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Regulation of the protein C anticoagulant and antiinflammatory pathways

Current medicinal chemistry, 2010-07, Vol.17 (19), p.2059-2069

Rezaie, A R

2010

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Polyphosphate elicits pro‐inflammatory responses that are counteracted by activated protein C in both cellular and animal models

Journal of thrombosis and haemostasis, 2012-06, Vol.10 (6), p.1145-1151

2012

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Inorganic polyphosphate promotes cyclin D1 synthesis through activation of mTOR/Wnt/β‐catenin signaling in endothelial cells

Journal of thrombosis and haemostasis, 2016-11, Vol.14 (11), p.2261-2273

2016

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Inorganic polyphosphate elicits pro‐inflammatory responses through activation of the mammalian target of rapamycin complexes 1 and 2 in vascular endothelial cells

Journal of thrombosis and haemostasis, 2015-05, Vol.13 (5), p.860-871

2015

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Activated protein C protects against myocardial ischemic/reperfusion injury through AMP‐activated protein kinase signaling

Journal of thrombosis and haemostasis, 2011-07, Vol.9 (7), p.1308-1317

2011

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Antithrombin is protective against myocardial ischemia and reperfusion injury

Journal of thrombosis and haemostasis, 2013-06, Vol.11 (6), p.1020-1028

2013

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Expression and functional characterization of two natural heparin‐binding site variants of antithrombin

Journal of thrombosis and haemostasis, 2018-02, Vol.16 (2), p.330-341

2018

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Calcium enhances heparin catalysis of the antithrombin-factor Xa reaction by a template mechanism. Evidence that calcium alleviates Gla domain antagonism of heparin binding to factor Xa

The Journal of biological chemistry, 1998-07, Vol.273 (27), p.16824-16827

Rezaie, A R

1998

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Pentasaccharide Enhances the Inactivation of Factor Xa by Antithrombin by Promoting the Assembly of a Michaelis-Type Intermediate Complex. Demonstration by Rapid Kinetic, Surface Plasmon Resonance, and Competitive Binding Studies

Biochemistry (Easton), 2006-04, Vol.45 (16), p.5324-5329

Rezaie, Alireza R

2006

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Activated protein C modulates cardiac metabolism and augments autophagy in the ischemic heart

Journal of thrombosis and haemostasis, 2012-09, Vol.10 (9), p.1736-1744

2012

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Expression and functional characterisation of natural R147W and K150del variants of protein C in the Chinese population

Thrombosis and haemostasis, 2013-04, Vol.109 (4), p.614-624

2013

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Lipid raft localization regulates the cleavage specificity of protease activated receptor 1 in endothelial cells

Journal of thrombosis and haemostasis, 2008-06, Vol.6 (6), p.954-961

2008

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Thrombin upregulates the angiopoietin–Tie2 Axis: endothelial protein C receptor occupancy prevents the thrombin mobilization of angiopoietin 2 and P‐selectin from Weibel–Palade bodies

Journal of thrombosis and haemostasis, 2010-05, Vol.8 (5), p.1107-1115

2010

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Calcium Enhances Heparin Catalysis of the Antithrombin−Factor Xa Reaction by Promoting the Assembly of an Intermediate Heparin−Antithrombin−Factor Xa Bridging Complex. Demonstration by Rapid Kinetics Studies

Biochemistry (Easton), 2000-10, Vol.39 (39), p.12083-12090

2000

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Vitronectin functions as a cofactor for rapid inhibition of activated protein C by plasminogen activator inhibitor-1. Implications for the mechanism of profibrinolytic action of activated protein C

The Journal of biological chemistry, 2001-05, Vol.276 (19), p.15567

Rezaie, A R

2001

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The function of calcium in protein C activation by thrombin and the thrombin-thrombomodulin complex can be distinguished by mutational analysis of protein C derivatives

The Journal of biological chemistry, 1992-12, Vol.267 (36), p.26104-26109

1992

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Contribution of exosite occupancy by heparin to the regulation of coagulation proteases by antithrombin

Thrombosis and haemostasis, 2010-02, Vol.103 (2), p.277-283

2010

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Mutagenesis studies toward understanding the intracellular signaling mechanism of antithrombin

Journal of thrombosis and haemostasis, 2009-05, Vol.7 (5), p.803-810

2009

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Functional role of O‐linked and N‐linked glycosylation sites present on the activation peptide of factor X

Journal of thrombosis and haemostasis, 2009-10, Vol.7 (10), p.1696-1702

2009

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Prothrombin protects factor Xa in the prothrombinase complex from inhibition by the heparin-antithrombin complex

Blood, 2001-04, Vol.97 (8), p.2308-2313

Rezaie, Alireza R.

