UB Paderborn / Katalog / Suche / in CDI

1 – 25 von 31

Sortiert nach

Aβ-induced acceleration of Alzheimer-related τ-pathology spreading and its association with prion protein

Acta neuropathologica, 2019-12, Vol.138 (6), p.913-941

2019

Volltextzugriff (PDF)

Biochemical stages of amyloid-β peptide aggregation and accumulation in the human brain and their association with symptomatic and pathologically preclinical Alzheimer's disease

Brain (London, England : 1878), 2014-03, Vol.137 (Pt 3), p.887-903

2014

Volltextzugriff (PDF)

Pathology of clinical and preclinical Alzheimer’s disease

European archives of psychiatry and clinical neuroscience, 2013-11, Vol.263 (Suppl 2), p.137-145

2013

Volltextzugriff (PDF)

Different aspects of Alzheimer's disease-related amyloid β-peptide pathology and their relationship to amyloid positron emission tomography imaging and dementia

Acta neuropathologica communications, 2019-11, Vol.7 (1), p.178-178, Article 178

2019

Volltextzugriff (PDF)

Modified amyloid variants in pathological subgroups of β‐amyloidosis

Annals of clinical and translational neurology, 2018-07, Vol.5 (7), p.815-831

2018

Volltextzugriff (PDF)

Impact of amyloid β aggregate maturation on antibody treatment in APP23 mice

Acta neuropathologica communications, 2015-07, Vol.3 (1), p.41-41, Article 41

2015

Volltextzugriff (PDF)

Seeding, maturation and propagation of amyloid β-peptide aggregates in Alzheimer's disease

Brain (London, England : 1878), 2022-10, Vol.145 (10), p.3558-3570

2022

Volltextzugriff (PDF)

High‐molecular weight Aβ oligomers and protofibrils are the predominant Aβ species in the native soluble protein fraction of the AD brain

Journal of cellular and molecular medicine, 2012-02, Vol.16 (2), p.287-295

2012

Volltextzugriff (PDF)

Dispersible amyloid β-protein oligomers, protofibrils, and fibrils represent diffusible but not soluble aggregates: their role in neurodegeneration in amyloid precursor protein (APP) transgenic mice

Neurobiology of aging, 2012-11, Vol.33 (11), p.2641-2660

2012

Volltextzugriff (PDF)

Correction to: Different aspects of Alzheimer’s disease-related amyloid β-peptide pathology and their relationship to amyloid positron emission tomography imaging and dementia

Acta neuropathologica communications, 2020-08, Vol.8 (1), p.1-121, Article 121

2020

Volltextzugriff (PDF)

The type of Aβ-related neuronal degeneration differs between amyloid precursor protein (APP23) and amyloid β-peptide (APP48) transgenic mice

Acta neuropathologica communications, 2013-11, Vol.1 (1), p.77-77, Article 77

2013

Volltextzugriff (PDF)

A[beta]-induced acceleration of Alzheimer-related [tau]-pathology spreading and its association with prion protein

Acta neuropathologica, 2019-12, Vol.138 (6), p.913

2019

Volltextzugriff (PDF)

Pyroglutamylated amyloid-β is associated with hyperphosphorylated tau and severity of Alzheimer’s disease

Acta neuropathologica, 2014-07, Vol.128 (1), p.67-79

2014

Volltextzugriff (PDF)

Transgenic expression of intraneuronal Aβ42 but not Aβ40 leads to cellular Aβ lesions, degeneration, and functional impairment without typical Alzheimer's disease pathology

The Journal of neuroscience, 2012-01, Vol.32 (4), p.1273-1283

2012

Volltextzugriff (PDF)

Cerebral Small Vessel Disease-Induced Apolipoprotein E Leakage Is Associated With Alzheimer Disease and the Accumulation of Amyloid β-Protein in Perivascular Astrocytes

Journal of neuropathology and experimental neurology, 2008-09, Vol.67 (9), p.842-856

2008

Volltextzugriff (PDF)

Pyroglutamylated amyloid-[beta] is associated with hyperphosphorylated tau and severity of Alzheimer's disease

Acta neuropathologica, 2014-07, Vol.128 (1), p.67

2014

Volltextzugriff (PDF)

Correction to: Different aspects of Alzheimer's disease-related amyloid [beta]-peptide pathology and their relationship to amyloid positron emission tomography imaging and dementia

Acta neuropathologica communications, 2020-08, Vol.8 (1)

2020

Volltextzugriff (PDF)

Amyloid-β protein modulates the perivascular clearance of neuronal apolipoprotein E in mouse models of Alzheimer’s disease

Journal of Neural Transmission, 2011-05, Vol.118 (5), p.699-712

2011

Volltextzugriff (PDF)

Different aspects of Alzheimer's disease-related amyloid [beta]-peptide pathology and their relationship to amyloid positron emission tomography imaging and dementia

Acta neuropathologica communications, 2019-11, Vol.7 (1)

2019

Volltextzugriff (PDF)

P2–058: Pyroglutamylated beta‐amyloid is associated with hyperphosphorylated tau in human post‐mortem brains

Alzheimer's & dementia, 2013-07, Vol.9 (4S_Part_9), p.P364-P364

2013

Volltextzugriff (PDF)

