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Infigratinib in children with achondroplasia: the PROPEL and PROPEL 2 studies

Therapeutic advances in musculoskeletal disease, 2022-03, Vol.14, p.1759720X221084848-1759720X221084848

2022

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Phase III trial of 8% vaginal progesterone gel for luteal phase support in Japanese women undergoing in vitro fertilization and fresh embryo transfer cycles

Reproductive medicine and biology, 2017-01, Vol.16 (1), p.52-57

2017

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Deletion of Hexose-6-phosphate Dehydrogenase Activates the Unfolded Protein Response Pathway and Induces Skeletal Myopathy

The Journal of biological chemistry, 2008-03, Vol.283 (13), p.8453-8461

2008

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Phase III trial comparing the efficacy and safety of recombinant‐ or urine‐derived human chorionic gonadotropin for ovulation triggering in Japanese women diagnosed with anovulation or oligo‐ovulation and undergoing ovulation induction with follitropin‐alfa

Reproductive medicine and biology, 2017-01, Vol.16 (1), p.45-51

2017

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SUN-087 FGFR-Selective Tyrosine Kinase Inhibitors, Such as Infigratinib, Show Potency and Selectivity for FGFR3 at Pharmacologically Relevant Doses for the Potential Treatment of Achondroplasia

Journal of the Endocrine Society, 2020-05, Vol.4 (Supplement_1)

2020

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Infigratinib in Children With Achondroplasia (ACH): Design of PROPEL2 - A Phase 2, Open-Label, Dose-Escalation and Dose-Expansion Study

Journal of the Endocrine Society, 2021-05, Vol.5 (Supplement_1), p.A667-A668

2021

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Efficacy, safety and population pharmacokinetics of sapropterin in PKU patients <4 years: results from the SPARK open-label, multicentre, randomized phase IIIb trial

Orphanet journal of rare diseases, 2017-03, Vol.12 (1), p.47, Article 47

2017

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SUN-093 Prospective Clinical Assessment Study in Children with Achondroplasia: The PROPEL Trial

Journal of the Endocrine Society, 2020-05, Vol.4 (Supplement_1)

2020

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THU165 PROPEL, PROPEL 2 And PROPEL OLE Studies Of Infigratinib In Children With Achondroplasia: Design And Status Of 3 Ongoing Trials

Journal of the Endocrine Society, 2023-10, Vol.7 (Supplement_1)

2023

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THU181 Evaluation Of Bone Mineral Density In A Cohort Of Children With ACH Participating In The PROPEL 2 Study Of Infigratinib

Journal of the Endocrine Society, 2023-10, Vol.7 (Supplement_1)

2023

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PSAT105 Evaluation of Body Mass Index and Metabolic Parameters in Children with Achondroplasia Participating in the PROPEL Study

Journal of the Endocrine Society, 2022-11, Vol.6 (Supplement_1), p.A632-A633

2022

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RF26 | PMON326 Medical History of Children Enrolled in PROPEL: A Prospective Clinical Assessment Study in Children with Achondroplasia

Journal of the Endocrine Society, 2022-11, Vol.6 (Supplement_1), p.A644-A644

2022

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PSAT106 Infigratinib in Children with Achondroplasia: Design of the PROPEL, PROPEL2 and PROPEL OLE Studies

Journal of the Endocrine Society, 2022-11, Vol.6 (Supplement_1), p.A633-A633

2022

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Hexose 6-phosphate dehydrogenase (H6PD) and corticosteroid metabolism

Molecular and cellular endocrinology, 2007-02, Vol.265-266, p.89-92

2007

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Abnormalities of Glucose Homeostasis and the Hypothalamic-Pituitary-Adrenal Axis in Mice Lacking Hexose-6-Phosphate Dehydrogenase

Endocrinology (Philadelphia), 2007-10, Vol.148 (10), p.5072-5080

2007

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Contribution of hexose-6-phosphate dehydrogenase to NADPH content and redox environment in the endoplasmic reticulum

Redox report : communications in free radical research, 2010-04, Vol.15 (2), p.64-70

2010

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P53 and Cellular Glucose Uptake

Endocrine research, 2013-01, Vol.38 (1), p.32-39

2013

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Physiological roles of 11 beta-hydroxysteroid dehydrogenase type 1 and hexose-6-phosphate dehydrogenase

Current opinion in pediatrics, 2008-08, Vol.20 (4), p.453-457

2008

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Deletion of Hexose-6-phosphate Dehydrogenase Activates the Unfolded Protein Response Pathway and Induces Skeletal MyopathyS

The Journal of biological chemistry, 2008-03, Vol.283 (13), p.8453-8461

2008

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Steroidogenesis in the human skin: 21-hydroxylation in cultured keratinocytes

