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Calcium Signaling in Cardiomyocyte Function

Cold Spring Harbor perspectives in biology, 2020-03, Vol.12 (3), p.a035428

2020

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Autophagosome formation from membrane compartments enriched in phosphatidylinositol 3-phosphate and dynamically connected to the endoplasmic reticulum

The Journal of cell biology, 2008-08, Vol.182 (4), p.685-701

2008

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Calcium signaling in cardiac myocytes

Cold Spring Harbor perspectives in biology, 2011-11, Vol.3 (11), p.a004242-a004242

2011

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Connexin hemichannels as candidate targets for cardioprotective and anti-arrhythmic treatments

The Journal of clinical investigation, 2023-03, Vol.133 (6), p.1-12

2023

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Global fibroblast activation throughout the left ventricle but localized fibrosis after myocardial infarction

Scientific reports, 2017-09, Vol.7 (1), p.10801-14, Article 10801

2017

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The role of the paracrine/autocrine mediator endothelin‐1 in regulation of cardiac contractility and growth

British journal of pharmacology, 2013-01, Vol.168 (2), p.296-317

2013

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BH4 domain of Bcl-2 inhibits ER calcium release and apoptosis by binding the regulatory and coupling domain of the IP3 receptor

Proceedings of the National Academy of Sciences - PNAS, 2009-08, Vol.106 (34), p.14397-14402

2009

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Epigenetics in the heart: the role of histone modifications in cardiac remodelling

Biochemical Society transactions, 2013-06, Vol.41 (3), p.789-796

2013

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Phosphorylation of inositol 1,4,5-trisphosphate receptors by protein kinase B/Akt inhibits Ca²⁺ release and apoptosis

Proceedings of the National Academy of Sciences - PNAS, 2008-02, Vol.105 (7), p.2427-2432

2008

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Targeting Bcl-2-IP3 Receptor Interaction to Reverse Bcl-2's Inhibition of Apoptotic Calcium Signals

Molecular cell, 2008-07, Vol.31 (2), p.255-265

2008

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Subcellular calcium dynamics in a whole-cell model of an atrial myocyte

Proceedings of the National Academy of Sciences - PNAS, 2012-02, Vol.109 (6), p.2150-2155

2012

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Emerging roles of inositol 1,4,5-trisphosphate signaling in cardiac myocytes

Journal of molecular and cellular cardiology, 2008-08, Vol.45 (2), p.128-147

2008

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Ventricular Arrhythmias in Ischemic Cardiomyopathy-New Avenues for Mechanism-Guided Treatment

Cells (Basel, Switzerland), 2021-10, Vol.10 (10), p.2629

2021

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Exposure to GSM RF fields does not affect calcium homeostasis in human endothelial cells, rat pheocromocytoma cells or rat hippocampal neurons

PloS one, 2010-07, Vol.5 (7), p.e11828-e11828

2010

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Assessing Cardiomyocyte Excitation-Contraction Coupling Site Detection From Live Cell Imaging Using a Structurally-Realistic Computational Model of Calcium Release

Frontiers in physiology, 2019-10, Vol.10, p.1263-1263

2019

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Ca2+ signalling checkpoints in cancer: remodelling Ca2+ for cancer cell proliferation and survival

Nature reviews. Cancer, 2008-05, Vol.8 (5), p.361-375

2008

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The cardiomyocyte firestarter—RyR clusters ignite their neighbours after augmentation of Ca2+ release by β‐stimulation

Acta Physiologica, 2022-04, Vol.234 (4), p.e13798-n/a

2022

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Inositol 1,4,5-trisphosphate receptors in cardiomyocyte physiology and disease

Philosophical transactions of the Royal Society of London. Series B. Biological sciences, 2022-11, Vol.377 (1864)

2022

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Killing in the name of: Reversing epigenetic silencing of ITPR3 to succumb cancer cells’ resistance to chemotherapeutics

Cell calcium (Edinburgh), 2022-03, Vol.102, p.102526-102526, Article 102526

2022

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The cardiomyocyte firestarter-RyR clusters ignite their neighbours after augmentation of Ca 2+ release by β-stimulation

