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Neuroendocrine and cardiovascular effects of serotonin: selective role of brain angiotensin on vasopressin

American journal of physiology: endocrinology and metabolism, 1996-03, Vol.270 (3), p.E513-E521

1996

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Long-term treatment with the antidepressants fluoxetine and desipramine potentiates endocrine responses to the serotonin agonists 6-chloro-2-[1-piperazinyl]-pyrazine (MK-212) and (+-)-1-(2,5-dimethoxy-4-iodophenyl)-2-aminopropane HCl (DOI)

The Journal of pharmacology and experimental therapeutics, 1993-08, Vol.266 (2), p.836

1993

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Enhanced serotonergic transmission stimulates oxytocin secretion in conscious male rats

The Journal of pharmacology and experimental therapeutics, 1991-04, Vol.257 (1), p.95

1991

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Amygdaloid lesions: differential effect on conditioned stress and immobilization-induced increases in corticosterone and renin secretion

Neuroendocrinology, 1991-08, Vol.54 (2), p.89-95

1991

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Neurons in the hypothalamic paraventricular nucleus mediate the serotonergic stimulation of prolactin secretion via 5-HT1c/2 receptors

Endocrinology (Philadelphia), 1993-08, Vol.133 (2), p.661-667

1993

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Ibotenic acid lesions in the bed nucleus of the stria terminalis attenuate conditioned stress-induced increases in prolactin, ACTH and corticosterone

Neuroendocrinology, 1993-03, Vol.57 (3), p.517-524

1993

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Cocaine-induced elevation of plasma adrenocorticotropin hormone and corticosterone is mediated by serotonergic neurons

The Journal of pharmacology and experimental therapeutics, 1991-11, Vol.259 (2), p.495

1991

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Evidence that the serotonin agonist, DOI, increases renin secretion and blood pressure through both central and peripheral 5-HT2 receptors

The Journal of pharmacology and experimental therapeutics, 1991-10, Vol.259 (1), p.58

1991

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Hypothalamic paraventricular, but not supraoptic neurons, mediate the serotonergic stimulation of oxytocin secretion

Brain research bulletin, 1995, Vol.36 (1), p.45-50

1995

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Investigation of the role of 5-HT3 receptors in the secretion of prolactin, ACTH and renin

Neuroendocrinology, 1993-07, Vol.58 (1), p.65-70

1993

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Prior chronic exposure to cocaine inhibits the serotonergic stimulation of acth and secretion of corticosterone

Neuropharmacology, 1992-02, Vol.31 (2), p.169-175

1992

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Neuroendocrine evidence for denervation supersensitivity of serotonin receptors: effects of the 5-HT agonist RU 24969 on corticotropin, corticosterone, prolactin and renin secretion

The Journal of pharmacology and experimental therapeutics, 1989-11, Vol.251 (2), p.428

1989

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Streptozotocin-induced diabetes produces a decrease in pituitary substance P content and preprotachykinin mRNA

Neuroscience letters, 1996-06, Vol.211 (2), p.77-80

1996

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Neurons in the hypothalamic paraventricular nucleus mediate the serotonergic stimulation of renin secretion

Brain research, 1992-10, Vol.593 (1), p.105-113

1992

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Influence of repeated cocaine exposure on the endocrine and behavioral responses to stress in rats

Psychopharmacologia, 1994, Vol.113 (3-4), p.547-554

1994

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Neuroendocrine responses to cocaine do not exhibit sensitization following repeated cocaine exposure

Life sciences (1973), 1992, Vol.51 (12), p.887-897

1992

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Repeated exposure to cocaine produces long-lasting deficits in the serotonergic stimulation of prolactin and renin, but not adrenocorticotropin secretion

European journal of pharmacology, 1993-09, Vol.241 (2-3), p.275-278

1993

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Serotonergic regulation of renin and prolactin secretion

Behavioural brain research, 1996, Vol.73 (1-2), p.203-208

1996

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Effect of cocaine injections on the neuroendocrine response to the serotonin agonist MK-212

