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An official American Thoracic Society Clinical Practice guideline: the diagnosis of intensive care unit-acquired weakness in adults

American journal of respiratory and critical care medicine, 2014-12, Vol.190 (12), p.1437-1446

2014

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The neuromuscular junction is a focal point of mTORC1 signaling in sarcopenia

Nature communications, 2020-09, Vol.11 (1), p.4510-21, Article 4510

2020

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Impaired Synaptic Vesicle Release and Immaturity of Neuromuscular Junctions in Spinal Muscular Atrophy Mice

The Journal of neuroscience, 2009-01, Vol.29 (3), p.842-851

2009

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Mechanosensory encoding in ex vivo muscle–nerve preparations

Experimental physiology, 2024-01, Vol.109 (1), p.35-44

2024

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A Novel Path to Chronic Proprioceptive Disability with Oxaliplatin: Distortion of Sensory Encoding

Neurobiology of disease, 2016-11, Vol.95, p.54-65

2016

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Reduced motor neuron excitability is an important contributor to weakness in a rat model of sepsis

Experimental neurology, 2016-08, Vol.282, p.1-8

2016

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The role of action potential changes in depolarization-induced failure of excitation contraction coupling in mouse skeletal muscle

eLife, 2022-01, Vol.11

2022

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The mechanism underlying transient weakness in myotonia congenita

eLife, 2021-04, Vol.10

2021

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Inactivation of sodium channels underlies reversible neuropathy during critical illness in rats

The Journal of clinical investigation, 2009-05, Vol.119 (5), p.1150-1158

2009

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Crucial Role of Sodium Channel Fast Inactivation in Muscle Fibre Inexcitability in a Rat Model of Critical Illness Myopathy

The Journal of physiology, 2003-03, Vol.547 (2), p.555-566

2003

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Acetylcholine receptor subunit expression in Huntington's disease mouse muscle

Biochemistry and biophysics reports, 2021-12, Vol.28, p.101182-101182, Article 101182

2021

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Compensatory changes in cellular excitability, not synaptic scaling, contribute to homeostatic recovery of embryonic network activity

Proceedings of the National Academy of Sciences - PNAS, 2009-04, Vol.106 (16), p.6760-6765

2009

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Homeostatic synaptic plasticity at the neuromuscular junction in myasthenia gravis

Annals of the New York Academy of Sciences, 2018-01, Vol.1412 (1), p.170-177

2018

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Reversible Recruitment of a Homeostatic Reserve Pool of Synaptic Vesicles Underlies Rapid Homeostatic Plasticity of Quantal Content

The Journal of neuroscience, 2016-01, Vol.36 (3), p.828-836

2016

Volltextzugriff (PDF)

Erratum to “A novel path to chronic proprioceptive disability with oxaliplatin: Distortion of sensory encoding” [Neurobiol. Dis. 95 (2016) 54–65]

Neurobiology of disease, 2017-02, Vol.98, p.158-158

2017

Volltextzugriff (PDF)

Diverging from the Norm: Reevaluating What Miniature Excitatory Postsynaptic Currents Tell Us about Homeostatic Synaptic Plasticity

The Neuroscientist, 2024-02, Vol.30 (1), p.49-70

2024

Volltextzugriff (PDF)

Plateau potentials contribute to myotonia in mouse models of myotonia congenita

Experimental neurology, 2023-03, Vol.361, p.114303-114303, Article 114303

2023

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Central Role of Subthreshold Currents in Myotonia

Annals of neurology, 2020-02, Vol.87 (2), p.175-183

2020

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Rescue of the spinal muscular atrophy phenotype in a mouse model by early postnatal delivery of SMN

Nature biotechnology, 2010-03, Vol.28 (3), p.271-274

2010

Volltextzugriff (PDF)

Neuromuscular junction transmission failure is a late phenotype in aging mice

Neurobiology of aging, 2020-02, Vol.86, p.182-190

2020

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Plastin 3 Expression Does Not Modify Spinal Muscular Atrophy Severity in the ∆7 SMA Mouse

PloS one, 2015, Vol.10 (7), p.e0132364-e0132364

2015

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Rabbit supraspinatus motor endplates are unaffected by a rotator cuff tear

Journal of orthopaedic research, 2013-01, Vol.31 (1), p.99-104

2013

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Muscle Nicotinic Acetylcholine Receptors May Mediate Trans-Synaptic Signaling at the Mouse Neuromuscular Junction

The Journal of neuroscience, 2018-02, Vol.38 (7), p.1725-1736

2018

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Increasing motor neuron excitability to treat weakness in sepsis

Annals of neurology, 2017-12, Vol.82 (6), p.961-971

2017

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Chronic defects in intraspinal mechanisms of spike encoding by spinal motoneurons following chemotherapy

Experimental neurology, 2020-09, Vol.331, p.113354-113354, Article 113354

2020

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An official American Thoracic Society Clinical Practice guideline: the diagnosis of intensive care unit-acquired weakness in adults

The neuromuscular junction is a focal point of mTORC1 signaling in sarcopenia

Impaired Synaptic Vesicle Release and Immaturity of Neuromuscular Junctions in Spinal Muscular Atrophy Mice

Mechanosensory encoding in ex vivo muscle–nerve preparations

A Novel Path to Chronic Proprioceptive Disability with Oxaliplatin: Distortion of Sensory Encoding

Reduced motor neuron excitability is an important contributor to weakness in a rat model of sepsis

The role of action potential changes in depolarization-induced failure of excitation contraction coupling in mouse skeletal muscle

The mechanism underlying transient weakness in myotonia congenita

Inactivation of sodium channels underlies reversible neuropathy during critical illness in rats

Crucial Role of Sodium Channel Fast Inactivation in Muscle Fibre Inexcitability in a Rat Model of Critical Illness Myopathy

Acetylcholine receptor subunit expression in Huntington's disease mouse muscle

Compensatory changes in cellular excitability, not synaptic scaling, contribute to homeostatic recovery of embryonic network activity

Homeostatic synaptic plasticity at the neuromuscular junction in myasthenia gravis

Reversible Recruitment of a Homeostatic Reserve Pool of Synaptic Vesicles Underlies Rapid Homeostatic Plasticity of Quantal Content

Erratum to “A novel path to chronic proprioceptive disability with oxaliplatin: Distortion of sensory encoding” [Neurobiol. Dis. 95 (2016) 54–65]

Diverging from the Norm: Reevaluating What Miniature Excitatory Postsynaptic Currents Tell Us about Homeostatic Synaptic Plasticity

Plateau potentials contribute to myotonia in mouse models of myotonia congenita

Central Role of Subthreshold Currents in Myotonia

Rescue of the spinal muscular atrophy phenotype in a mouse model by early postnatal delivery of SMN

Neuromuscular junction transmission failure is a late phenotype in aging mice

Plastin 3 Expression Does Not Modify Spinal Muscular Atrophy Severity in the ∆7 SMA Mouse

Rabbit supraspinatus motor endplates are unaffected by a rotator cuff tear

Muscle Nicotinic Acetylcholine Receptors May Mediate Trans-Synaptic Signaling at the Mouse Neuromuscular Junction

Increasing motor neuron excitability to treat weakness in sepsis

Chronic defects in intraspinal mechanisms of spike encoding by spinal motoneurons following chemotherapy

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