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Tumor suppressor role of the complement inhibitor CSMD1 and its role in TNF-induced neuroinflammation in gliomas

Journal of experimental & clinical cancer research, 2024-04, Vol.43 (1), p.98-98, Article 98

2024

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Hypoxia-Induced Reactivity of Tumor-Associated Astrocytes Affects Glioma Cell Properties

Cells (Basel, Switzerland), 2021-03, Vol.10 (3), p.613

2021

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840 Presence and distribution of immunosuppressive peptide P3028 in relation to immune phenotype of tonsillar cancer

Journal for immunotherapy of cancer, 2020-11, Vol.8 (Suppl 3), p.A501-A502

2020

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TMIC-73. UNRAVELING THE ALTERED EXTRACELLULAR MATRIX LANDSCAPE IN GLIOBLASTOMA AFTER RADIATION TREATMENT

Neuro-oncology (Charlottesville, Va.), 2023-11, Vol.25 (Supplement_5), p.v294-v295

2023

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TMIC-54. RADIOTHERAPY-INDUCED REMODELING OF THE GLIOBLASTOMA MICROENVIRONMENT

Neuro-oncology (Charlottesville, Va.), 2023-11, Vol.25 (Supplement_5), p.v290-v290

2023

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TAMI-67. INVESTIGATION OF THE COMPLEMENT SYSTEM IN THE IRRADIATED BRAIN TUMOR MICROENVIRONMENT

Neuro-oncology (Charlottesville, Va.), 2021-11, Vol.23 (Supplement_6), p.vi212-vi212

2021

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Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation

Cell metabolism, 2019-01, Vol.29 (1), p.202-210.e6

2019

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TMIC-12. COMPLEMENT SIGNALING IN HYPOXIC NICHES OF THE GLIOMA MICROENVIRONMENT

Neuro-oncology (Charlottesville, Va.), 2022-11, Vol.24 (Supplement_7), p.vii273-vii273

2022

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TMIC-01. PERICYTES ORCHESTRATE A TUMOR-SUPPRESSIVE MICROENVIRONMENT BY IMPINGING ON THE CROSSTALK BETWEEN MACROPHAGES AND TUMOR-INITIATING CELLS IN GLIOBLASTOMA MULTIFORME

Neuro-oncology (Charlottesville, Va.), 2023-11, Vol.25 (Supplement_5), p.v277-v278

2023

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A cryptic non–GPI‐anchored cytosolic isoform of CD59 controls insulin exocytosis in pancreatic β‐cells by interaction with SNARE proteins

The FASEB journal, 2019-11, Vol.33 (11), p.12425-12434

2019

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TMIC-30. HYPOXIA-INDUCED COMPLEMENT COMPONENT 3 PROMOTES AGGRESSIVE TUMOR GROWTH IN THE GLIOBLASTOMA MICROENVIRONMENT

Neuro-oncology (Charlottesville, Va.), 2023-11, Vol.25 (Supplement_5), p.v284-v285

2023

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TMIC-62. COMPLEMENT FACTOR H IS A NOVEL LIGAND FOR THE INDUCIBLE T CELL CO-STIMULATOR AND PROMOTES SURVIVAL OF REGULATORY T CELLS IN THE GLIOMA MICROENVIRONMENT

Neuro-oncology (Charlottesville, Va.), 2023-11, Vol.25 (Supplement_5), p.v292-v292

2023

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27 Elucidating the molecular mechanisms mediating the tumor suppressive actions of complement inhibitor CSMD1 in gliomas

Immunobiology (1979), 2023-09, Vol.228 (5), p.152484, Article 152484

2023

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Complement factor H is an ICOS ligand modulating Tregs in the glioma microenvironment

Cancer immunology research, 2024-10

2024

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Intracellular non-GPI anchored isoform of CD59 is responsible for facilitating of insulin secretion from pancreatic β-cells

