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Different mechanisms contribute to motor cortex hyperexcitability in amyotrophic lateral sclerosis

Clinical neurophysiology, 2002-11, Vol.113 (11), p.1688-1697

2002

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Detection of Pathologic Prion Protein in the Olfactory Epithelium in Sporadic Creutzfeldt–Jakob Disease

The New England journal of medicine, 2003-02, Vol.348 (8), p.711-719

2003

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Cerebral amyloidoses: molecular pathways and therapeutic challenges

Current medicinal chemistry, 2006-07, Vol.13 (16), p.1903-1913

2006

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pH-dependent Prion Protein Conformation in Classical Creutzfeldt-Jakob Disease

The Journal of biological chemistry, 2001-11, Vol.276 (44), p.40377-40380

2001

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Two-dimensional mapping of three phenotype-associated isoforms of the prion protein in sporadic Creutzfeldt-Jakob disease

Electrophoresis, 2002-01, Vol.23 (2), p.347-355

2002

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Hepatitis C virus infection of peripheral nerves in type II cryoglobulinaemia

Virchows Archiv : an international journal of pathology, 1999-06, Vol.434 (6), p.533-535

1999

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Critical illness myopathy and neuropathy

The Lancet (British edition), 1996-06, Vol.347 (9015), p.1579-1582

1996

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Different Prion Conformers Target the Olfactory Pathway in Sporadic Creutzfeldt-Jakob Disease

Annals of the New York Academy of Sciences, 2009-07, Vol.1170 (1), p.637-643

2009

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Activation of NF-κB and c-jun transcription factors in multiple sclerosis lesions: Implications for oligodendrocyte pathology

The American journal of pathology, 1999-11, Vol.155 (5), p.1433-1438

1999

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Experimental Induction of Myelin Changes by Anti-MAG Antibodies and Terminal Complement Complex

Journal of neuropathology and experimental neurology, 1995-01, Vol.54 (1), p.96-104

1995

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Novel prion protein conformation and glycotype in Creutzfeldt-Jakob disease

Archives of neurology (Chicago), 2007-04, Vol.64 (4), p.595

2007

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Multineuropathy in a patient with HBV infection, polyarteritis nodosa and celiac disease

Rheumatology international, 2009-03, Vol.29 (5), p.579-581

2009

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A 49-YEAR-OLD MAN WITH NEUROPSYCHIATRIC SYMPTOMS FOLLOWED BY PROGRESSIVE COGNITIVE DECLINE

Brain pathology (Zurich, Switzerland), 2006-07, Vol.16 (3), p.237-238

2006

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T-cell-mediated epineurial vasculitis and humoral-mediated microangiopathy in cryoglobulinemic neuropathy

Journal of neuroimmunology, 1997-03, Vol.73 (1), p.145-154

1997

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Clearance of 14-3-3 Protein from Cerebrospinal Fluid Heralds the Resolution of Bacterial Meningitis

Clinical infectious diseases, 2003-06, Vol.36 (11), p.1492-1495

2003

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Decerebrate rigidity in acute head injury

Journal of neurosurgery, 1977-11, Vol.47 (5), p.680-698

1977

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Antisulfatide polyneuropathy : antibody-mediated complement attack on peripheral myelin

Acta neuropathologica, 1998-12, Vol.96 (6), p.569-574

1998

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Paired Helical Filaments from Alzheimer Disease Patients Contain Cytoskeletal Components

Proceedings of the National Academy of Sciences - PNAS, 1985-06, Vol.82 (11), p.3916-3920

1985

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Simultaneous occurrence of spongiform encephalopathy in a man and his cat in Italy

The Lancet (British edition), 1998-10, Vol.352 (9134), p.1116-1117

1998

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Complement-Mediated Demyelination in Patients with IgM Monoclonal Gammopathy and Polyneuropathy

The New England journal of medicine, 1990-03, Vol.322 (10), p.649-652

1990

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Activation of NF-κB and c- jun Transcription Factors in Multiple Sclerosis Lesions

The American journal of pathology, 1999-11, Vol.155 (5), p.1433-1438

1999

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Systemic passive transfer studies using IgM monoclonal antibodies to sulfatide

Journal of neuroimmunology, 1995-12, Vol.63 (1), p.29-37

1995

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Activation of NF-kappaB and c-jun transcription factors in multiple sclerosis lesions. Implications for oligodendrocyte pathology

The American journal of pathology, 1999-11, Vol.155 (5), p.1433

1999

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Charcot-Marie-Tooth disease: an intermediate form

Neuromuscular disorders : NMD, 1998-08, Vol.8 (6), p.392-393

1998

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Mutations associated with very late-onset metachromatic leukodystrophy

Clinical genetics, 1999-02, Vol.55 (2), p.130-130

1999

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Different mechanisms contribute to motor cortex hyperexcitability in amyotrophic lateral sclerosis

Detection of Pathologic Prion Protein in the Olfactory Epithelium in Sporadic Creutzfeldt–Jakob Disease

Cerebral amyloidoses: molecular pathways and therapeutic challenges

pH-dependent Prion Protein Conformation in Classical Creutzfeldt-Jakob Disease

Two-dimensional mapping of three phenotype-associated isoforms of the prion protein in sporadic Creutzfeldt-Jakob disease

Hepatitis C virus infection of peripheral nerves in type II cryoglobulinaemia

Critical illness myopathy and neuropathy

Different Prion Conformers Target the Olfactory Pathway in Sporadic Creutzfeldt-Jakob Disease

Activation of NF-κB and c-jun transcription factors in multiple sclerosis lesions: Implications for oligodendrocyte pathology

Experimental Induction of Myelin Changes by Anti-MAG Antibodies and Terminal Complement Complex

Novel prion protein conformation and glycotype in Creutzfeldt-Jakob disease

Multineuropathy in a patient with HBV infection, polyarteritis nodosa and celiac disease

A 49-YEAR-OLD MAN WITH NEUROPSYCHIATRIC SYMPTOMS FOLLOWED BY PROGRESSIVE COGNITIVE DECLINE

T-cell-mediated epineurial vasculitis and humoral-mediated microangiopathy in cryoglobulinemic neuropathy

Clearance of 14-3-3 Protein from Cerebrospinal Fluid Heralds the Resolution of Bacterial Meningitis

Decerebrate rigidity in acute head injury

Antisulfatide polyneuropathy : antibody-mediated complement attack on peripheral myelin

Paired Helical Filaments from Alzheimer Disease Patients Contain Cytoskeletal Components

Simultaneous occurrence of spongiform encephalopathy in a man and his cat in Italy

Complement-Mediated Demyelination in Patients with IgM Monoclonal Gammopathy and Polyneuropathy

Activation of NF-κB and c- jun Transcription Factors in Multiple Sclerosis Lesions

Systemic passive transfer studies using IgM monoclonal antibodies to sulfatide

Activation of NF-kappaB and c-jun transcription factors in multiple sclerosis lesions. Implications for oligodendrocyte pathology

Charcot-Marie-Tooth disease: an intermediate form

Mutations associated with very late-onset metachromatic leukodystrophy

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