Biological psychiatry (1969), 1992-08, Vol.32 (3), p.258-269

1992

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ICV injection of the serotonin 5-HT1B agonist CP-93, 129 increases the secretion of ACTH, prolactin, and renin and increases blood pressure by nonserotonergic mechanisms

Pharmacology, biochemistry and behavior, 1994-06, Vol.48 (2), p.429-436

1994

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Central stimulation of renin secretion through serotonergic, noncardiovascular mechanisms

Neuroendocrinology, 1994-08, Vol.60 (2), p.205-214

1994

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Acute, but not repeated, cocaine administration decreases renin secretion in the conscious male rat

Neuroscience letters, 1991-06, Vol.127 (2), p.181-184

1991

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Cocaine-induced suppression of renin secretion is partially mediated by serotonergic mechanisms

Pharmacology, biochemistry and behavior, 1992-07, Vol.42 (3), p.481-486

1992

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Cocaine-induced suppression of renin secretion is mediated in the brain: Investigation of cardiovascular and local anesthetic mechanisms

Brain research bulletin, 1992-05, Vol.28 (5), p.837-842

1992

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Amplified behavioral and endocrine responses to forced swim stress in the Wistar–Kyoto rat

Psychoneuroendocrinology, 2002-04, Vol.27 (3), p.303-318

2002

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Neuroendocrine and cardiovascular effects of serotonin: selective role of brain angiotensin on vasopressin

Long-term treatment with the antidepressants fluoxetine and desipramine potentiates endocrine responses to the serotonin agonists 6-chloro-2-[1-piperazinyl]-pyrazine (MK-212) and (+-)-1-(2,5-dimethoxy-4-iodophenyl)-2-aminopropane HCl (DOI)

Enhanced serotonergic transmission stimulates oxytocin secretion in conscious male rats

Amygdaloid lesions: differential effect on conditioned stress and immobilization-induced increases in corticosterone and renin secretion

Neurons in the hypothalamic paraventricular nucleus mediate the serotonergic stimulation of prolactin secretion via 5-HT1c/2 receptors

Ibotenic acid lesions in the bed nucleus of the stria terminalis attenuate conditioned stress-induced increases in prolactin, ACTH and corticosterone

Cocaine-induced elevation of plasma adrenocorticotropin hormone and corticosterone is mediated by serotonergic neurons

Evidence that the serotonin agonist, DOI, increases renin secretion and blood pressure through both central and peripheral 5-HT2 receptors

Hypothalamic paraventricular, but not supraoptic neurons, mediate the serotonergic stimulation of oxytocin secretion

Investigation of the role of 5-HT3 receptors in the secretion of prolactin, ACTH and renin

Prior chronic exposure to cocaine inhibits the serotonergic stimulation of acth and secretion of corticosterone

Neuroendocrine evidence for denervation supersensitivity of serotonin receptors: effects of the 5-HT agonist RU 24969 on corticotropin, corticosterone, prolactin and renin secretion

Streptozotocin-induced diabetes produces a decrease in pituitary substance P content and preprotachykinin mRNA

Neurons in the hypothalamic paraventricular nucleus mediate the serotonergic stimulation of renin secretion

Influence of repeated cocaine exposure on the endocrine and behavioral responses to stress in rats

Neuroendocrine responses to cocaine do not exhibit sensitization following repeated cocaine exposure

Repeated exposure to cocaine produces long-lasting deficits in the serotonergic stimulation of prolactin and renin, but not adrenocorticotropin secretion

Serotonergic regulation of renin and prolactin secretion

Effect of cocaine injections on the neuroendocrine response to the serotonin agonist MK-212

ICV injection of the serotonin 5-HT1B agonist CP-93, 129 increases the secretion of ACTH, prolactin, and renin and increases blood pressure by nonserotonergic mechanisms

Central stimulation of renin secretion through serotonergic, noncardiovascular mechanisms

Acute, but not repeated, cocaine administration decreases renin secretion in the conscious male rat

Cocaine-induced suppression of renin secretion is partially mediated by serotonergic mechanisms

Cocaine-induced suppression of renin secretion is mediated in the brain: Investigation of cardiovascular and local anesthetic mechanisms

Amplified behavioral and endocrine responses to forced swim stress in the Wistar–Kyoto rat

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