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Amplified behavioral and endocrine responses to forced swim stress in the Wistar–Kyoto rat

Psychoneuroendocrinology, 2002-04, Vol.27 (3), p.303-318

2002

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Sexually dimorphic effects of maternal adrenalectomy on hypothalamic corticotrophin-releasing factor, glucocorticoid receptor and anterior pituitary POMC mRNA levels in rat neonates

Brain research. Developmental brain research, 1997-06, Vol.100 (2), p.198-204

1997

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Amygdaloid lesions: differential effect on conditioned stress and immobilization-induced increases in corticosterone and renin secretion

Neuroendocrinology, 1991-08, Vol.54 (2), p.89-95

1991

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Comparison of neuroendocrine and behavioral effects of ipsapirone, a 5-HT1A agonist, in three stress paradigms: immobilization, forced swim and conditioned fear

Brain research, 1992-05, Vol.580 (1-2), p.205-214

1992

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IL-22's gut feeling

SciBX, 2008-03, Vol.1 (7), p.155-155

Rittenhouse, Peter A.

2008

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Thyroxine administration prevents streptococcal cell wall-induced inflammatory responses

Endocrinology (Philadelphia), 1997-04, Vol.138 (4), p.1434-1439

1997

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Thyroxine Administration Prevents Streptococcal Cell Wall-Induced Inflammatory Responses1

Endocrinology (Philadelphia), 1997-04, Vol.138 (4), p.1434-1439

1997

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Serotonergic regulation of renin and prolactin secretion

Behavioural brain research, 1995-12, Vol.73 (1), p.203-208

1995

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Ibotenic acid lesions in the bed nucleus of the stria terminalis attenuate conditioned stress-induced increases in prolactin, ACTH and corticosterone

Neuroendocrinology, 1993-03, Vol.57 (3), p.517-524

1993

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A Novel Endogenous Corticotropin Release Inhibiting Factor

Annals of the New York Academy of Sciences, 1998-05, Vol.840 (1), p.456-469

1998

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Hypothalamic paraventricular, but not supraoptic neurons, mediate the serotonergic stimulation of oxytocin secretion

Brain research bulletin, 1995, Vol.36 (1), p.45-50

1995

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Streptozotocin-induced diabetes is associated with altered expression of peptide-encoding mRNAs in rat sensory neurons

Peptides (New York, N.Y. : 1980), 1996, Vol.17 (6), p.1017-1022

1996

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Repeated cocaine modifies the neuroendocrine responses to the 5-HT1C/5-HT2 receptor agonist DOI

European journal of pharmacology, 1992-10, Vol.221 (1), p.121-127

1992

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Streptozotocin-induced diabetes produces a decrease in pituitary substance P content and preprotachykinin mRNA

Neuroscience letters, 1996-06, Vol.211 (2), p.77-80

1996

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Neurons in the hypothalamic paraventricular nucleus mediate the serotonergic stimulation of renin secretion

Brain research, 1992-10, Vol.593 (1), p.105-113

1992

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Influence of repeated cocaine exposure on the endocrine and behavioral responses to stress in rats

Psychopharmacologia, 1994, Vol.113 (3-4), p.547-554

1994

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Investigation of the role of 5-HT3 receptors in the secretion of prolactin, ACTH and renin

Neuroendocrinology, 1993-07, Vol.58 (1), p.65-70

1993

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Neuroendocrine responses to cocaine do not exhibit sensitization following repeated cocaine exposure

Life sciences (1973), 1992, Vol.51 (12), p.887-897

1992

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Repeated exposure to cocaine produces long-lasting deficits in the serotonergic stimulation of prolactin and renin, but not adrenocorticotropin secretion

European journal of pharmacology, 1993-09, Vol.241 (2-3), p.275-278

1993

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Repeated injections of cocaine inhibit the serotonergic regulation of prolactin and renin secretion in rats

Brain research, 1992-05, Vol.580 (1), p.6-11

1992

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Effect of cocaine injections on the neuroendocrine response to the serotonin agonist MK-212

Biological psychiatry (1969), 1992-08, Vol.32 (3), p.258-269

1992

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ICV injection of the serotonin 5-HT1B agonist CP-93, 129 increases the secretion of ACTH, prolactin, and renin and increases blood pressure by nonserotonergic mechanisms

Pharmacology, biochemistry and behavior, 1994-06, Vol.48 (2), p.429-436

1994

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Central stimulation of renin secretion through serotonergic, noncardiovascular mechanisms

Neuroendocrinology, 1994-08, Vol.60 (2), p.205-214

1994

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Acute, but not repeated, cocaine administration decreases renin secretion in the conscious male rat

Neuroscience letters, 1991-06, Vol.127 (2), p.181-184

1991

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Cocaine-induced suppression of renin secretion is partially mediated by serotonergic mechanisms

Pharmacology, biochemistry and behavior, 1992-07, Vol.42 (3), p.481-486

1992

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Amplified behavioral and endocrine responses to forced swim stress in the Wistar–Kyoto rat

Sexually dimorphic effects of maternal adrenalectomy on hypothalamic corticotrophin-releasing factor, glucocorticoid receptor and anterior pituitary POMC mRNA levels in rat neonates

Amygdaloid lesions: differential effect on conditioned stress and immobilization-induced increases in corticosterone and renin secretion

Comparison of neuroendocrine and behavioral effects of ipsapirone, a 5-HT1A agonist, in three stress paradigms: immobilization, forced swim and conditioned fear

IL-22's gut feeling

Thyroxine administration prevents streptococcal cell wall-induced inflammatory responses

Thyroxine Administration Prevents Streptococcal Cell Wall-Induced Inflammatory Responses1

Serotonergic regulation of renin and prolactin secretion

Ibotenic acid lesions in the bed nucleus of the stria terminalis attenuate conditioned stress-induced increases in prolactin, ACTH and corticosterone

A Novel Endogenous Corticotropin Release Inhibiting Factor

Hypothalamic paraventricular, but not supraoptic neurons, mediate the serotonergic stimulation of oxytocin secretion

Streptozotocin-induced diabetes is associated with altered expression of peptide-encoding mRNAs in rat sensory neurons

Repeated cocaine modifies the neuroendocrine responses to the 5-HT1C/5-HT2 receptor agonist DOI

Streptozotocin-induced diabetes produces a decrease in pituitary substance P content and preprotachykinin mRNA

Neurons in the hypothalamic paraventricular nucleus mediate the serotonergic stimulation of renin secretion

Influence of repeated cocaine exposure on the endocrine and behavioral responses to stress in rats

Investigation of the role of 5-HT3 receptors in the secretion of prolactin, ACTH and renin

Neuroendocrine responses to cocaine do not exhibit sensitization following repeated cocaine exposure

Repeated exposure to cocaine produces long-lasting deficits in the serotonergic stimulation of prolactin and renin, but not adrenocorticotropin secretion

Repeated injections of cocaine inhibit the serotonergic regulation of prolactin and renin secretion in rats

Effect of cocaine injections on the neuroendocrine response to the serotonin agonist MK-212

ICV injection of the serotonin 5-HT1B agonist CP-93, 129 increases the secretion of ACTH, prolactin, and renin and increases blood pressure by nonserotonergic mechanisms

Central stimulation of renin secretion through serotonergic, noncardiovascular mechanisms

Acute, but not repeated, cocaine administration decreases renin secretion in the conscious male rat

Cocaine-induced suppression of renin secretion is partially mediated by serotonergic mechanisms

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