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G-Protein–Coupled Receptors in Heart Disease

Circulation research, 2018-08, Vol.123 (6), p.716-735

2018

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Lethal Cardiomyopathy in Mice Lacking Transferrin Receptor in the Heart

Cell reports (Cambridge), 2015-10, Vol.13 (3), p.533-545

2015

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Circulating Exosomes Induced by Cardiac Pressure Overload Contain Functional Angiotensin II Type 1 Receptors

Circulation (New York, N.Y.), 2015-06, Vol.131 (24), p.2120-2130

2015

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Gαi is required for carvedilol-induced β1 adrenergic receptor β-arrestin biased signaling

Nature communications, 2017-11, Vol.8 (1), p.1-14, Article 1706

2017

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TRPC1 Channels Are Critical for Hypertrophic Signaling in the Heart

Circulation research, 2009-11, Vol.105 (10), p.1023-1030

2009

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β-Blockers alprenolol and carvedilol stimulate β-arrestin-mediated EGFR transactivation

Proceedings of the National Academy of Sciences - PNAS, 2008-09, Vol.105 (38), p.14555-14560

2008

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How carvedilol does not activate β2-adrenoceptors

Nature communications, 2023-11, Vol.14 (1), p.7866-7866, Article 7866

2023

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Endogenous S-nitrosothiols protect against myocardial injury

Proceedings of the National Academy of Sciences - PNAS, 2009-04, Vol.106 (15), p.6297-6302

2009

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Muscle-Specific RING Finger 1 Is a Bona Fide Ubiquitin Ligase That Degrades Cardiac Troponin I

Proceedings of the National Academy of Sciences - PNAS, 2004-12, Vol.101 (52), p.18135-18140

2004

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Drosophila as a model for the identification of genes causing adult human heart disease

Proceedings of the National Academy of Sciences - PNAS, 2006-01, Vol.103 (5), p.1394-1399

2006

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β-arrestin 1 regulates β2-adrenergic receptor-mediated skeletal muscle hypertrophy and contractility

Skeletal muscle, 2018-12, Vol.8 (1), p.39-39, Article 39

2018

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β-Arrestin2-Mediated Inotropic Effects of the Angiotensin II Type 1A Receptor in Isolated Cardiac Myocytes

Proceedings of the National Academy of Sciences - PNAS, 2006-10, Vol.103 (44), p.16284-16289

2006

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G protein-coupled receptor signaling: transducers and effectors

American Journal of Physiology: Cell Physiology, 2022-09, Vol.323 (3), p.C731-C748

2022

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Overexpression of TNNI3K, a cardiac-specific MAPKKK, promotes cardiac dysfunction

Journal of molecular and cellular cardiology, 2013-01, Vol.54, p.101-111

2013

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Phosphorylation of Src by phosphoinositide 3-kinase regulates beta-adrenergic receptor-mediated EGFR transactivation

Cellular signalling, 2016-10, Vol.28 (10), p.1580-1592

2016

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p53 functions in endothelial cells to prevent radiation-induced myocardial injury in mice

Science signaling, 2012-07, Vol.5 (234), p.ra52

2012

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An Essential Role for Mitochondrial Aldehyde Dehydrogenase in Nitroglycerin Bioactivation

Proceedings of the National Academy of Sciences - PNAS, 2005-08, Vol.102 (34), p.12159-12164

2005

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Pathophysiology and pharmacology of G protein-coupled receptors in the heart

Cardiovascular research, 2023-05, Vol.119 (5), p.1117-1129

2023

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Tnni3k modifies disease progression in murine models of cardiomyopathy

PLoS genetics, 2009-09, Vol.5 (9), p.e1000647-e1000647

2009

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G Protein–Dependent and G Protein–Independent Signaling Pathways and Their Impact on Cardiac Function

Circulation research, 2011-07, Vol.109 (2), p.217-230

2011

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Enhanced Postischemic Functional Recovery in CYP2J2 Transgenic Hearts Involves Mitochondrial ATP-Sensitive K+ Channels and p42/p44 MAPK Pathway

Circulation research, 2004-09, Vol.95 (5), p.506-514

2004

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G protein-coupled receptors: from radioligand binding to cellular signaling

The Journal of clinical investigation, 2024-03, Vol.134 (5)

2024

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Biased Ligands for Better Cardiovascular Drugs: Dissecting G-Protein-Coupled Receptor Pharmacology

Circulation research, 2011-07, Vol.109 (2), p.205-216

2011

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Biased G Protein–Coupled Receptor Signaling: Changing the Paradigm of Drug Discovery

Circulation (New York, N.Y.), 2018-05, Vol.137 (22), p.2315-2317

2018

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Role of Soluble Epoxide Hydrolase in Postischemic Recovery of Heart Contractile Function

Circulation research, 2006-08, Vol.99 (4), p.442-450

2006

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G-Protein–Coupled Receptors in Heart Disease

Lethal Cardiomyopathy in Mice Lacking Transferrin Receptor in the Heart

Circulating Exosomes Induced by Cardiac Pressure Overload Contain Functional Angiotensin II Type 1 Receptors

Gαi is required for carvedilol-induced β1 adrenergic receptor β-arrestin biased signaling

TRPC1 Channels Are Critical for Hypertrophic Signaling in the Heart

β-Blockers alprenolol and carvedilol stimulate β-arrestin-mediated EGFR transactivation

How carvedilol does not activate β2-adrenoceptors

Endogenous S-nitrosothiols protect against myocardial injury

Muscle-Specific RING Finger 1 Is a Bona Fide Ubiquitin Ligase That Degrades Cardiac Troponin I

Drosophila as a model for the identification of genes causing adult human heart disease

β-arrestin 1 regulates β2-adrenergic receptor-mediated skeletal muscle hypertrophy and contractility

β-Arrestin2-Mediated Inotropic Effects of the Angiotensin II Type 1A Receptor in Isolated Cardiac Myocytes

G protein-coupled receptor signaling: transducers and effectors

Overexpression of TNNI3K, a cardiac-specific MAPKKK, promotes cardiac dysfunction

Phosphorylation of Src by phosphoinositide 3-kinase regulates beta-adrenergic receptor-mediated EGFR transactivation

p53 functions in endothelial cells to prevent radiation-induced myocardial injury in mice

An Essential Role for Mitochondrial Aldehyde Dehydrogenase in Nitroglycerin Bioactivation

Pathophysiology and pharmacology of G protein-coupled receptors in the heart

Tnni3k modifies disease progression in murine models of cardiomyopathy

G Protein–Dependent and G Protein–Independent Signaling Pathways and Their Impact on Cardiac Function

Enhanced Postischemic Functional Recovery in CYP2J2 Transgenic Hearts Involves Mitochondrial ATP-Sensitive K+ Channels and p42/p44 MAPK Pathway

G protein-coupled receptors: from radioligand binding to cellular signaling

Biased Ligands for Better Cardiovascular Drugs: Dissecting G-Protein-Coupled Receptor Pharmacology

Biased G Protein–Coupled Receptor Signaling: Changing the Paradigm of Drug Discovery

Role of Soluble Epoxide Hydrolase in Postischemic Recovery of Heart Contractile Function

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