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The Osteocyte: New Insights

Annual review of physiology, 2020-02, Vol.82 (1), p.485-506

2020

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The Wnt pathway: An important control mechanism in bone's response to mechanical loading

Bone (New York, N.Y.), 2021-12, Vol.153, p.116087-116087, Article 116087

2021

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An osteocalcin-deficient mouse strain without endocrine abnormalities

PLoS genetics, 2020-05, Vol.16 (5), p.e1008361-e1008361

2020

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Is Bone's Response to Mechanical Signals Dominated by Muscle Forces?

Medicine and science in sports and exercise, 2009-11, Vol.41 (11), p.2044-2049

ROBLING, Alexander G

2009

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Bone and skeletal muscle: Key players in mechanotransduction and potential overlapping mechanisms

Bone (New York, N.Y.), 2015-11, Vol.80, p.24-36

2015

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Biomechanical and molecular regulation of bone remodeling

Annual review of biomedical engineering, 2006-01, Vol.8 (1), p.455-498

2006

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The Skeleton as a Secretory Organ

Calcified tissue international, 2023-07, Vol.113 (1), p.1-3

2023

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Sost downregulation and local Wnt signaling are required for the osteogenic response to mechanical loading

Bone (New York, N.Y.), 2012-01, Vol.50 (1), p.209-217

2012

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Control of bone mass and remodeling by PTH receptor signaling in osteocytes

PloS one, 2008-08, Vol.3 (8), p.e2942-e2942

2008

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Mechanical signaling for bone modeling and remodeling

Critical reviews in eukaryotic gene expression, 2009, Vol.19 (4), p.319-338

2009

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Skeletal cell YAP and TAZ combinatorially promote bone development

The FASEB journal, 2018-05, Vol.32 (5), p.2706-2721

2018

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Mechanical Stimulation of Bone in Vivo Reduces Osteocyte Expression of Sost/Sclerostin

The Journal of biological chemistry, 2008-02, Vol.283 (9), p.5866-5875

2008

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Missense Mutations in LRP5 Associated with High Bone Mass Protect the Mouse Skeleton from Disuse- and Ovariectomy-Induced Osteopenia

PloS one, 2015-11, Vol.10 (11), p.e0140775-e0140775

2015

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Lrp5 functions in bone to regulate bone mass

Nature medicine, 2011-06, Vol.17 (6), p.684-691

2011

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Iron deficiency drives an autosomal dominant hypophosphatemic rickets (ADHR) phenotype in fibroblast growth factor-23 (Fgf23) knock-in mice

Proceedings of the National Academy of Sciences - PNAS, 2011-11, Vol.108 (46), p.E1146-E1155

2011

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YAP and TAZ Mediate Osteocyte Perilacunar/Canalicular Remodeling

Journal of bone and mineral research, 2020-01, Vol.35 (1), p.196-210

2020

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The expanding role of Wnt signaling in bone metabolism

Bone (New York, N.Y.), 2013-07, Vol.55 (1), p.256-257

Robling, Alexander G

2013

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Perspective: The current state of Cre driver mouse lines in skeletal research: Challenges and opportunities

Bone (New York, N.Y.), 2023-05, Vol.170, p.116719-116719, Article 116719

2023

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Conditional Deletion of Sost in MSC‐Derived Lineages Identifies Specific Cell‐Type Contributions to Bone Mass and B‐Cell Development

Journal of bone and mineral research, 2018-10, Vol.33 (10), p.1748-1759

2018

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Sensitive detection of Cre-mediated recombination using droplet digital PCR reveals Tg(BGLAP-Cre) and Tg(DMP1-Cre) are active in multiple non-skeletal tissues

Bone (New York, N.Y.), 2021-01, Vol.142, p.115674-115674, Article 115674

2021

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Absence of Cx43 selectively from osteocytes enhances responsiveness to mechanical force in mice

Journal of orthopaedic research, 2013-07, Vol.31 (7), p.1075-1081

2013

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Osteocyte RANKL Drives Bone Resorption in Mouse Ligature-Induced Periodontitis

Journal of bone and mineral research, 2023-10, Vol.38 (10), p.1521-1540

2023

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Postnatal β-catenin deletion from Dmp1-expressing osteocytes/osteoblasts reduces structural adaptation to loading, but not periosteal load-induced bone formation

Bone (New York, N.Y.), 2016-07, Vol.88, p.138-145

2016

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The role of Meteorin‐like in skeletal development and bone fracture healing

Journal of orthopaedic research, 2022-11, Vol.40 (11), p.2510-2521

2022

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Targeted inhibition of Wnt signaling with a Clostridioides difficile toxin B fragment suppresses breast cancer tumor growth

PLoS biology, 2023-11, Vol.21 (11), p.e3002353-e3002353

2023

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The Osteocyte: New Insights

The Wnt pathway: An important control mechanism in bone's response to mechanical loading

An osteocalcin-deficient mouse strain without endocrine abnormalities

Is Bone's Response to Mechanical Signals Dominated by Muscle Forces?

Bone and skeletal muscle: Key players in mechanotransduction and potential overlapping mechanisms

Biomechanical and molecular regulation of bone remodeling

The Skeleton as a Secretory Organ

Sost downregulation and local Wnt signaling are required for the osteogenic response to mechanical loading

Control of bone mass and remodeling by PTH receptor signaling in osteocytes

Mechanical signaling for bone modeling and remodeling

Skeletal cell YAP and TAZ combinatorially promote bone development

Mechanical Stimulation of Bone in Vivo Reduces Osteocyte Expression of Sost/Sclerostin

Missense Mutations in LRP5 Associated with High Bone Mass Protect the Mouse Skeleton from Disuse- and Ovariectomy-Induced Osteopenia

Lrp5 functions in bone to regulate bone mass

Iron deficiency drives an autosomal dominant hypophosphatemic rickets (ADHR) phenotype in fibroblast growth factor-23 (Fgf23) knock-in mice

YAP and TAZ Mediate Osteocyte Perilacunar/Canalicular Remodeling

The expanding role of Wnt signaling in bone metabolism

Perspective: The current state of Cre driver mouse lines in skeletal research: Challenges and opportunities

Conditional Deletion of Sost in MSC‐Derived Lineages Identifies Specific Cell‐Type Contributions to Bone Mass and B‐Cell Development

Sensitive detection of Cre-mediated recombination using droplet digital PCR reveals Tg(BGLAP-Cre) and Tg(DMP1-Cre) are active in multiple non-skeletal tissues

Absence of Cx43 selectively from osteocytes enhances responsiveness to mechanical force in mice

Osteocyte RANKL Drives Bone Resorption in Mouse Ligature-Induced Periodontitis

Postnatal β-catenin deletion from Dmp1-expressing osteocytes/osteoblasts reduces structural adaptation to loading, but not periosteal load-induced bone formation

The role of Meteorin‐like in skeletal development and bone fracture healing

Targeted inhibition of Wnt signaling with a Clostridioides difficile toxin B fragment suppresses breast cancer tumor growth

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