CD8+ T cell activation through caspase-8-dependent NF-kappa B activation

Fas Ligand Deficiency Impairs Host Inflammatory Response against Infection with the Spirochete Borrelia burgdorferi

Activation of gamma delta T cells by Borrelia burgdorferi is indirect via a TLR- and caspase-dependent pathway

Cellular FLIP Long Form Augments Caspase Activity and Death of T Cells through Heterodimerization with and Activation of Caspase-8

Effector CD4+ T Cells Generate Intermediate Caspase Activity and Cleavage of Caspase-8 Substrates

Liver Damage by Infiltrating CD8+ T Cells Is Fas Dependent

Activation of γδ T Cells by Borrelia burgdorferi Is Indirect via a TLR- and Caspase-Dependent Pathway

Caspase-8 and c-FLIPL Associate in Lipid Rafts with NF-κB Adaptors during T Cell Activation

Spatial differences in active caspase-8 defines its role in T-cell activation versus cell death

Anti-CD40L Accelerates Renal Disease and Adenopathy in MRL-lpr Mice in Parallel with Decreased Thymocyte Apoptosis

A Model for the Origin of TCR-αβ+ CD4−CD8− B220+ Cells Based on High Affinity TCR Signals

Activation of γδ T Cells by Borrelia burgdorferi is Indirect via a TLR- and Caspase-Dependent Pathway1

c‐FLIP S reduces activation of caspase and NF‐κB pathways and decreases T cell survival

c-FLIP(S) reduces activation of caspase and NF-kappaB pathways and decreases T cell survival

Cellular FLIP (Long Form) Regulates CD8 super(+) T Cell Activation through Caspase-8-Dependent NF- Kappa B Activation

Caspase-8 and c-FLIPL associate in lipid rafts with NF-kappaB adaptors during T cell activation

Caspase-8 and c-FLIP sub(L) Associate in Lipid Rafts with NF- Kappa B Adaptors during T Cell Activation

Caspase-8 and c-FLIPL Associate in Lipid Rafts with NF-ÎºB Adaptors during T Cell Activation

Effector CD4+ T Cells Generate Intermediate Caspase Activity and Cleavage of Caspase-8 Substrates1

Effector CD4 super(+) T Cells Generate Intermediate Caspase Activity and Cleavage of Caspase-8 Substrates

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