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The Role of the Na+Channel in the Accumulation of Intracellular Na+During Myocardial Ischemia: Consequences for Post-ischemic Recovery

Journal of molecular and cellular cardiology, 1997-01, Vol.29 (1), p.85-96

1997

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Both Na+-K+ATPase and Na+-H+Exchanger are Immediately Active upon Post-ischemic Reperfusion in Isolated Rat Hearts

Journal of molecular and cellular cardiology, 1998-02, Vol.30 (2), p.337-348

1998

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Brain death-related energetic failure of the donor heart becomes apparent only during storage and reperfusion: an ex vivo phosphorus-31 magnetic resonance spectroscopy study on the feline heart

The Journal of heart and lung transplantation, 2001-09, Vol.20 (9), p.996-1004

2001

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‘Myocardial hibernation’—questions and controversies

Cardiovascular research, 1997-12, Vol.36 (3), p.301-309

1997

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Bio-energetic response of the heart to dopamine following brain death–related reduced myocardial workload: A phosphorus-31 magnetic resonance spectroscopy study in the cat

The Journal of heart and lung transplantation, 1999-12, Vol.18 (12), p.1189-1197

1999

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Manipulation of Intracellular Sodium by Extracellular Divalent Cations: a23Na and31P NMR Study on Intact Rat Hearts

Journal of molecular and cellular cardiology, 1998-01, Vol.30 (1), p.119-126

1998

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Protective effect of pretreatment with the calcium antagonist anipamil on the ischemic-reperfused rat myocardium: A phosphorus-31 nuclear magnetic resonance study

Journal of the American College of Cardiology, 1988-05, Vol.11 (5), p.1087-1093

1988

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Postischemic Na+-K+-ATPase reactivation is delayed in the absence of glycolytic ATP in isolated rat hearts

American journal of physiology. Heart and circulatory physiology, 2001-05, Vol.280 (5), p.H2189-H2195

2001

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Acute Increase of Myocardial Workload, Hemodynamic Instability, and Myocardial Histological Changes Induced by Brain Death in the Cat

The Journal of surgical research, 1997-02, Vol.68 (1), p.7-15

1997

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Depletion of Preischemic Glycogen Reduces the Increase of Low Molecular Weight Iron during Ischemia

Annals of the New York Academy of Sciences, 1996-09, Vol.793 (1), p.74-84

1996

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Metabolic and Functional Consequences of Successive No-flow and Sustained Low-flow Ischaemia; a31P MRS Study in Rat Hearts

Journal of molecular and cellular cardiology, 1996-12, Vol.28 (12), p.2373-2381

1996

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Protective effect of pretreatment with tetrandrine in ischemic-reperfused rat heart

Journal of molecular and cellular cardiology, 1991-07, Vol.23, p.S107-S107

1991

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Metabolic and Functional Consequences of Successive No-flow and Sustained Low-flow Ischaemia; a 31P MRS Study in Rat Hearts

Journal of molecular and cellular cardiology, 1996-12, Vol.28 (12), p.2373-2381

1996

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A 31-P NMR study on postischemic myocardial ATP recovery during transient low-calcium reperfusion

Journal of the American College of Cardiology, 1991-02, Vol.17 (2), p.A77-A77

1991

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Glycolytic ATP production is not essential for Na+-K+ ATPase function and contractile recovery during postischemic reperfusion in isolated rat hearts

Magma (New York, N.Y.), 1998-09, Vol.6 (2-3), p.177-178

van Emous, J

1998

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Biochemical Properties of Membranes Isolated from Calcium-Depleted Rabbit Hearts

Circulation research, 1984-03, Vol.54 (3), p.217-226

1984

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Post-ischemic Contractile Dysfunction does not Correlate with an Elevated Intracellular Free [Mg2+]: A 31P-NMR Study on Isolated Rat and Rabbit Hearts

