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Reep1 null mice reveal a converging role for hereditary spastic paraplegia proteins in lipid droplet regulation

Human molecular genetics, 2016-12, Vol.25 (23), p.5111-5125

2016

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Spg20―/― mice reveal multimodal functions for Troyer syndrome protein spartin in lipid droplet maintenance, cytokinesis and BMP signaling

Human molecular genetics, 2012-08, Vol.21 (16), p.3604-3618

2012

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Emerging themes of ER organization in the development and maintenance of axons

Current opinion in neurobiology, 2010-10, Vol.20 (5), p.531-537

2010

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Structural basis for midbody targeting of spastin by the ESCRT-III protein CHMP1B

Nature structural & molecular biology, 2008-12, Vol.15 (12), p.1278-1286

2008

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Transverse endoplasmic reticulum expansion in hereditary spastic paraplegia corticospinal axons

Human molecular genetics, 2022-08, Vol.31 (16), p.2779-2795

2022

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Mammalian knock out cells reveal prominent roles for atlastin GTPases in ER network morphology

Experimental cell research, 2016-11, Vol.349 (1), p.32-44

2016

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Spastin-interacting protein NA14/SSNA1 functions in cytokinesis and axon development

PloS one, 2014-11, Vol.9 (11), p.e112428-e112428

2014

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MITD1 is recruited to midbodies by ESCRT-III and participates in cytokinesis

Molecular biology of the cell, 2012-11, Vol.23 (22), p.4347-4361

2012

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SPG20 protein spartin is recruited to midbodies by ESCRT-III protein Ist1 and participates in cytokinesis

Molecular biology of the cell, 2010-10, Vol.21 (19), p.3293-3303

2010

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Mutation in CPT1C Associated With Pure Autosomal Dominant Spastic Paraplegia

JAMA neurology, 2015-05, Vol.72 (5), p.561

2015

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A role for protein phosphatase PP1γ in SMN complex formation and subnuclear localization to Cajal bodies

Journal of cell science, 2012-06, Vol.125 (Pt 12), p.2862-2874

2012

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A new glucocerebrosidase-deficient neuronal cell model provides a tool to probe pathophysiology and therapeutics for Gaucher disease

Disease models & mechanisms, 2016-07, Vol.9 (7), p.769-778

2016

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Lysosomal abnormalities in hereditary spastic paraplegia types SPG15 and SPG11

Annals of clinical and translational neurology, 2014-06, Vol.1 (6), p.379-389

2014

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The loss of the snoRNP chaperone Nopp140 from Cajal bodies of patient fibroblasts correlates with the severity of spinal muscular atrophy

Human molecular genetics, 2009-04, Vol.18 (7), p.1181-1189

2009

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Distinct domains of the spinal muscular atrophy protein SMN are required for targeting to Cajal bodies in mammalian cells

Journal of cell science, 2006-02, Vol.119 (4), p.680-692

2006

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Neuronal Cx3cr1 Deficiency Protects against Amyloid β-Induced Neurotoxicity

PloS one, 2015-06, Vol.10 (6), p.e0127730

2015

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Structural basis for midbody targeting of spastin by the ESCRT‐III protein CHMP1B

The FASEB journal, 2009-04, Vol.23 (S1)

2009

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Neuronal Cx3cr1 Deficiency Protects against Amyloid beta -Induced Neurotoxicity: e0127730

PloS one, 2015-06, Vol.10 (6)

2015

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Neuronal Cx3cr1 Deficiency Protects against Amyloid [beta]-Induced Neurotoxicity

PloS one, 2015-06, Vol.10 (6)

2015

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Hereditary spastic paraplegias types 15 and 11 are associated with lysosomal abnormalities

Molecular genetics and metabolism, 2014-02, Vol.111 (2), p.S86-S86

2014

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Lysosomal abnormalities in hereditary spastic paraplegia types SPG 15 and SPG 11

Annals of clinical and translational neurology, 2014-06, Vol.1 (6), p.379-389

2014

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Chapter 69 - Hereditary Spastic Paraplegias: Genetics and Clinical Features

Movement Disorders, 2015, p.1063-1071

Second Edition, 2015

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Microvascular injuries, secondary edema, and inconsistencies in lung vascularization between affected and nonaffected pulmonary segments of non-critically ill hospitalized COVID-19 patients presenting with clinical deterioration

Therapeutic advances in respiratory disease, 2022, Vol.16, p.17534666221096040-17534666221096040

2022

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Quantitative variations of the mitochondrial proteome and phosphoproteome during fermentative and respiratory growth in Saccharomyces cerevisiae

Journal of proteomics, 2014-06, Vol.106, p.140-150

2014

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A surge of MDR and XDR tuberculosis in France among patients born in the Former Soviet Union

Euro surveillance : bulletin européen sur les maladies transmissibles, 2013-08, Vol.18 (33), p.20555-20555

2013

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Reep1 null mice reveal a converging role for hereditary spastic paraplegia proteins in lipid droplet regulation

Spg20―/― mice reveal multimodal functions for Troyer syndrome protein spartin in lipid droplet maintenance, cytokinesis and BMP signaling

Emerging themes of ER organization in the development and maintenance of axons

Structural basis for midbody targeting of spastin by the ESCRT-III protein CHMP1B

Transverse endoplasmic reticulum expansion in hereditary spastic paraplegia corticospinal axons

Mammalian knock out cells reveal prominent roles for atlastin GTPases in ER network morphology

Spastin-interacting protein NA14/SSNA1 functions in cytokinesis and axon development

MITD1 is recruited to midbodies by ESCRT-III and participates in cytokinesis

SPG20 protein spartin is recruited to midbodies by ESCRT-III protein Ist1 and participates in cytokinesis

Mutation in CPT1C Associated With Pure Autosomal Dominant Spastic Paraplegia

A role for protein phosphatase PP1γ in SMN complex formation and subnuclear localization to Cajal bodies

A new glucocerebrosidase-deficient neuronal cell model provides a tool to probe pathophysiology and therapeutics for Gaucher disease

Lysosomal abnormalities in hereditary spastic paraplegia types SPG15 and SPG11

The loss of the snoRNP chaperone Nopp140 from Cajal bodies of patient fibroblasts correlates with the severity of spinal muscular atrophy

Distinct domains of the spinal muscular atrophy protein SMN are required for targeting to Cajal bodies in mammalian cells

Neuronal Cx3cr1 Deficiency Protects against Amyloid β-Induced Neurotoxicity

Structural basis for midbody targeting of spastin by the ESCRT‐III protein CHMP1B

Neuronal Cx3cr1 Deficiency Protects against Amyloid beta -Induced Neurotoxicity: e0127730

Neuronal Cx3cr1 Deficiency Protects against Amyloid [beta]-Induced Neurotoxicity

Hereditary spastic paraplegias types 15 and 11 are associated with lysosomal abnormalities

Lysosomal abnormalities in hereditary spastic paraplegia types SPG 15 and SPG 11

Chapter 69 - Hereditary Spastic Paraplegias: Genetics and Clinical Features

Microvascular injuries, secondary edema, and inconsistencies in lung vascularization between affected and nonaffected pulmonary segments of non-critically ill hospitalized COVID-19 patients presenting with clinical deterioration

Quantitative variations of the mitochondrial proteome and phosphoproteome during fermentative and respiratory growth in Saccharomyces cerevisiae

A surge of MDR and XDR tuberculosis in France among patients born in the Former Soviet Union

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