Pyroglutamylated beta-amyloid is associated with hyperphosphorylated tau in human post-mortem brains

Alzheimer's & dementia, 2013-07, Vol.9 (4), p.P364-P364

2013

Volltextzugriff (PDF)

S3‐03‐06: tau pathology in Young people and its progression Before Abeta Deposition

Alzheimer's & dementia, 2011-07, Vol.7 (4S_Part_14), p.S492-S492

2011

Volltextzugriff (PDF)

tau pathology in Young people and its progression Before Abeta Deposition

Alzheimer's & dementia, 2011-07, Vol.7 (4), p.S492-S492

2011

Volltextzugriff (PDF)

Transgenic Expression of Intraneuronal Aβ 42 But Not Aβ 40 Leads to Cellular Aβ Lesions, Degeneration, and Functional Impairment without Typical Alzheimer's Disease Pathology

The Journal of neuroscience, 2012-01, Vol.32 (4), p.1273-1283

2012

Volltextzugriff (PDF)

Impact of amyloid [beta] aggregate maturation on antibody treatment in APP23 mice

Acta neuropathologica communications, 2015-07, Vol.3

2015

Volltextzugriff (PDF)

1 – 25 von 31

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(30)
Mit Volltextzugriff
(31)

Publikationsform

Artikel
(31)

Sprache

Englisch
(31)
Japanisch
(1)

Erscheinungsjahr

n.n

Thema

Alzheimer's Disease
(15)
Life Sciences & Biomedicine
(13)
Neurosciences
(13)
Science & Technology
(13)
Neurosciences & Neurology
(12)
Alzheimer Disease - Pathology
(11)
Humans
(11)
Amyloid Beta-Peptides - Metabolism
(10)
Brain - Pathology
(9)
Clinical Neurology
(8)
Neurology
(8)
Alzheimer Disease - Metabolism
(7)
Female
(7)
Pathology
(7)
Animals
(6)
Male
(6)
Mice, Transgenic
(6)
Aged
(5)
Amyloid Beta-Protein Precursor - Genetics
(5)
Dementia
(5)

15 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

Aβ-induced acceleration of Alzheimer-related τ-pathology spreading and its association with prion protein

Biochemical stages of amyloid-β peptide aggregation and accumulation in the human brain and their association with symptomatic and pathologically preclinical Alzheimer's disease

Pathology of clinical and preclinical Alzheimer’s disease

Different aspects of Alzheimer's disease-related amyloid β-peptide pathology and their relationship to amyloid positron emission tomography imaging and dementia

Modified amyloid variants in pathological subgroups of β‐amyloidosis

Impact of amyloid β aggregate maturation on antibody treatment in APP23 mice

Seeding, maturation and propagation of amyloid β-peptide aggregates in Alzheimer's disease

High‐molecular weight Aβ oligomers and protofibrils are the predominant Aβ species in the native soluble protein fraction of the AD brain

Dispersible amyloid β-protein oligomers, protofibrils, and fibrils represent diffusible but not soluble aggregates: their role in neurodegeneration in amyloid precursor protein (APP) transgenic mice

Correction to: Different aspects of Alzheimer’s disease-related amyloid β-peptide pathology and their relationship to amyloid positron emission tomography imaging and dementia

The type of Aβ-related neuronal degeneration differs between amyloid precursor protein (APP23) and amyloid β-peptide (APP48) transgenic mice

A[beta]-induced acceleration of Alzheimer-related [tau]-pathology spreading and its association with prion protein

Pyroglutamylated amyloid-β is associated with hyperphosphorylated tau and severity of Alzheimer’s disease

Transgenic expression of intraneuronal Aβ42 but not Aβ40 leads to cellular Aβ lesions, degeneration, and functional impairment without typical Alzheimer's disease pathology

Cerebral Small Vessel Disease-Induced Apolipoprotein E Leakage Is Associated With Alzheimer Disease and the Accumulation of Amyloid β-Protein in Perivascular Astrocytes

Pyroglutamylated amyloid-[beta] is associated with hyperphosphorylated tau and severity of Alzheimer's disease

Correction to: Different aspects of Alzheimer's disease-related amyloid [beta]-peptide pathology and their relationship to amyloid positron emission tomography imaging and dementia

Amyloid-β protein modulates the perivascular clearance of neuronal apolipoprotein E in mouse models of Alzheimer’s disease

Different aspects of Alzheimer's disease-related amyloid [beta]-peptide pathology and their relationship to amyloid positron emission tomography imaging and dementia

P2–058: Pyroglutamylated beta‐amyloid is associated with hyperphosphorylated tau in human post‐mortem brains

Pyroglutamylated beta-amyloid is associated with hyperphosphorylated tau in human post-mortem brains

S3‐03‐06: tau pathology in Young people and its progression Before Abeta Deposition

tau pathology in Young people and its progression Before Abeta Deposition

Transgenic Expression of Intraneuronal Aβ 42 But Not Aβ 40 Leads to Cellular Aβ Lesions, Degeneration, and Functional Impairment without Typical Alzheimer's Disease Pathology

Impact of amyloid [beta] aggregate maturation on antibody treatment in APP23 mice

Suchergebnisse filtern