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Tumor Suppressor WWOX Contributes to the Elimination of Tumorigenic Cells in Drosophila melanogaster

PloS one, 2015-08, Vol.10 (8), p.e0136356-e0136356

2015

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Molecular Biology of the WWOX Gene That Spans Chromosomal Fragile Site FRA16D

Cells (Basel, Switzerland), 2021-06, Vol.10 (7), p.1637

2021

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The Enemy within: Innate Surveillance-Mediated Cell Death, the Common Mechanism of Neurodegenerative Disease

Frontiers in neuroscience, 2016-05, Vol.10, p.193-193

2016

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Tumor suppressor WWOX moderates the mitochondrial respiratory complex

Genes chromosomes & cancer, 2015-12, Vol.54 (12), p.745-761

2015

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Single-cell sequencing shows mosaic aneuploidy in most human embryos

The Journal of clinical investigation, 2024-03, Vol.134 (6)

2024

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Comparative toxicity of polyglutamine, polyalanine and polyleucine tracts in Drosophila models of expanded repeat disease

Human molecular genetics, 2012-02, Vol.21 (3), p.536-547

2012

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Common chromosomal fragile sites and cancer: Focus on FRA16D

Cancer letters, 2006-01, Vol.232 (1), p.37-47

2006

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Ubiquitous expression of CUG or CAG trinucleotide repeat RNA causes common morphological defects in a Drosophila model of RNA-mediated pathology

PloS one, 2012-06, Vol.7 (6), p.e38516

2012

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Neurodegenerative diseases have genetic hallmarks of autoinflammatory disease

Human molecular genetics, 2018-08, Vol.27 (R2), p.R108-R118

2018

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Contribution of mGluR and Fmr1 functional pathways to neurite morphogenesis, craniofacial development and fragile X syndrome

Human molecular genetics, 2006-12, Vol.15 (23), p.3446-3458

2006

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The pathogenic agent in Drosophila models of ‘polyglutamine’ diseases

Human molecular genetics, 2005-04, Vol.14 (8), p.1041-1048

2005

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WWOX, the chromosomal fragile site FRA16D spanning gene: Its role in metabolism and contribution to cancer

Experimental biology and medicine (Maywood, N.J.), 2015-03, Vol.240 (3), p.338-344

2015

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Non-self mutation: double-stranded RNA elicits antiviral pathogenic response in a Drosophila model of expanded CAG repeat neurodegenerative diseases

Human molecular genetics, 2019-09, Vol.28 (18), p.3000-3012

2019

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Common chromosomal fragile site FRA16D tumor suppressor WWOX gene expression and metabolic reprograming in cells

Genes chromosomes & cancer, 2013-09, Vol.52 (9), p.823-831

2013

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Huntingtin-deficient zebrafish exhibit defects in iron utilization and development

Human molecular genetics, 2007-08, Vol.16 (16), p.1905-1920

2007

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Fragile and unstable chromosomes in cancer: causes and consequences

Trends in Genetics, 2001-06, Vol.17 (6), p.339-345

Richards, Robert I.

2001

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Human Chromosomal Fragile Site FRA16B Is an Amplified AT-Rich Minisatellite Repeat

Cell, 1997-02, Vol.88 (3), p.367-374

1997

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Neuronal-specific impairment of heparan sulfate degradation in Drosophila reveals pathogenic mechanisms for Mucopolysaccharidosis type IIIA

Experimental neurology, 2018-05, Vol.303, p.38-47

2018

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Zebrafish Chromosome 14 Gene Differential Expression in the fmr1hu2787 Model of Fragile X Syndrome

Frontiers in genetics, 2021-05, Vol.12

2021

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Drosophila orthologue of WWOX, the chromosomal fragile site FRA16D tumour suppressor gene, functions in aerobic metabolism and regulates reactive oxygen species

Human molecular genetics, 2011-02, Vol.20 (3), p.497-509

2011

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Distinct roles for Toll and autophagy pathways in double-stranded RNA toxicity in a Drosophila model of expanded repeat neurodegenerative diseases

Human molecular genetics, 2013-07, Vol.22 (14), p.2811-2819

2013

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Double-stranded RNA is pathogenic in Drosophila models of expanded repeat neurodegenerative diseases

Human molecular genetics, 2011-10, Vol.20 (19), p.3757-3768

2011

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Selective neuronal requirement for huntingtin in the developing zebrafish

Human molecular genetics, 2009-12, Vol.18 (24), p.4830-4842

2009

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Dynamic mutations : a decade of unstable expanded repeats in human genetic disease

Human molecular genetics, 2001-10, Vol.10 (20), p.2187-2194

RICHARDS, Robert I

2001

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Common chromosomal fragile site FRA16D sequence : identification of the FOR gene spanning FRA16D and homozygous deletions and translocation breakpoints in cancer cells

Human molecular genetics, 2000-07, Vol.9 (11), p.1651-1663

2000

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Tumor Suppressor WWOX Contributes to the Elimination of Tumorigenic Cells in Drosophila melanogaster

Molecular Biology of the WWOX Gene That Spans Chromosomal Fragile Site FRA16D

The Enemy within: Innate Surveillance-Mediated Cell Death, the Common Mechanism of Neurodegenerative Disease

Tumor suppressor WWOX moderates the mitochondrial respiratory complex

Single-cell sequencing shows mosaic aneuploidy in most human embryos

Comparative toxicity of polyglutamine, polyalanine and polyleucine tracts in Drosophila models of expanded repeat disease

Common chromosomal fragile sites and cancer: Focus on FRA16D

Ubiquitous expression of CUG or CAG trinucleotide repeat RNA causes common morphological defects in a Drosophila model of RNA-mediated pathology

Neurodegenerative diseases have genetic hallmarks of autoinflammatory disease

Contribution of mGluR and Fmr1 functional pathways to neurite morphogenesis, craniofacial development and fragile X syndrome

The pathogenic agent in Drosophila models of ‘polyglutamine’ diseases

WWOX, the chromosomal fragile site FRA16D spanning gene: Its role in metabolism and contribution to cancer

Non-self mutation: double-stranded RNA elicits antiviral pathogenic response in a Drosophila model of expanded CAG repeat neurodegenerative diseases

Common chromosomal fragile site FRA16D tumor suppressor WWOX gene expression and metabolic reprograming in cells

Huntingtin-deficient zebrafish exhibit defects in iron utilization and development

Fragile and unstable chromosomes in cancer: causes and consequences

Human Chromosomal Fragile Site FRA16B Is an Amplified AT-Rich Minisatellite Repeat

Neuronal-specific impairment of heparan sulfate degradation in Drosophila reveals pathogenic mechanisms for Mucopolysaccharidosis type IIIA

Zebrafish Chromosome 14 Gene Differential Expression in the fmr1hu2787 Model of Fragile X Syndrome

Drosophila orthologue of WWOX, the chromosomal fragile site FRA16D tumour suppressor gene, functions in aerobic metabolism and regulates reactive oxygen species

Distinct roles for Toll and autophagy pathways in double-stranded RNA toxicity in a Drosophila model of expanded repeat neurodegenerative diseases

Double-stranded RNA is pathogenic in Drosophila models of expanded repeat neurodegenerative diseases

Selective neuronal requirement for huntingtin in the developing zebrafish

Dynamic mutations : a decade of unstable expanded repeats in human genetic disease

Common chromosomal fragile site FRA16D sequence : identification of the FOR gene spanning FRA16D and homozygous deletions and translocation breakpoints in cancer cells

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