UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

The fetal and maternal consequences of gestational diabetes mellitus

The journal of maternal-fetal & neonatal medicine, 2010-03, Vol.23 (3), p.199-203

Reece, E. Albert

2010

Volltextzugriff (PDF)

Decoding the oxidative stress hypothesis in diabetic embryopathy through proapoptotic kinase signaling

American journal of obstetrics and gynecology, 2015-05, Vol.212 (5), p.569-579

2015

Volltextzugriff (PDF)

Effect of maternal diabetes on the embryo, fetus, and children: Congenital anomalies, genetic and epigenetic changes and developmental outcomes

Birth defects research. Part C. Embryo today, 2015-03, Vol.105 (1), p.53-72

2015

Volltextzugriff (PDF)

Curcumin ameliorates high glucose-induced neural tube defects by suppressing cellular stress and apoptosis

American journal of obstetrics and gynecology, 2015-06, Vol.212 (6), p.802.e1-802.e8

2015

Volltextzugriff (PDF)

Gestational diabetes: the need for a common ground

Lancet, 2009-05, Vol.373 (9677), p.1789-1797

2009

Volltextzugriff (PDF)

The green tea polyphenol EGCG alleviates maternal diabetes–induced neural tube defects by inhibiting DNA hypermethylation

American journal of obstetrics and gynecology, 2016-09, Vol.215 (3), p.368.e1-368.e10

2016

Volltextzugriff (PDF)

Punicalagin exerts protective effect against high glucose-induced cellular stress and neural tube defects

Biochemical and biophysical research communications, 2015-11, Vol.467 (2), p.179-184

2015

Volltextzugriff (PDF)

Tip60- and sirtuin 2-regulated MARCKS acetylation and phosphorylation are required for diabetic embryopathy

Nature communications, 2019-01, Vol.10 (1), p.282-282, Article 282

2019

Volltextzugriff (PDF)

Protein kinase C-alpha suppresses autophagy and induces neural tube defects via miR-129-2 in diabetic pregnancy

Nature communications, 2017-05, Vol.8 (1), p.15182-14, Article 15182

2017

Volltextzugriff (PDF)

Type 2 diabetes mellitus induces congenital heart defects in murine embryos by increasing oxidative stress, endoplasmic reticulum stress, and apoptosis

American journal of obstetrics and gynecology, 2016-09, Vol.215 (3), p.366.e1-366.e10

2016

Volltextzugriff (PDF)

Oxidative stress is responsible for maternal diabetes-impaired transforming growth factor beta signaling in the developing mouse heart

American journal of obstetrics and gynecology, 2015-05, Vol.212 (5), p.650.e1-650.e11

2015

Volltextzugriff (PDF)

High glucose suppresses embryonic stem cell differentiation into neural lineage cells

Biochemical and biophysical research communications, 2016-04, Vol.472 (2), p.306-312

2016

Volltextzugriff (PDF)

Advances in revealing the molecular targets downstream of oxidative stress–induced proapoptotic kinase signaling in diabetic embryopathy

American journal of obstetrics and gynecology, 2015-08, Vol.213 (2), p.125-134

2015

Volltextzugriff (PDF)

Oxidative stress-induced miR-27a targets the redox gene nuclear factor erythroid 2-related factor 2 in diabetic embryopathy

American journal of obstetrics and gynecology, 2018-01, Vol.218 (1), p.136.e1-136.e10

2018

Volltextzugriff (PDF)

Diabetes-Induced Birth Defects: What Do We Know? What Can We Do?

Current diabetes reports, 2012-02, Vol.12 (1), p.24-32

Reece, E. Albert

2012

Volltextzugriff (PDF)

Formation of neurodegenerative aggresome and death-inducing signaling complex in maternal diabetes-induced neural tube defects

Proceedings of the National Academy of Sciences - PNAS, 2017-04, Vol.114 (17), p.4489-4494

2017

Volltextzugriff (PDF)

Ask1 gene deletion blocks maternal diabetes-induced endoplasmic reticulum stress in the developing embryo by disrupting the unfolded protein response signalosome

Diabetes (New York, N.Y.), 2015-03, Vol.64 (3), p.973-988

2015

Volltextzugriff (PDF)

New development of the yolk sac theory in diabetic embryopathy: molecular mechanism and link to structural birth defects

American journal of obstetrics and gynecology, 2016-02, Vol.214 (2), p.192-202

2016

Volltextzugriff (PDF)

Oxidative Stress―Induced JNK1/2 Activation Triggers Proapoptotic Signaling and Apoptosis That Leads to Diabetic Embryopathy

