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The identification and characterization of breast cancer CTCs competent for brain metastasis

Science translational medicine, 2013-04, Vol.5 (180), p.180ra48

2013

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Sustained Activation of JNK/p38 MAPK Pathways in Response to Cisplatin Leads to Fas Ligand Induction and Cell Death in Ovarian Carcinoma Cells

The Journal of biological chemistry, 2003-05, Vol.278 (21), p.19245-19256

2003

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Heparanase-induced GEF-H1 signaling regulates the cytoskeletal dynamics of brain metastatic breast cancer cells

Molecular cancer research, 2012-06, Vol.10 (6), p.689-702

2012

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Modulation of GEF-H1 Induced Signaling by Heparanase in Brain Metastatic Melanoma Cells

Journal of cellular biochemistry, 2010-12, Vol.111 (5), p.1299-1309

2010

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MAGI-1 interacts with Slo1 channel proteins and suppresses Slo1 expression on the cell surface

American Journal of Physiology: Cell Physiology, 2009-07, Vol.297 (1), p.C55-C65

2009

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Heparanase modulates Shh and Wnt3a signaling in human medulloblastoma cells

Experimental and therapeutic medicine, 2011-03, Vol.2 (2), p.229-237

2011

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Interactions with Filamin A Stimulate Surface Expression of Large-Conductance Ca2+-Activated K+ Channels in the Absence of Direct Actin Binding

Molecular pharmacology, 2007-09, Vol.72 (3), p.622-630

2007

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Cisplatin resistance in an ovarian carcinoma is associated with a defect in programmed cell death control through XIAP regulation

Oncology research, 2003-01, Vol.13 (6-10), p.399-404

2003

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Activation of the JNK/SAPK and P38 Mitogen-Activated Protein Kinase Signaling Pathways Sensitize Tumor Cells to Cisplatin-Induced Apoptosis

TheScientificWorld, 2001, Vol.1, p.35-35

2001

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Alternatively spliced C-terminal domains regulate the surface expression of large conductance calcium-activated potassium channels

Neuroscience, 2007-06, Vol.146 (4), p.1652-1661

2007

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Abstract 3190: Heparanase modulates Shh and Wnt3a signaling in human medulloblastoma cells

Cancer research (Chicago, Ill.), 2010-04, Vol.70 (8_Supplement), p.3190-3190

2010

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MAGI-1 interacts with Slo1 channel proteins and suppresses Slo1 expression on the cell surface

American Journal of Physiology: Cell Physiology, 2009, Vol.66 (1)

2009

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Interactions with Filamin A Stimulate Surface Expression of Large-Conductance Ca 2+ -Activated K + Channels in the Absence of Direct Actin Binding

Molecular pharmacology, 2007-09, Vol.72 (3), p.622-630

2007

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β1-Subunits Increase Surface Expression of a Large-Conductance Ca 2+ -Activated K + Channel Isoform

Journal of neurophysiology, 2007-05, Vol.97 (5), p.3508-3516

2007

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beta1-Subunits Increase Surface Expression of a Large-Conductance Ca2+-Activated K+ Channel Isoform

Journal of neurophysiology, 2007-05, Vol.97 (5), p.3508-3516

2007

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beta 1-Subunits Increase Surface Expression of a Large-Conductance Ca super(2+)-Activated K super(+) Channel Isoform

Journal of neurophysiology, 2007-05, Vol.97 (5), p.3508-3516

2007

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ACTIVATION OF THE JNK/SAPK AND P38 MITOGEN-ACTIVATED PROTEIN KINASE SIGNALING PATHWAYS SENSITIZE TUMOR CELLS TO CISPLATIN-INDUCED APOPTOSIS

TheScientificWorld, 2001-01, Vol.1 (S3), p.35-35

2001

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The identification and characterization of breast cancer CTCs competent for brain metastasis

Sustained Activation of JNK/p38 MAPK Pathways in Response to Cisplatin Leads to Fas Ligand Induction and Cell Death in Ovarian Carcinoma Cells

Heparanase-induced GEF-H1 signaling regulates the cytoskeletal dynamics of brain metastatic breast cancer cells

Modulation of GEF-H1 Induced Signaling by Heparanase in Brain Metastatic Melanoma Cells

MAGI-1 interacts with Slo1 channel proteins and suppresses Slo1 expression on the cell surface

Heparanase modulates Shh and Wnt3a signaling in human medulloblastoma cells

Interactions with Filamin A Stimulate Surface Expression of Large-Conductance Ca2+-Activated K+ Channels in the Absence of Direct Actin Binding

Cisplatin resistance in an ovarian carcinoma is associated with a defect in programmed cell death control through XIAP regulation

Activation of the JNK/SAPK and P38 Mitogen-Activated Protein Kinase Signaling Pathways Sensitize Tumor Cells to Cisplatin-Induced Apoptosis

Alternatively spliced C-terminal domains regulate the surface expression of large conductance calcium-activated potassium channels

Abstract 3190: Heparanase modulates Shh and Wnt3a signaling in human medulloblastoma cells

MAGI-1 interacts with Slo1 channel proteins and suppresses Slo1 expression on the cell surface

Interactions with Filamin A Stimulate Surface Expression of Large-Conductance Ca 2+ -Activated K + Channels in the Absence of Direct Actin Binding

β1-Subunits Increase Surface Expression of a Large-Conductance Ca 2+ -Activated K + Channel Isoform

beta1-Subunits Increase Surface Expression of a Large-Conductance Ca2+-Activated K+ Channel Isoform

beta 1-Subunits Increase Surface Expression of a Large-Conductance Ca super(2+)-Activated K super(+) Channel Isoform

ACTIVATION OF THE JNK/SAPK AND P38 MITOGEN-ACTIVATED PROTEIN KINASE SIGNALING PATHWAYS SENSITIZE TUMOR CELLS TO CISPLATIN-INDUCED APOPTOSIS

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