Acta Physiologica, 2022-04, Vol.234 (4), p.e13798

2022

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Incomplete Assembly of the Dystrophin-Associated Protein Complex in 2D and 3D-Cultured Human Induced Pluripotent Stem Cell-Derived Cardiomyocytes

Frontiers in cell and developmental biology, 2021-11, Vol.9 (Article number 737840), p.737840-737840

2021

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Temporal expression and spatial distribution of the proteoglycan versican during cardiac fibrosis development

Matrix biology plus, 2023-12, Vol.19-20, p.100135-100135, Article 100135

2023

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Ca2+ release via InsP3Rs enhances RyR recruitment during Ca2+ transients by increasing dyadic [Ca2+] in cardiomyocytes

Journal of cell science, 2021-07, Vol.134 (14)

2021

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Myofibroblast Phenotype and Reversibility of Fibrosis in Patients With End-Stage Heart Failure

Journal of the American College of Cardiology, 2019-05, Vol.73 (18), p.2267-2282

2019

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Bcl-2 suppresses Ca2+ release through inositol 1,4,5-trisphosphate receptors and inhibits Ca2+ uptake by mitochondria without affecting ER calcium store content

Cell calcium (Edinburgh), 2008-09, Vol.44 (3), p.324-338

2008

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Calcium Signaling in Cardiomyocyte Function

Autophagosome formation from membrane compartments enriched in phosphatidylinositol 3-phosphate and dynamically connected to the endoplasmic reticulum

Calcium signaling in cardiac myocytes

Connexin hemichannels as candidate targets for cardioprotective and anti-arrhythmic treatments

Global fibroblast activation throughout the left ventricle but localized fibrosis after myocardial infarction

The role of the paracrine/autocrine mediator endothelin‐1 in regulation of cardiac contractility and growth

BH4 domain of Bcl-2 inhibits ER calcium release and apoptosis by binding the regulatory and coupling domain of the IP3 receptor

Epigenetics in the heart: the role of histone modifications in cardiac remodelling

Phosphorylation of inositol 1,4,5-trisphosphate receptors by protein kinase B/Akt inhibits Ca²⁺ release and apoptosis

Targeting Bcl-2-IP3 Receptor Interaction to Reverse Bcl-2's Inhibition of Apoptotic Calcium Signals

Subcellular calcium dynamics in a whole-cell model of an atrial myocyte

Emerging roles of inositol 1,4,5-trisphosphate signaling in cardiac myocytes

Ventricular Arrhythmias in Ischemic Cardiomyopathy-New Avenues for Mechanism-Guided Treatment

Exposure to GSM RF fields does not affect calcium homeostasis in human endothelial cells, rat pheocromocytoma cells or rat hippocampal neurons

Assessing Cardiomyocyte Excitation-Contraction Coupling Site Detection From Live Cell Imaging Using a Structurally-Realistic Computational Model of Calcium Release

Ca2+ signalling checkpoints in cancer: remodelling Ca2+ for cancer cell proliferation and survival

The cardiomyocyte firestarter—RyR clusters ignite their neighbours after augmentation of Ca2+ release by β‐stimulation

Inositol 1,4,5-trisphosphate receptors in cardiomyocyte physiology and disease

Killing in the name of: Reversing epigenetic silencing of ITPR3 to succumb cancer cells’ resistance to chemotherapeutics

The cardiomyocyte firestarter-RyR clusters ignite their neighbours after augmentation of Ca 2+ release by β-stimulation

Incomplete Assembly of the Dystrophin-Associated Protein Complex in 2D and 3D-Cultured Human Induced Pluripotent Stem Cell-Derived Cardiomyocytes

Temporal expression and spatial distribution of the proteoglycan versican during cardiac fibrosis development

Ca2+ release via InsP3Rs enhances RyR recruitment during Ca2+ transients by increasing dyadic [Ca2+] in cardiomyocytes

Myofibroblast Phenotype and Reversibility of Fibrosis in Patients With End-Stage Heart Failure

Bcl-2 suppresses Ca2+ release through inositol 1,4,5-trisphosphate receptors and inhibits Ca2+ uptake by mitochondria without affecting ER calcium store content

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