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Clustered Mutations in Yeast and in Human Cancers Can Arise from Damaged Long Single-Strand DNA Regions

Molecular cell, 2012-05, Vol.46 (4), p.424-435

2012

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The Human TLR Innate Immune Gene Family Is Differentially Influenced by DNA Stress and p53 Status in Cancer Cells

Cancer research (Chicago, Ill.), 2012-08, Vol.72 (16), p.3948-3957

2012

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Break-Induced Replication Is a Source of Mutation Clusters Underlying Kataegis

Cell reports (Cambridge), 2014-06, Vol.7 (5), p.1640-1648

2014

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Interactions between the tumor suppressor p53 and immune responses

Current opinion in oncology, 2013-01, Vol.25 (1), p.85-92

2013

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The Toll-like receptor gene family is integrated into human DNA damage and p53 networks

PLoS genetics, 2011-03, Vol.7 (3), p.e1001360

2011

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Diverse stresses dramatically alter genome-wide p53 binding and transactivation landscape in human cancer cells

Nucleic acids research, 2013-08, Vol.41 (15), p.7286

2013

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ETV7-Mediated DNAJC15 Repression Leads to Doxorubicin Resistance in Breast Cancer Cells

Neoplasia (New York, N.Y.), 2018-08, Vol.20 (8), p.857-870

2018

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Double-Strand Breaks Associated with Repetitive DNA Can Reshape the Genome

Proceedings of the National Academy of Sciences - PNAS, 2008-08, Vol.105 (33), p.11845-11850

2008

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Hypermutability of damaged single-strand DNA formed at double-strand breaks and uncapped telomeres in yeast Saccharomyces cerevisiae

PLoS genetics, 2008-11, Vol.4 (11), p.e1000264-e1000264

2008

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The Cytidine Deaminase APOBEC3 Family Is Subject to Transcriptional Regulation by p53

Molecular cancer research, 2017-06, Vol.15 (6), p.735-743

2017

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APOBEC3G Is a p53-Dependent Restriction Factor in Respiratory Syncytial Virus Infection of Human Cells Included in the p53/Immune Axis

International journal of molecular sciences, 2023-12, Vol.24 (23), p.16793

2023

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Coincident resection at both ends of random, γ-induced double-strand breaks requires MRX (MRN), Sae2 (Ctp1), and Mre11-nuclease

PLoS genetics, 2013-03, Vol.9 (3), p.e1003420

2013

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Mutant TP53 Posttranslational Modifications: Challenges and Opportunities

Human mutation, 2014-06, Vol.35 (6), p.738-755

2014

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Cohesin Is limiting for the suppression of DNA damage-induced recombination between homologous chromosomes

PLoS genetics, 2010-07, Vol.6 (7), p.e1001006

2010

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Base damage within single-strand DNA underlies in vivo hypermutability induced by a ubiquitous environmental agent

PLoS genetics, 2012-12, Vol.8 (12), p.e1003149

2012

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The expanding universe of p53 targets

Nature reviews. Cancer, 2009-10, Vol.9 (10), p.724-737

2009

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Flexibility of Eukaryotic Okazaki Fragment Maturation through Regulated Strand Displacement Synthesis

The Journal of biological chemistry, 2008-12, Vol.283 (49), p.34129-34140

2008

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Homologous recombination rescues ssDNA gaps generated by nucleotide excision repair and reduced translesion DNA synthesis in yeast G2 cells

Proceedings of the National Academy of Sciences - PNAS, 2013-07, Vol.110 (31), p.E2895-E2904

2013

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Estrogen Receptor Acting in Cis Enhances WT and Mutant p53 Transactivation at Canonical and Noncanonical p53 Target Sequences

Proceedings of the National Academy of Sciences - PNAS, 2010-01, Vol.107 (4), p.1500-1505

2010

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Functional Mutants of the Sequence-Specific Transcription Factor p53 and Implications for Master Genes of Diversity

Proceedings of the National Academy of Sciences - PNAS, 2003-08, Vol.100 (17), p.9934-9939

2003

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The sister chromatid cohesion pathway suppresses multiple chromosome gain and chromosome amplification

Genetics (Austin), 2014-02, Vol.196 (2), p.373-384

2014

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In vivo site-directed mutagenesis using oligonucleotides

Nature biotechnology, 2001-08, Vol.19 (8), p.773-776

2001

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Interaction between p53 and estradiol pathways in transcriptional responses to chemotherapeutics

Cell cycle (Georgetown, Tex.), 2013-04, Vol.12 (8), p.1211-1224

2013

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Quantitative Analysis of NF-κB Transactivation Specificity Using a Yeast-Based Functional Assay

PloS one, 2015-07, Vol.10 (7), p.e0130170-e0130170

2015

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Noncanonical DNA motifs as transactivation targets by wild type and mutant p53

PLoS genetics, 2008-06, Vol.4 (6), p.e1000104

2008

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Clustered Mutations in Yeast and in Human Cancers Can Arise from Damaged Long Single-Strand DNA Regions

The Human TLR Innate Immune Gene Family Is Differentially Influenced by DNA Stress and p53 Status in Cancer Cells

Break-Induced Replication Is a Source of Mutation Clusters Underlying Kataegis

Interactions between the tumor suppressor p53 and immune responses

The Toll-like receptor gene family is integrated into human DNA damage and p53 networks

Diverse stresses dramatically alter genome-wide p53 binding and transactivation landscape in human cancer cells

ETV7-Mediated DNAJC15 Repression Leads to Doxorubicin Resistance in Breast Cancer Cells

Double-Strand Breaks Associated with Repetitive DNA Can Reshape the Genome

Hypermutability of damaged single-strand DNA formed at double-strand breaks and uncapped telomeres in yeast Saccharomyces cerevisiae

The Cytidine Deaminase APOBEC3 Family Is Subject to Transcriptional Regulation by p53

APOBEC3G Is a p53-Dependent Restriction Factor in Respiratory Syncytial Virus Infection of Human Cells Included in the p53/Immune Axis

Coincident resection at both ends of random, γ-induced double-strand breaks requires MRX (MRN), Sae2 (Ctp1), and Mre11-nuclease

Mutant TP53 Posttranslational Modifications: Challenges and Opportunities

Cohesin Is limiting for the suppression of DNA damage-induced recombination between homologous chromosomes

Base damage within single-strand DNA underlies in vivo hypermutability induced by a ubiquitous environmental agent

The expanding universe of p53 targets

Flexibility of Eukaryotic Okazaki Fragment Maturation through Regulated Strand Displacement Synthesis

Homologous recombination rescues ssDNA gaps generated by nucleotide excision repair and reduced translesion DNA synthesis in yeast G2 cells

Estrogen Receptor Acting in Cis Enhances WT and Mutant p53 Transactivation at Canonical and Noncanonical p53 Target Sequences

Functional Mutants of the Sequence-Specific Transcription Factor p53 and Implications for Master Genes of Diversity

The sister chromatid cohesion pathway suppresses multiple chromosome gain and chromosome amplification

In vivo site-directed mutagenesis using oligonucleotides

Interaction between p53 and estradiol pathways in transcriptional responses to chemotherapeutics

Quantitative Analysis of NF-κB Transactivation Specificity Using a Yeast-Based Functional Assay

Noncanonical DNA motifs as transactivation targets by wild type and mutant p53

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