The Journal of steroid biochemistry and molecular biology, 2001-07, Vol.78 (1), p.77-81

2001

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The codon 213 of the 11β-hydroxysteroid dehydrogenase type 2 gene is a hot spot for mutations in apparent mineralocorticoid excess

The journal of clinical endocrinology and metabolism, 1998, Vol.83 (12), p.4391-4393

1998

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The Codon 213 of the 11β-Hydroxysteroid Dehydrogenase Type 2 Gene Is a Hot Spot for Mutations in Apparent Mineralocorticoid Excess 1

The journal of clinical endocrinology and metabolism, 1998-12, Vol.83 (12), p.4391-4393

1998

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The Codon 213 of the 11β-Hydroxysteroid Dehydrogenase Type 2 Gene Is a Hot Spot for Mutations in Apparent Mineralocorticoid Excess1

The journal of clinical endocrinology and metabolism, 1998-12, Vol.83 (12), p.4391-4393

1998

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Intermittent hyperaldosteronism in a child due to an adrenal adenoma

European journal of pediatrics, 2001-02, Vol.160 (2), p.114-116

2001

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Infigratinib in children with achondroplasia: the PROPEL and PROPEL 2 studies

Phase III trial of 8% vaginal progesterone gel for luteal phase support in Japanese women undergoing in vitro fertilization and fresh embryo transfer cycles

Deletion of Hexose-6-phosphate Dehydrogenase Activates the Unfolded Protein Response Pathway and Induces Skeletal Myopathy

Phase III trial comparing the efficacy and safety of recombinant‐ or urine‐derived human chorionic gonadotropin for ovulation triggering in Japanese women diagnosed with anovulation or oligo‐ovulation and undergoing ovulation induction with follitropin‐alfa

SUN-087 FGFR-Selective Tyrosine Kinase Inhibitors, Such as Infigratinib, Show Potency and Selectivity for FGFR3 at Pharmacologically Relevant Doses for the Potential Treatment of Achondroplasia

Infigratinib in Children With Achondroplasia (ACH): Design of PROPEL2 - A Phase 2, Open-Label, Dose-Escalation and Dose-Expansion Study

Efficacy, safety and population pharmacokinetics of sapropterin in PKU patients <4 years: results from the SPARK open-label, multicentre, randomized phase IIIb trial

SUN-093 Prospective Clinical Assessment Study in Children with Achondroplasia: The PROPEL Trial

THU165 PROPEL, PROPEL 2 And PROPEL OLE Studies Of Infigratinib In Children With Achondroplasia: Design And Status Of 3 Ongoing Trials

OR27-03 Oral Infigratinib Treatment Is Well Tolerated And Significantly Increases Height Velocity In Children With Achondroplasia: Month 6 Results From The PROPEL 2 Dose-finding Study

THU181 Evaluation Of Bone Mineral Density In A Cohort Of Children With ACH Participating In The PROPEL 2 Study Of Infigratinib

PSAT105 Evaluation of Body Mass Index and Metabolic Parameters in Children with Achondroplasia Participating in the PROPEL Study

RF26 | PMON326 Medical History of Children Enrolled in PROPEL: A Prospective Clinical Assessment Study in Children with Achondroplasia

PSAT106 Infigratinib in Children with Achondroplasia: Design of the PROPEL, PROPEL2 and PROPEL OLE Studies

Hexose 6-phosphate dehydrogenase (H6PD) and corticosteroid metabolism

Abnormalities of Glucose Homeostasis and the Hypothalamic-Pituitary-Adrenal Axis in Mice Lacking Hexose-6-Phosphate Dehydrogenase

Contribution of hexose-6-phosphate dehydrogenase to NADPH content and redox environment in the endoplasmic reticulum

P53 and Cellular Glucose Uptake

Physiological roles of 11 beta-hydroxysteroid dehydrogenase type 1 and hexose-6-phosphate dehydrogenase

Deletion of Hexose-6-phosphate Dehydrogenase Activates the Unfolded Protein Response Pathway and Induces Skeletal MyopathyS

Steroidogenesis in the human skin: 21-hydroxylation in cultured keratinocytes

The codon 213 of the 11β-hydroxysteroid dehydrogenase type 2 gene is a hot spot for mutations in apparent mineralocorticoid excess

The Codon 213 of the 11β-Hydroxysteroid Dehydrogenase Type 2 Gene Is a Hot Spot for Mutations in Apparent Mineralocorticoid Excess 1

The Codon 213 of the 11β-Hydroxysteroid Dehydrogenase Type 2 Gene Is a Hot Spot for Mutations in Apparent Mineralocorticoid Excess1

Intermittent hyperaldosteronism in a child due to an adrenal adenoma

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