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Single atom substitution in mouse protein kinase G eliminates oxidant sensing to cause hypertension

Nature medicine, 2012-02, Vol.18 (2), p.286-290

2012

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Biochemical methods for monitoring protein thiol redox states in biological systems

Redox biology, 2014-01, Vol.2 (C), p.803-813

2014

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Protection from hypertension in mice by the Mediterranean diet is mediated by nitro fatty acid inhibition of soluble epoxide hydrolase

Proceedings of the National Academy of Sciences - PNAS, 2014-06, Vol.111 (22), p.8167-8172

2014

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Protein kinase G oxidation is a major cause of injury during sepsis

Proceedings of the National Academy of Sciences - PNAS, 2013-06, Vol.110 (24), p.9909-9913

2013

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Disulfide-activated protein kinase G Iα regulates cardiac diastolic relaxation and fine-tunes the Frank-Starling response

Nature communications, 2016-10, Vol.7 (1), p.13187-11, Article 13187

2016

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Complex interrelationships between nitro-alkene-dependent inhibition of soluble epoxide hydrolase, inflammation and tumor growth

Redox biology, 2020-01, Vol.29, p.101405-101405, Article 101405

2020

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Experimental Hyperleptinemia in Neonatal Rats Leads to Selective Leptin Responsiveness, Hypertension, and Altered Myocardial Function

Hypertension (Dallas, Tex. 1979), 2013-09, Vol.62 (3), p.627-633

2013

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Examining a role for PKG Iα oxidation in the pathogenesis of cardiovascular dysfunction during diet-induced obesity

Free radical biology & medicine, 2017-09, Vol.110, p.390-398

2017

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Increased cardiovascular reactivity to acute stress and salt-loading in adult male offspring of fat fed non-obese rats

PloS one, 2011-10, Vol.6 (10), p.e25250-e25250

2011

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Protein Kinase G Iα Oxidation Paradoxically Underlies Blood Pressure Lowering by the Reductant Hydrogen Sulfide

Hypertension (Dallas, Tex. 1979), 2014-12, Vol.64 (6), p.1344-1351

2014

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Redox Regulation, Oxidative Stress, and Inflammation in Group 3 Pulmonary Hypertension

Advances in experimental medicine and biology, 2021-01, Vol.1303, p.209-241

2021

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cGMP-Dependent Activation of Protein Kinase G Precludes Disulfide Activation: Implications for Blood Pressure Control

Hypertension (Dallas, Tex. 1979), 2012-11, Vol.60 (5), p.1301-1308

2012

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A novel inhibitor of soluble Epoxide Hydrolase that adducts C521 is cardioprotective

Free radical biology & medicine, 2022-11, Vol.192, p.56-56

2022

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Cyclin D-CDK4 Disulfide Bond Attenuates Pulmonary Vascular Cell Proliferation

Circulation research, 2023-12, Vol.133 (12), p.966

2023

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Nitroglycerin Fails to Lower Blood Pressure in Redox-Dead Cys42Ser PKG1α Knock-In Mouse

Circulation (New York, N.Y.), 2012-07, Vol.126 (3), p.287-295

2012

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Oxidation of PKGIα mediates an endogenous adaptation to pulmonary hypertension

Proceedings of the National Academy of Sciences - PNAS, 2019-06, Vol.116 (26), p.13016-13025

2019

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Redox-Dead Protein Kinase a RIα knock-in Mouse Is Not Hypertensive But Has Increased Vascular Reactivity to Oxidants

Free radical biology & medicine, 2013-11, Vol.65, p.S82

2013

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PSS171 - Redox-Dead Protein Kinase a RIα knock-in Mouse Is Not Hypertensive But Has Increased Vascular Reactivity to Oxidants

Free radical biology & medicine, 2013-11, Vol.65, p.S82-S82

2013

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Redox-dependent dimerization of p38α mitogen-activated protein kinase with mitogen-activated protein kinase kinase 3

