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Leptin and CCK selectively activate vagal afferent neurons innervating the stomach and duodenum

American journal of physiology. Regulatory, integrative and comparative physiology, 2006-06, Vol.290 (6), p.R1544-R1549

2006

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Capsaicin-induced neuronal death and proliferation of the primary sensory neurons located in the nodose ganglia of adult rats

Neuroscience, 2008-06, Vol.154 (2), p.621-630

2008

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Leptin-induced satiation mediated by abdominal vagal afferents

American journal of physiology. Regulatory, integrative and comparative physiology, 2005-04, Vol.288 (4), p.R879-R884

2005

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Cooperative Activation of Cultured Vagal Afferent Neurons by Leptin and Cholecystokinin

Endocrinology (Philadelphia), 2004-08, Vol.145 (8), p.3652-3657

2004

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NMDA NR2 receptors participate in CCK-induced reduction of food intake and hindbrain neuronal activation

Brain research, 2009-04, Vol.1266, p.37-44

2009

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Hindbrain administration of NMDA receptor antagonist AP-5 increases food intake in the rat

American journal of physiology. Regulatory, integrative and comparative physiology, 2006-03, Vol.290 (3), p.R642-R651

2006

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NMDA channels control meal size via central vagal afferent terminals

American journal of physiology. Regulatory, integrative and comparative physiology, 2005-11, Vol.289 (5), p.R1504-R1511

2005

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Leptin and CCK modulate complementary background conductances to depolarize cultured nodose neurons

American Journal of Physiology: Cell Physiology, 2006-02, Vol.290 (2), p.C427-C432

2006

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Leptin Analog Antagonizes Leptin Effects on Food Intake and Body Weight but Mimics Leptin-Induced Vagal Afferent Activation

Endocrinology (Philadelphia), 2007-06, Vol.148 (6), p.2878-2885

2007

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CCK enhances response to gastric distension by acting on capsaicin-insensitive vagal afferents

American journal of physiology. Regulatory, integrative and comparative physiology, 2005-09, Vol.289 (3), p.R695-R703

2005

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Gastrointestinal mechanisms of satiation for food

Physiology & behavior, 2004-04, Vol.81 (2), p.249-273

Ritter, Robert C

2004

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Changes in microglial activation within the hindbrain, nodose ganglia, and the spinal cord following subdiaphragmatic vagotomy

Neuroscience letters, 2012-03, Vol.513 (1), p.31-36

2012

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Cholecystokinin: proofs and prospects for involvement in control of food intake and body weight

Neuropeptides (Edinburgh), 1999-10, Vol.33 (5), p.387-399

1999

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Intracerebroventricular administration of MK-801 increases food intake through mechanisms independent of gastric emptying

American journal of physiology. Regulatory, integrative and comparative physiology, 2004-12, Vol.287 (6), p.R1462-R1467

2004

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High fat maintenance diet attenuates hindbrain neuronal response to CCK

Regulatory peptides, 2000-01, Vol.86 (1), p.83-88

2000

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The Non-Competitive NMDA Antagonist MK-801 Increases Food Intake in Rats

Pharmacology, biochemistry and behavior, 1997-01, Vol.56 (1), p.145-149

1997

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Lesions of the dorsal vagal complex abolish increases in meal size induced by NMDA receptor blockade

Brain research, 2000-07, Vol.872 (1), p.37-43

2000

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Torsion balances, torsion pendulums, and related devices

Review of scientific instruments, 1993-02, Vol.64 (2), p.283-309

1993

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Visceral afferent participation in delayed satiation following NMDA receptor blockade

Physiology & behavior, 1998-11, Vol.65 (2), p.361-366

1998

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Experimental study of the magnetic stereotaxis system for catheter manipulation within the brain

Journal of neurosurgery, 2000-08, Vol.93 (2), p.282

2000

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Measurement of the force required to move a neurosurgical probe through in vivo human brain tissue

IEEE transactions on biomedical engineering, 1999-07, Vol.46 (7), p.891-894

1999

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IEEE_Xplore

Delay in meal termination follows blockade of N-methyl- d-aspartate receptors in the dorsal hindbrain

Brain research, 1998-11, Vol.810 (1), p.34-40

1998

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The noncalcemic analogue of vitamin D, 22-oxacalcitriol, suppresses parathyroid hormone synthesis and secretion

The Journal of clinical investigation, 1989-09, Vol.84 (3), p.728-732

1989

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CCK-A receptor activation induces Fos expression in myenteric neurons of rat small intestine

Regulatory peptides, 2000-03, Vol.88 (1), p.75-81

2000

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CCK-receptor antagonists attenuate suppression of sham feeding by intestinal nutrients

American journal of physiology. Regulatory, integrative and comparative physiology, 1992-04, Vol.262 (4), p.554-R561

1992

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Leptin and CCK selectively activate vagal afferent neurons innervating the stomach and duodenum

Capsaicin-induced neuronal death and proliferation of the primary sensory neurons located in the nodose ganglia of adult rats

Leptin-induced satiation mediated by abdominal vagal afferents

Cooperative Activation of Cultured Vagal Afferent Neurons by Leptin and Cholecystokinin

NMDA NR2 receptors participate in CCK-induced reduction of food intake and hindbrain neuronal activation

Hindbrain administration of NMDA receptor antagonist AP-5 increases food intake in the rat

NMDA channels control meal size via central vagal afferent terminals

Leptin and CCK modulate complementary background conductances to depolarize cultured nodose neurons

Leptin Analog Antagonizes Leptin Effects on Food Intake and Body Weight but Mimics Leptin-Induced Vagal Afferent Activation

CCK enhances response to gastric distension by acting on capsaicin-insensitive vagal afferents

Gastrointestinal mechanisms of satiation for food

Changes in microglial activation within the hindbrain, nodose ganglia, and the spinal cord following subdiaphragmatic vagotomy

Cholecystokinin: proofs and prospects for involvement in control of food intake and body weight

Intracerebroventricular administration of MK-801 increases food intake through mechanisms independent of gastric emptying

High fat maintenance diet attenuates hindbrain neuronal response to CCK

The Non-Competitive NMDA Antagonist MK-801 Increases Food Intake in Rats

Lesions of the dorsal vagal complex abolish increases in meal size induced by NMDA receptor blockade

Torsion balances, torsion pendulums, and related devices

Visceral afferent participation in delayed satiation following NMDA receptor blockade

Experimental study of the magnetic stereotaxis system for catheter manipulation within the brain

Measurement of the force required to move a neurosurgical probe through in vivo human brain tissue

Delay in meal termination follows blockade of N-methyl- d-aspartate receptors in the dorsal hindbrain

The noncalcemic analogue of vitamin D, 22-oxacalcitriol, suppresses parathyroid hormone synthesis and secretion

CCK-A receptor activation induces Fos expression in myenteric neurons of rat small intestine

CCK-receptor antagonists attenuate suppression of sham feeding by intestinal nutrients

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