UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

The Anti-DNA Antibodies: Their Specificities for Unique DNA Structures and Their Unresolved Clinical Impact-A System Criticism and a Hypothesis

Frontiers in immunology, 2022-01, Vol.12, p.808008-808008

Rekvig, Ole Petter

2022

Volltextzugriff (PDF)

Autoimmunity and SLE: Factual and Semantic Evidence-Based Critical Analyses of Definitions, Etiology, and Pathogenesis

Frontiers in immunology, 2020-10, Vol.11, p.569234

Rekvig, Ole Petter

2020

Volltextzugriff (PDF)

The dsDNA, Anti-dsDNA Antibody, and Lupus Nephritis: What We Agree on, What Must Be Done, and What the Best Strategy Forward Could Be

Frontiers in immunology, 2019, Vol.10, p.1104-1104

Rekvig, Ole Petter

2019

Volltextzugriff (PDF)

SLE: a cognitive step forward-a synthesis of rethinking theories, causality, and ignored DNA structures

Frontiers in immunology, 2024-06, Vol.15, p.1393814

Rekvig, Ole Petter

2024

Volltextzugriff (PDF)

SLE classification criteria: Science-based icons or algorithmic distractions – an intellectually demanding dilemma

Frontiers in immunology, 2022-09, Vol.13, p.1011591-1011591

Rekvig, Ole Petter

2022

Volltextzugriff (PDF)

The greatest contribution to medical science is the transformation from studying symptoms to studying their causes-the unrelenting legacy of Robert Koch and Louis Pasteur-and a causality perspective to approach a definition of SLE

Frontiers in immunology, 2024-02, Vol.15, p.1346619-1346619

Rekvig, Ole Petter

2024

Volltextzugriff (PDF)

Systemic Lupus Erythematosus: Definitions, Contexts, Conflicts, Enigmas

Frontiers in immunology, 2018-03, Vol.9, p.387-387

Rekvig, Ole Petter

2018

Volltextzugriff (PDF)

TNFα Amplifies DNaseI Expression in Renal Tubular Cells while IL-1β Promotes Nuclear DNaseI Translocation in an Endonuclease-Inactive Form

PloS one, 2015-06, Vol.10 (6), p.e0129485

2015

Volltextzugriff (PDF)

Nephritogenic Potential of Anti-DNA Antibodies against Necrotic Nucleosomes

Journal of the American Society of Nephrology, 2009-04, Vol.20 (4), p.696-704

2009

Volltextzugriff (PDF)

Anti-dsDNA antibodies promote initiation, and acquired loss of renal Dnase1 promotes progression of lupus nephritis in autoimmune (NZBxNZW)F1 mice

PloS one, 2009-12, Vol.4 (12), p.e8474-e8474

2009

Volltextzugriff (PDF)

Alterations in Wnt pathway activity in mouse serum and kidneys during lupus development

Arthritis & rheumatology (Hoboken, N.J.), 2011-02, Vol.63 (2), p.513-522

2011

Volltextzugriff (PDF)

Renal Dnase1 enzyme activity and protein expression is selectively shut down in murine and human membranoproliferative lupus nephritis

PloS one, 2010-08, Vol.5 (8), p.e12096

2010

Volltextzugriff (PDF)

Lupus nephritis: enigmas, conflicting models and an emerging concept

Molecular medicine (Cambridge, Mass.), 2013, Vol.19 (1), p.161-169

2013

Volltextzugriff (PDF)

SLE classification criteria: Is “The causality principle” integrated and operative – and do the molecular and genetical network, on which criteria depend on, support the definition of SLE as “a one disease entity” – A theoretical discussion

Autoimmunity reviews, 2023-12, Vol.22 (12), p.103470, Article 103470

Rekvig, Ole Petter

2023

Volltextzugriff (PDF)

Murine and Human Lupus Nephritis: Pathogenic Mechanisms and Theoretical Strategies for Therapy

Seminars in nephrology, 2015-09, Vol.35 (5), p.427-438

2015

Volltextzugriff (PDF)

Lupus nephritis progression in FcγRIIB-/-yaa mice is associated with early development of glomerular electron dense deposits and loss of renal DNase I in severe disease

PloS one, 2017, Vol.12 (11), p.e0188863-e0188863

2017

Volltextzugriff (PDF)

B Cell Tolerance to Deiminated Histones in BALB/c, C57BL/6, and Autoimmune-Prone Mouse Strains

Frontiers in immunology, 2017-03, Vol.8, p.362-362

2017

Volltextzugriff (PDF)

Silencing of renal DNaseI in murine lupus nephritis imposes exposure of large chromatin fragments and activation of Toll like receptors and the Clec4e

PloS one, 2012-03, Vol.7 (3), p.e34080-e34080

2012

Volltextzugriff (PDF)

Heparin exerts a dual effect on murine lupus nephritis by enhancing enzymatic chromatin degradation and preventing chromatin binding in glomerular membranes

Arthritis & rheumatology (Hoboken, N.J.), 2011-04, Vol.63 (4), p.1065-1075

2011

Volltextzugriff (PDF)

Pure anti-dsDNA mAbs need chromatin structures to promote glomerular mesangial deposits in BALB/c mice

Autoimmunity (Chur, Switzerland), 2010-03, Vol.43 (2), p.179-188

2010

Volltextzugriff (PDF)

A Central Role of Nucleosomes in Lupus Nephritis

Annals of the New York Academy of Sciences, 2007-06, Vol.1108 (1), p.104-113

2007

Volltextzugriff (PDF)

LMW Heparin Prevents Increased Kidney Expression of Proinflammatory Mediators in (NZBxNZW)F1 Mice

Clinical & developmental immunology, 2013-01, Vol.2013 (2013), p.1-11

2013

Volltextzugriff (PDF)

Pathogenesis of SLE Dermatitis - A Reflection of the Process in SLE Nephritis?