Journal of molecular and cellular cardiology, 1993-09, Vol.25 (9), p.1015-1024

1993

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The effect of Lidocaine on concentration and localization of Ca 2+ in Ca 2+-depleted and Ca 2+-repleted hearts

Journal of molecular and cellular cardiology, 1991, Vol.23, p.S161-S161

1991

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Myocardial intracellular Na + in the absence of extracellular Ca 2+ or both Ca 2+ and Mg 2

Journal of molecular and cellular cardiology, 1991, Vol.23, p.S156-S156

1991

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31P NMR study of myocardial energy metabolism response to heart rate steps

Journal of molecular and cellular cardiology, 1991-07, Vol.23, p.S106-S106

1991

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Low Ca2+ Reperfusion and Enhanced Susceptibility of the Postischemic Heart to the Calcium Paradox

Circulation research, 1989-06, Vol.64 (6), p.1158-1164

1989

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Reply to the Preceding Letter

Circulation research, 1985-02, Vol.56 (2), p.285-286

1985

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Different effects of thiopental in severe hypoxia, total ischemia, and low-flow ischemia in rat heart muscle

Anesthesiology (Philadelphia), 1985-08, Vol.63 (2), p.172-178

1985

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The Role of the Na+Channel in the Accumulation of Intracellular Na+During Myocardial Ischemia: Consequences for Post-ischemic Recovery

Both Na+-K+ATPase and Na+-H+Exchanger are Immediately Active upon Post-ischemic Reperfusion in Isolated Rat Hearts

Brain death-related energetic failure of the donor heart becomes apparent only during storage and reperfusion: an ex vivo phosphorus-31 magnetic resonance spectroscopy study on the feline heart

‘Myocardial hibernation’—questions and controversies

Bio-energetic response of the heart to dopamine following brain death–related reduced myocardial workload: A phosphorus-31 magnetic resonance spectroscopy study in the cat

Manipulation of Intracellular Sodium by Extracellular Divalent Cations: a23Na and31P NMR Study on Intact Rat Hearts

Protective effect of pretreatment with the calcium antagonist anipamil on the ischemic-reperfused rat myocardium: A phosphorus-31 nuclear magnetic resonance study

Postischemic Na+-K+-ATPase reactivation is delayed in the absence of glycolytic ATP in isolated rat hearts

Acute Increase of Myocardial Workload, Hemodynamic Instability, and Myocardial Histological Changes Induced by Brain Death in the Cat

Depletion of Preischemic Glycogen Reduces the Increase of Low Molecular Weight Iron during Ischemia

Metabolic and Functional Consequences of Successive No-flow and Sustained Low-flow Ischaemia; a31P MRS Study in Rat Hearts

Protective effect of pretreatment with tetrandrine in ischemic-reperfused rat heart

Metabolic and Functional Consequences of Successive No-flow and Sustained Low-flow Ischaemia; a 31P MRS Study in Rat Hearts

A 31-P NMR study on postischemic myocardial ATP recovery during transient low-calcium reperfusion

Glycolytic ATP production is not essential for Na+-K+ ATPase function and contractile recovery during postischemic reperfusion in isolated rat hearts

Biochemical Properties of Membranes Isolated from Calcium-Depleted Rabbit Hearts

Post-ischemic Contractile Dysfunction does not Correlate with an Elevated Intracellular Free [Mg2+]: A 31P-NMR Study on Isolated Rat and Rabbit Hearts

The effect of Lidocaine on concentration and localization of Ca 2+ in Ca 2+-depleted and Ca 2+-repleted hearts

Myocardial intracellular Na + in the absence of extracellular Ca 2+ or both Ca 2+ and Mg 2

31P NMR study of myocardial energy metabolism response to heart rate steps

Low Ca2+ Reperfusion and Enhanced Susceptibility of the Postischemic Heart to the Calcium Paradox

Reply to the Preceding Letter

Different effects of thiopental in severe hypoxia, total ischemia, and low-flow ischemia in rat heart muscle

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