The Journal of biological chemistry, 2017-09, Vol.292 (39), p.16161-16173

2017

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Phospholemman Phosphorylation Regulates Vascular Tone, Blood Pressure, and Hypertension in Mice and Humans

Circulation (New York, N.Y.), 2021-03, Vol.143 (11), p.1123-1138

2021

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Protein Kinase a Regulatory RI Oxidation Mediates Angiogenesis

Free radical biology & medicine, 2012-11, Vol.53, p.S156-S156

2012

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Deficient angiogenesis in redox-dead Cys17Ser PKARIα knock-in mice

Nature communications, 2015-08, Vol.6 (1), p.7920, Article 7920

2015

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Cys42Ser Substitution in Murine Protein Kinase G1alpha Eliminates Oxidant Sensing To Cause Hypertension

Free radical biology & medicine, 2011-11, Vol.51, p.S49-S49

2011

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Quantification of microcirculatory blood flow: a sensitive and clinically relevant prognostic marker in murine models of sepsis

Journal of applied physiology (1985), 2015-02, Vol.118 (3), p.344-354

2015

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cGMP Impedes Disulfide Activation of Protein Kinase G: Implications in Blood Pressure Regulation

Free radical biology & medicine, 2012-11, Vol.53, p.S156-S156

2012

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Single atom substitution in mouse protein kinase G eliminates oxidant sensing to cause hypertension

Biochemical methods for monitoring protein thiol redox states in biological systems

Protection from hypertension in mice by the Mediterranean diet is mediated by nitro fatty acid inhibition of soluble epoxide hydrolase

Protein kinase G oxidation is a major cause of injury during sepsis

Disulfide-activated protein kinase G Iα regulates cardiac diastolic relaxation and fine-tunes the Frank-Starling response

Complex interrelationships between nitro-alkene-dependent inhibition of soluble epoxide hydrolase, inflammation and tumor growth

Experimental Hyperleptinemia in Neonatal Rats Leads to Selective Leptin Responsiveness, Hypertension, and Altered Myocardial Function

Examining a role for PKG Iα oxidation in the pathogenesis of cardiovascular dysfunction during diet-induced obesity

Increased cardiovascular reactivity to acute stress and salt-loading in adult male offspring of fat fed non-obese rats

Protein Kinase G Iα Oxidation Paradoxically Underlies Blood Pressure Lowering by the Reductant Hydrogen Sulfide

Redox Regulation, Oxidative Stress, and Inflammation in Group 3 Pulmonary Hypertension

cGMP-Dependent Activation of Protein Kinase G Precludes Disulfide Activation: Implications for Blood Pressure Control

A novel inhibitor of soluble Epoxide Hydrolase that adducts C521 is cardioprotective

Cyclin D-CDK4 Disulfide Bond Attenuates Pulmonary Vascular Cell Proliferation

Nitroglycerin Fails to Lower Blood Pressure in Redox-Dead Cys42Ser PKG1α Knock-In Mouse

Oxidation of PKGIα mediates an endogenous adaptation to pulmonary hypertension

Redox-Dead Protein Kinase a RIα knock-in Mouse Is Not Hypertensive But Has Increased Vascular Reactivity to Oxidants

PSS171 - Redox-Dead Protein Kinase a RIα knock-in Mouse Is Not Hypertensive But Has Increased Vascular Reactivity to Oxidants

Redox-dependent dimerization of p38α mitogen-activated protein kinase with mitogen-activated protein kinase kinase 3

Phospholemman Phosphorylation Regulates Vascular Tone, Blood Pressure, and Hypertension in Mice and Humans

Protein Kinase a Regulatory RI Oxidation Mediates Angiogenesis

Deficient angiogenesis in redox-dead Cys17Ser PKARIα knock-in mice

Cys42Ser Substitution in Murine Protein Kinase G1alpha Eliminates Oxidant Sensing To Cause Hypertension

Quantification of microcirculatory blood flow: a sensitive and clinically relevant prognostic marker in murine models of sepsis

cGMP Impedes Disulfide Activation of Protein Kinase G: Implications in Blood Pressure Regulation

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