2001

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Probing the molecular basis of factor Xa specificity by mutagenesis of the serpin, antithrombin

Biochimica et biophysica acta, 2001-10, Vol.1528 (2), p.167-176

2001

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Identification of Basic Residues in the Heparin-binding Exosite of Factor Xa Critical for Heparin and Factor Va Binding

The Journal of biological chemistry, 2000-02, Vol.275 (5), p.3320-3327

Rezaie, Alireza R.

2000

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Reactivities of the S2 and S3 subsite residues of thrombin with the native and heparin‐induced conformers of antithrombin

Protein science, 1998-02, Vol.7 (2), p.349-357

Rezaie, Alireza R.

1998

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Mutation of Glu-80-->Lys results in a protein C mutant that no longer requires Ca2+ for rapid activation by the thrombin-thrombomodulin complex

The Journal of biological chemistry, 1994-02, Vol.269 (5), p.3151-3154

1994

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Conversion of glutamic acid 192 to glutamine in activated protein C changes the substrate specificity and increases reactivity toward macromolecular inhibitors

The Journal of biological chemistry, 1993-09, Vol.268 (27), p.19943-19948

1993

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Regulation of the protein C anticoagulant and antiinflammatory pathways

Polyphosphate elicits pro‐inflammatory responses that are counteracted by activated protein C in both cellular and animal models

Inorganic polyphosphate promotes cyclin D1 synthesis through activation of mTOR/Wnt/β‐catenin signaling in endothelial cells

Inorganic polyphosphate elicits pro‐inflammatory responses through activation of the mammalian target of rapamycin complexes 1 and 2 in vascular endothelial cells

Activated protein C protects against myocardial ischemic/reperfusion injury through AMP‐activated protein kinase signaling

Antithrombin is protective against myocardial ischemia and reperfusion injury

Expression and functional characterization of two natural heparin‐binding site variants of antithrombin

Calcium enhances heparin catalysis of the antithrombin-factor Xa reaction by a template mechanism. Evidence that calcium alleviates Gla domain antagonism of heparin binding to factor Xa

Pentasaccharide Enhances the Inactivation of Factor Xa by Antithrombin by Promoting the Assembly of a Michaelis-Type Intermediate Complex. Demonstration by Rapid Kinetic, Surface Plasmon Resonance, and Competitive Binding Studies

Activated protein C modulates cardiac metabolism and augments autophagy in the ischemic heart

Expression and functional characterisation of natural R147W and K150del variants of protein C in the Chinese population

Lipid raft localization regulates the cleavage specificity of protease activated receptor 1 in endothelial cells

Thrombin upregulates the angiopoietin–Tie2 Axis: endothelial protein C receptor occupancy prevents the thrombin mobilization of angiopoietin 2 and P‐selectin from Weibel–Palade bodies

Calcium Enhances Heparin Catalysis of the Antithrombin−Factor Xa Reaction by Promoting the Assembly of an Intermediate Heparin−Antithrombin−Factor Xa Bridging Complex. Demonstration by Rapid Kinetics Studies

Vitronectin functions as a cofactor for rapid inhibition of activated protein C by plasminogen activator inhibitor-1. Implications for the mechanism of profibrinolytic action of activated protein C

The function of calcium in protein C activation by thrombin and the thrombin-thrombomodulin complex can be distinguished by mutational analysis of protein C derivatives

Contribution of exosite occupancy by heparin to the regulation of coagulation proteases by antithrombin

Mutagenesis studies toward understanding the intracellular signaling mechanism of antithrombin

Functional role of O‐linked and N‐linked glycosylation sites present on the activation peptide of factor X

Prothrombin protects factor Xa in the prothrombinase complex from inhibition by the heparin-antithrombin complex

Probing the molecular basis of factor Xa specificity by mutagenesis of the serpin, antithrombin

Identification of Basic Residues in the Heparin-binding Exosite of Factor Xa Critical for Heparin and Factor Va Binding

Reactivities of the S2 and S3 subsite residues of thrombin with the native and heparin‐induced conformers of antithrombin

Mutation of Glu-80-->Lys results in a protein C mutant that no longer requires Ca2+ for rapid activation by the thrombin-thrombomodulin complex

Conversion of glutamic acid 192 to glutamine in activated protein C changes the substrate specificity and increases reactivity toward macromolecular inhibitors

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