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Tolvaptan inhibits ERK-dependent cell proliferation, Cl⁻ secretion, and in vitro cyst growth of human ADPKD cells stimulated by vasopressin

American journal of physiology. Renal physiology, 2011-11, Vol.301 (5), p.F1005-F1013

2011

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Deficient transient receptor potential vanilloid type 4 function contributes to compromised [Ca 2+ ] i homeostasis in human autosomal-dominant polycystic kidney disease cells

The FASEB journal, 2018-08, Vol.32 (8), p.4612-4623

2018

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Calcium restores a normal proliferation phenotype in human polycystic kidney disease epithelial cells

Journal of the American Society of Nephrology, 2006, Vol.17 (1), p.178-187

2006

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Phosphodiesterase Isoform Regulation of Cell Proliferation and Fluid Secretion in Autosomal Dominant Polycystic Kidney Disease

Journal of the American Society of Nephrology, 2016-04, Vol.27 (4), p.1124-1134

2016

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Integrin-Linked Kinase Signaling Promotes Cyst Growth and Fibrosis in Polycystic Kidney Disease

Journal of the American Society of Nephrology, 2017-09, Vol.28 (9), p.2708-2719

2017

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Periostin overexpression in collecting ducts accelerates renal cyst growth and fibrosis in polycystic kidney disease

American journal of physiology. Renal physiology, 2018-12, Vol.315 (6), p.F1695-F1707

2018

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CaMK4 overexpression in polycystic kidney disease promotes mTOR-mediated cell proliferation

Journal of molecular cell biology, 2022-09, Vol.14 (7)

2022

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Expression of active B-Raf proto-oncogene in kidney collecting ducts induces cyst formation in normal mice and accelerates cyst growth in mice with polycystic kidney disease

Kidney international, 2022-11, Vol.102 (5), p.1103-1114

2022

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Periostin promotes renal cyst growth and interstitial fibrosis in polycystic kidney disease

Kidney international, 2014-04, Vol.85 (4), p.845-854

2014

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The lonidamine derivative H2-gamendazole reduces cyst formation in polycystic kidney disease

American journal of physiology. Renal physiology, 2022-10, Vol.323 (4), p.F492-F506

2022

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Sorafenib inhibits cAMP-dependent ERK activation, cell proliferation, and in vitro cyst growth of human ADPKD cyst epithelial cells

American Journal of Physiology - Renal Physiology, 2010-11, Vol.299 (5), p.F944-F951

2010

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Ouabain promotes partial epithelial to mesenchymal transition (EMT) changes in human autosomal dominant polycystic kidney disease (ADPKD) cells

Experimental cell research, 2017-06, Vol.355 (2), p.142-152

2017

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Endogenous concentrations of ouabain act as a cofactor to stimulate fluid secretion and cyst growth of in vitro ADPKD models via cAMP and EGFR-Src-MEK pathways

American journal of physiology. Renal physiology, 2012-10, Vol.303 (7), p.F982-F990

2012

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In vitro cyst formation of ADPKD cells

Methods in Kidney Cell Biology Part A, 2019, Vol.153, p.93-111

2019

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Calmodulin-sensitive adenylyl cyclases mediate AVP-dependent cAMP production and Cl- secretion by human autosomal dominant polycystic kidney cells

American journal of physiology. Renal physiology, 2012-11, Vol.303 (10), p.F1412-F1424

2012

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Cyclic AMP promotes growth and secretion in human polycystic kidney epithelial cells

Kidney international, 2004-09, Vol.66 (3), p.964-973

2004

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ADPKD cell proliferation and Cl--dependent fluid secretion

Methods in Kidney Cell Biology Part A, 2019, Vol.153, p.69-92

2019

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Ouabain activates the Na-K-ATPase signalosome to induce autosomal dominant polycystic kidney disease cell proliferation

American journal of physiology. Renal physiology, 2011-10, Vol.301 (4), p.F897-F906

2011

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Polycystin-1 Interacting Protein-1 (CU062) Interacts with the Ectodomain of Polycystin-1 (PC1)

Cells (Basel, Switzerland), 2023-08, Vol.12 (17), p.2166

2023

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Generation of primary cells from ADPKD and normal human kidneys