Current rheumatology reviews, 2007-05, Vol.3 (2), p.159-165

2007

Volltextzugriff (PDF)

A framework for remission in SLE: consensus findings from a large international task force on definitions of remission in SLE (DORIS)

Annals of the rheumatic diseases, 2017-03, Vol.76 (3), p.554-561

2017

Volltextzugriff (PDF)

Autoimmunity to nucleosomes related to viral infection: a focus on hapten-carrier complex formation

Journal of autoimmunity, 2003-03, Vol.20 (2), p.171-182

2003

Volltextzugriff (PDF)

1 – 25 von 84

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(74)
Mit Volltextzugriff
(71)

Publikationsform

Sprache

Englisch
(82)
Norwegisch
(2)

Erscheinungsjahr

n.n

Thema

Science & Technology
(65)
Life Sciences & Biomedicine
(58)
Humans
(47)
Animals
(39)
Immunology
(29)
Mice
(27)
Systemic Lupus Erythematosus
(24)
Female
(22)
Antibodies, Antinuclear - Immunology
(21)
Lupus Nephritis
(20)
Rheumatology
(20)
Dna - Immunology
(18)
Biological And Medical Sciences
(17)
Lupus Erythematosus, Systemic - Immunology
(17)
Lupus Nephritis - Immunology
(16)
Medical Sciences
(15)
Vdp
(15)
Autoimmunity
(14)
Nucleosomes - Immunology
(14)
Medical Disciplines: 700
(13)

15 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 90 Tagen
(1)

Menü

Weitere Dienste

Einstellungen

The Anti-DNA Antibodies: Their Specificities for Unique DNA Structures and Their Unresolved Clinical Impact-A System Criticism and a Hypothesis

Autoimmunity and SLE: Factual and Semantic Evidence-Based Critical Analyses of Definitions, Etiology, and Pathogenesis

The dsDNA, Anti-dsDNA Antibody, and Lupus Nephritis: What We Agree on, What Must Be Done, and What the Best Strategy Forward Could Be

SLE: a cognitive step forward-a synthesis of rethinking theories, causality, and ignored DNA structures

SLE classification criteria: Science-based icons or algorithmic distractions – an intellectually demanding dilemma

The greatest contribution to medical science is the transformation from studying symptoms to studying their causes-the unrelenting legacy of Robert Koch and Louis Pasteur-and a causality perspective to approach a definition of SLE

Systemic Lupus Erythematosus: Definitions, Contexts, Conflicts, Enigmas

TNFα Amplifies DNaseI Expression in Renal Tubular Cells while IL-1β Promotes Nuclear DNaseI Translocation in an Endonuclease-Inactive Form

Nephritogenic Potential of Anti-DNA Antibodies against Necrotic Nucleosomes

Anti-dsDNA antibodies promote initiation, and acquired loss of renal Dnase1 promotes progression of lupus nephritis in autoimmune (NZBxNZW)F1 mice

Alterations in Wnt pathway activity in mouse serum and kidneys during lupus development

Renal Dnase1 enzyme activity and protein expression is selectively shut down in murine and human membranoproliferative lupus nephritis

Lupus nephritis: enigmas, conflicting models and an emerging concept

SLE classification criteria: Is “The causality principle” integrated and operative – and do the molecular and genetical network, on which criteria depend on, support the definition of SLE as “a one disease entity” – A theoretical discussion

Murine and Human Lupus Nephritis: Pathogenic Mechanisms and Theoretical Strategies for Therapy

Lupus nephritis progression in FcγRIIB-/-yaa mice is associated with early development of glomerular electron dense deposits and loss of renal DNase I in severe disease

B Cell Tolerance to Deiminated Histones in BALB/c, C57BL/6, and Autoimmune-Prone Mouse Strains

Silencing of renal DNaseI in murine lupus nephritis imposes exposure of large chromatin fragments and activation of Toll like receptors and the Clec4e

Heparin exerts a dual effect on murine lupus nephritis by enhancing enzymatic chromatin degradation and preventing chromatin binding in glomerular membranes

Pure anti-dsDNA mAbs need chromatin structures to promote glomerular mesangial deposits in BALB/c mice

A Central Role of Nucleosomes in Lupus Nephritis

LMW Heparin Prevents Increased Kidney Expression of Proinflammatory Mediators in (NZBxNZW)F1 Mice

Pathogenesis of SLE Dermatitis - A Reflection of the Process in SLE Nephritis?

A framework for remission in SLE: consensus findings from a large international task force on definitions of remission in SLE (DORIS)

Autoimmunity to nucleosomes related to viral infection: a focus on hapten-carrier complex formation

Suchergebnisse filtern