Diabetes (New York, N.Y.), 2012-08, Vol.61 (8), p.2084-2092

2012

Volltextzugriff (PDF)

Pregestational type 2 diabetes mellitus induces cardiac hypertrophy in the murine embryo through cardiac remodeling and fibrosis

American journal of obstetrics and gynecology, 2017-08, Vol.217 (2), p.216.e1-216.e13

2017

Volltextzugriff (PDF)

Superoxide dismutase 1 overexpression in mice abolishes maternal diabetes–induced endoplasmic reticulum stress in diabetic embryopathy

American journal of obstetrics and gynecology, 2013-10, Vol.209 (4), p.345.e1-345.e7

2013

Volltextzugriff (PDF)

Maternal diabetes induces senescence and neural tube defects sensitive to the senomorphic rapamycin

Science advances, 2021-06, Vol.7 (27)

2021

Volltextzugriff (PDF)

Diabetes mellitus and birth defects

American journal of obstetrics and gynecology, 2008-09, Vol.199 (3), p.237.e1-237.e9

2008

Volltextzugriff (PDF)

Perspectives on obesity, pregnancy and birth outcomes in the United States: The scope of the problem

American journal of obstetrics and gynecology, 2008, Vol.198 (1), p.23-27

Reece, E. Albert, MD, PhD, MBA

2008

Volltextzugriff (PDF)

Birth defects in pregestational diabetes： Defect range,glycemic threshold and pathogenesis

World journal of diabetes, 2015-04, Vol.6 (3), p.481-488

2015

Volltextzugriff (PDF)

1 – 25 von 569

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(363)
Mit Volltextzugriff
(443)

Publikationsform

1 weiteren anzeigen...

Weniger anzeigen...

Sprache

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 30 Tagen
(3)
... seit 90 Tagen
(4)

Menü

Weitere Dienste

Einstellungen

The fetal and maternal consequences of gestational diabetes mellitus

Decoding the oxidative stress hypothesis in diabetic embryopathy through proapoptotic kinase signaling

Effect of maternal diabetes on the embryo, fetus, and children: Congenital anomalies, genetic and epigenetic changes and developmental outcomes

Curcumin ameliorates high glucose-induced neural tube defects by suppressing cellular stress and apoptosis

Gestational diabetes: the need for a common ground

The green tea polyphenol EGCG alleviates maternal diabetes–induced neural tube defects by inhibiting DNA hypermethylation

Punicalagin exerts protective effect against high glucose-induced cellular stress and neural tube defects

Tip60- and sirtuin 2-regulated MARCKS acetylation and phosphorylation are required for diabetic embryopathy

Protein kinase C-alpha suppresses autophagy and induces neural tube defects via miR-129-2 in diabetic pregnancy

Type 2 diabetes mellitus induces congenital heart defects in murine embryos by increasing oxidative stress, endoplasmic reticulum stress, and apoptosis

Oxidative stress is responsible for maternal diabetes-impaired transforming growth factor beta signaling in the developing mouse heart

High glucose suppresses embryonic stem cell differentiation into neural lineage cells

Advances in revealing the molecular targets downstream of oxidative stress–induced proapoptotic kinase signaling in diabetic embryopathy

Oxidative stress-induced miR-27a targets the redox gene nuclear factor erythroid 2-related factor 2 in diabetic embryopathy

Diabetes-Induced Birth Defects: What Do We Know? What Can We Do?

Formation of neurodegenerative aggresome and death-inducing signaling complex in maternal diabetes-induced neural tube defects

Ask1 gene deletion blocks maternal diabetes-induced endoplasmic reticulum stress in the developing embryo by disrupting the unfolded protein response signalosome

New development of the yolk sac theory in diabetic embryopathy: molecular mechanism and link to structural birth defects

Oxidative Stress―Induced JNK1/2 Activation Triggers Proapoptotic Signaling and Apoptosis That Leads to Diabetic Embryopathy

Pregestational type 2 diabetes mellitus induces cardiac hypertrophy in the murine embryo through cardiac remodeling and fibrosis

Superoxide dismutase 1 overexpression in mice abolishes maternal diabetes–induced endoplasmic reticulum stress in diabetic embryopathy

Maternal diabetes induces senescence and neural tube defects sensitive to the senomorphic rapamycin

Diabetes mellitus and birth defects

Perspectives on obesity, pregnancy and birth outcomes in the United States: The scope of the problem

Birth defects in pregestational diabetes： Defect range,glycemic threshold and pathogenesis

Suchergebnisse filtern