Methods in Kidney Cell Biology Part A, 2019, Vol.153, p.1-23

2019

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Ouabain Regulates CFTR-Mediated Anion Secretion and Na,K-ATPase Transport in ADPKD Cells

The Journal of membrane biology, 2015-12, Vol.248 (6), p.1145-1157

2015

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Identification of a forskolin-like molecule in human renal cysts

Journal of the American Society of Nephrology, 2007-03, Vol.18 (3), p.934-943

2007

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Extracellular matrix, integrins, and focal adhesion signaling in polycystic kidney disease

Cellular signalling, 2020-08, Vol.72, p.109646-109646, Article 109646

2020

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Elevated Expression of CaMK4 Contributes to mTOR-dependent Cell Proliferation and Cyst Growth of ADPKD Cells: PO1509

Journal of the American Society of Nephrology, 2020-10, Vol.31 (10S), p.484-484

2020

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Active BRAF Inhibits LKB1-AMPK Signaling and Acetyl-CoA Carboxylase Phosphorylation and Increases Renal Fibrosis in a Model of Polycystic Kidney Disease: FR-PO253

Journal of the American Society of Nephrology, 2022-11, Vol.33 (11S), p.395-395

2022

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Tolvaptan inhibits ERK-dependent cell proliferation, Cl⁻ secretion, and in vitro cyst growth of human ADPKD cells stimulated by vasopressin

Deficient transient receptor potential vanilloid type 4 function contributes to compromised [Ca 2+ ] i homeostasis in human autosomal-dominant polycystic kidney disease cells

Calcium restores a normal proliferation phenotype in human polycystic kidney disease epithelial cells

Phosphodiesterase Isoform Regulation of Cell Proliferation and Fluid Secretion in Autosomal Dominant Polycystic Kidney Disease

Integrin-Linked Kinase Signaling Promotes Cyst Growth and Fibrosis in Polycystic Kidney Disease

Periostin overexpression in collecting ducts accelerates renal cyst growth and fibrosis in polycystic kidney disease

CaMK4 overexpression in polycystic kidney disease promotes mTOR-mediated cell proliferation

Expression of active B-Raf proto-oncogene in kidney collecting ducts induces cyst formation in normal mice and accelerates cyst growth in mice with polycystic kidney disease

Periostin promotes renal cyst growth and interstitial fibrosis in polycystic kidney disease

The lonidamine derivative H2-gamendazole reduces cyst formation in polycystic kidney disease

Sorafenib inhibits cAMP-dependent ERK activation, cell proliferation, and in vitro cyst growth of human ADPKD cyst epithelial cells

Ouabain promotes partial epithelial to mesenchymal transition (EMT) changes in human autosomal dominant polycystic kidney disease (ADPKD) cells

Endogenous concentrations of ouabain act as a cofactor to stimulate fluid secretion and cyst growth of in vitro ADPKD models via cAMP and EGFR-Src-MEK pathways

In vitro cyst formation of ADPKD cells

Calmodulin-sensitive adenylyl cyclases mediate AVP-dependent cAMP production and Cl- secretion by human autosomal dominant polycystic kidney cells

Cyclic AMP promotes growth and secretion in human polycystic kidney epithelial cells

ADPKD cell proliferation and Cl--dependent fluid secretion

Ouabain activates the Na-K-ATPase signalosome to induce autosomal dominant polycystic kidney disease cell proliferation

Polycystin-1 Interacting Protein-1 (CU062) Interacts with the Ectodomain of Polycystin-1 (PC1)

Generation of primary cells from ADPKD and normal human kidneys

Ouabain Regulates CFTR-Mediated Anion Secretion and Na,K-ATPase Transport in ADPKD Cells

Identification of a forskolin-like molecule in human renal cysts

Extracellular matrix, integrins, and focal adhesion signaling in polycystic kidney disease

Elevated Expression of CaMK4 Contributes to mTOR-dependent Cell Proliferation and Cyst Growth of ADPKD Cells: PO1509

Active BRAF Inhibits LKB1-AMPK Signaling and Acetyl-CoA Carboxylase Phosphorylation and Increases Renal Fibrosis in a Model of Polycystic Kidney Disease: FR-PO253

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