Molecular immunology, 2018-10, Vol.102, p.155-156

2018

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Complement component C3 regulates cytoprotective autophagy in pancreatic islets via ATG16L1 interaction

Molecular immunology, 2018-10, Vol.102, p.175-175

2018

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A cryptic non-GPI anchored form of CD59 facilitates insulin secretion from pancreatic beta cells

16th European Meeting of Complement in Human Disease,Copenhagen, Denmark,2017-09-08 - 2017-09-12, 2017-09, Vol.89, p.135-135

2017

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Hypoxia-induced complement component 3 promotes aggressive tumor growth in the glioblastoma microenvironment

JCI insight, 2024-08, Vol.9 (19)

2024

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A cryptic non-GPI anchored form of CD59 facilitates insulin secretion from pancreatic beta cells

Immunobiology (1979), 2016-10, Vol.221 (10), p.1165-1165

2016

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Recombinant lactobacilli expressing linoleic acid isomerase can modulate the fatty acid composition of host adipose tissue in mice

Microbiology (Society for General Microbiology), 2011-02, Vol.157 (Pt 2), p.609-615

2011

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Tumor suppressor role of the complement inhibitor CSMD1 and its role in TNF-induced neuroinflammation in gliomas

Hypoxia-Induced Reactivity of Tumor-Associated Astrocytes Affects Glioma Cell Properties

840 Presence and distribution of immunosuppressive peptide P3028 in relation to immune phenotype of tonsillar cancer

TMIC-73. UNRAVELING THE ALTERED EXTRACELLULAR MATRIX LANDSCAPE IN GLIOBLASTOMA AFTER RADIATION TREATMENT

TMIC-54. RADIOTHERAPY-INDUCED REMODELING OF THE GLIOBLASTOMA MICROENVIRONMENT

TAMI-67. INVESTIGATION OF THE COMPLEMENT SYSTEM IN THE IRRADIATED BRAIN TUMOR MICROENVIRONMENT

Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation

TMIC-12. COMPLEMENT SIGNALING IN HYPOXIC NICHES OF THE GLIOMA MICROENVIRONMENT

TMIC-01. PERICYTES ORCHESTRATE A TUMOR-SUPPRESSIVE MICROENVIRONMENT BY IMPINGING ON THE CROSSTALK BETWEEN MACROPHAGES AND TUMOR-INITIATING CELLS IN GLIOBLASTOMA MULTIFORME

A cryptic non–GPI‐anchored cytosolic isoform of CD59 controls insulin exocytosis in pancreatic β‐cells by interaction with SNARE proteins

TMIC-30. HYPOXIA-INDUCED COMPLEMENT COMPONENT 3 PROMOTES AGGRESSIVE TUMOR GROWTH IN THE GLIOBLASTOMA MICROENVIRONMENT

TMIC-62. COMPLEMENT FACTOR H IS A NOVEL LIGAND FOR THE INDUCIBLE T CELL CO-STIMULATOR AND PROMOTES SURVIVAL OF REGULATORY T CELLS IN THE GLIOMA MICROENVIRONMENT

27 Elucidating the molecular mechanisms mediating the tumor suppressive actions of complement inhibitor CSMD1 in gliomas

Complement factor H is an ICOS ligand modulating Tregs in the glioma microenvironment

Intracellular non-GPI anchored isoform of CD59 is responsible for facilitating of insulin secretion from pancreatic β-cells

Complement component C3 regulates cytoprotective autophagy in pancreatic islets via ATG16L1 interaction

A cryptic non-GPI anchored form of CD59 facilitates insulin secretion from pancreatic beta cells

Hypoxia-induced complement component 3 promotes aggressive tumor growth in the glioblastoma microenvironment

A cryptic non-GPI anchored form of CD59 facilitates insulin secretion from pancreatic beta cells

Recombinant lactobacilli expressing linoleic acid isomerase can modulate the fatty acid composition of host adipose tissue in mice

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